Anti Dysrhythmias Medications

A patient is prescribed quinidine for the treatment of supraventricular dysrhythmia. Which of the following potential effects of quinidine on the heart should the nurse monitor the patient for?

A patient is started on lidocaine (Xylocaine) for ventricular dysrhythmias. Which of the following effects on the ECG would be expected?

A patient with a history of dysrhythmias is prescribed a Class IC antiarrhythmic drug. What potentially serious adverse effect should the healthcare provider closely monitor for?

A patient taking quinidine develops ringing in the ears, blurred vision, and a headache. The patient's labs also indicate they have cardiotoxicity. The nurse recognizes these symptoms as indicative of:

A patient is prescribed propranolol for the treatment of dysrhythmias. Propranolol is in which class of anti-dysrhythmic drugs?

A patient's ECG shows a prolonged QT interval. Which class of antidysrhythmic drugs should be administered with extreme caution due to the risk of Torsades de pointes?

A patient is diagnosed with a bradydysrhythmia. Which area of the heart is MOST likely to be affected, causing this condition?

A patient with a history of sustained supraventricular tachycardia (SVT) is prescribed an antidysrhythmic drug. The medication works by slowing conduction through the AV node. Which drug is MOST likely prescribed?

A patient is experiencing atrial fibrillation. The ECG reading would NOT show which of the following?

Which of the following best describes the mechanism of action of Class I antidysrhythmic drugs?

A patient is prescribed digoxin for heart failure and subsequently develops ventricular premature beats. Which electrolyte imbalance is MOST likely contributing to this digoxin-induced dysrhythmia?

Which ECG component represents ventricular repolarization?

A patient is diagnosed with a re-entry arrhythmia. Which mechanism is primarily responsible for this type of dysrhythmia?

Which of the following effects of propranolol contributes to its therapeutic use in treating dysrhythmias caused by excessive sympathetic stimulation?

A patient with recurrent ventricular fibrillation is prescribed amiodarone. What electrophysiological effect of amiodarone is most directly related to its therapeutic action in this scenario?

A patient taking amiodarone begins experiencing excessive slowing of heart rate. Which concurrent medication is most likely contributing to this adverse effect?

Verapamil is prescribed to a patient with atrial fibrillation. The primary goal of this medication in this situation is to:

A patient on digoxin develops hypokalemia. Which of the following is the most significant risk associated with this electrolyte imbalance in the context of digoxin therapy?

A patient with a history of asthma is prescribed an antiarrhythmic drug. Which of the following medications should be avoided, if possible, due to its potential to exacerbate the patient's respiratory condition?

Which antidysrhythmic drug is also commonly used in the treatment of heart failure?

Which of the following is a common adverse effect associated with verapamil and diltiazem?

A patient is prescribed amiodarone and simvastatin concurrently. What potential drug interaction should the prescriber be most concerned about?

Sotalol shares mechanism of action with which class of antiarrhythmic drugs?

Which of the following is the primary mechanism of action of Class IA antiarrhythmic drugs like quinidine?

Which electrocardiogram (ECG) change is MOST likely to be observed in a patient taking a Class IA antiarrhythmic drug?

A patient receiving lidocaine for ventricular dysrhythmias begins to exhibit confusion and paresthesias. Which of the following is the MOST likely explanation for these adverse effects?

Why are Class IC antiarrhythmic drugs generally reserved for use in patients with otherwise structurally normal hearts and limited dysrhythmias?

A patient is prescribed a beta-blocker for the management of a supraventricular tachyarrhythmia originating from increased sympathetic drive. Given the limited selection of beta-blockers specifically approved for dysrhythmias, and considering the need for rapid control during an acute episode, which of the following would be the MOST appropriate initial choice?

Which electrophysiological effect is associated with amiodarone?

A patient with recurrent ventricular fibrillation is prescribed amiodarone. What is the primary therapeutic action of amiodarone in this scenario?

Verapamil and diltiazem are effective in treating which type of supraventricular tachycardia (SVT)?

Which adverse effect is most closely associated with amiodarone's long half-life?

Which concurrent medication increases the risk of excessive slowing of heart rate when taken with amiodarone?

Which electrolyte imbalance potentiates digoxin toxicity, increasing the risk of cardiotoxicity?

A patient is prescribed digoxin for heart failure and subsequently develops visual disturbances and gastrointestinal upset. Which of the following is the most likely cause?

What is the primary mechanism by which digoxin suppresses supraventricular dysrhythmias?

A patient with heart failure and atrial fibrillation is prescribed digoxin. Besides slowing the ventricular rate, what other potential benefit does digoxin offer in this case?

A patient with a history of asthma is prescribed an antiarrhythmic drug. Which of the following beta-blockers should be avoided, if possible, due to its non-selective beta-adrenergic blocking properties?

Which of the following best describes the primary mechanism by which Class IV antidysrhythmic drugs exert their effects on cardiac tissue?

A patient develops a dysrhythmia characterized by rapid, chaotic atrial activity. Which Vaughn Williams classification of antidysrhythmic drug would least be appropriate for rate control?

Which of the following is a potential consequence of a drug-induced disturbance in automaticity within the heart?

A patient is experiencing re-entry arrhythmia. Which of the following is the most likely underlying mechanism?

Which of the following scenarios presents the greatest risk associated with antidysrhythmic drug therapy?

A patient with a history of asthma is prescribed an antidysrhythmic drug. Which of the following medications has the LEAST impact on a patient's respiratory condition?

A patient is prescribed a medication that selectively blocks $I_{Kr}$ channels in cardiac myocytes. What effect would you expect this medication to have on the action potential?

A researcher is investigating a novel compound that prolongs the effective refractory period (ERP) in ventricular myocytes without significantly affecting the QT interval. Which ionic current is MOST likely targeted by this compound?

Class IA Agents

Class IB Agents

Class IC Agents

Quinidine

Lidocaine

Propranolol (Inderal)

Propranolol's Cardiac Effects

Propranolol Therapeutic Use

Amiodarone Therapeutic Use

Amiodarone's Effects on ECG

Amiodarone Drug Interactions

Amiodarone Adverse Effects

Verapamil/Diltiazem Effects

Verapamil/Diltiazem Uses

Verapamil/Diltiazem Adverse Effects

Dysrhythmia

SA Node

AV Node

P Wave

QRS Complex

T Wave

Causes of Dysrhythmias

Supraventricular Dysrhythmias

Quinidine's Cardiac Effects

Major Quinidine Adverse Effects

Quinidine-Digoxin Interaction

Lidocaine's Main Adverse Effects

Risk with Class IC Agents

Tachydysrhythmias

Bradydysrhythmias

Electrocardiogram (ECG)

Atrioventricular Block

Reentry (recirculating activation)

Class I Antidysrhythmics

Class II Antidysrhythmics

Class III Antidysrhythmics

Propranolol Class

Propranolol Adverse Cardiac Effects

Digoxin's Action on Dysrhythmias

Class III Mechanisms

Amiodarone's Notorious Adverse Effects

Diuretics and Antiarrhythmics Risk

Amiodarone + Beta Blocker Risk

Verapamil & Diltiazem: Uses

Digoxin's Action on AV/SA Nodes

Digoxin's Main Adverse Effect