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Questions and Answers
Which effect of β-blockers makes them the drugs of choice to treat effort-induced angina?
Which effect of β-blockers makes them the drugs of choice to treat effort-induced angina?
- Decreasing heart rate and contractility (correct)
- Reducing cardiac output and blood pressure
- Lowering the rate of contraction of the heart
- Lowering the force of contraction of the heart
Why are β-blockers ineffective against vasospastic angina?
Why are β-blockers ineffective against vasospastic angina?
- They reduce cardiac output and blood pressure
- They are nonselective at high doses and can inhibit β2 receptors (correct)
- They increase heart rate and contractility
- They suppress the activation of the heart
Why are agents with intrinsic sympathomimetic activity less effective and should be avoided in angina?
Why are agents with intrinsic sympathomimetic activity less effective and should be avoided in angina?
- They are nonselective at high doses and can inhibit β2 receptors
- They reduce the work of the heart
- They are less effective in reducing the frequency and severity of angina attacks (correct)
- They suppress the activation of the heart
What is particularly important to remember in the case of asthmatic patients when using β-blockers?
What is particularly important to remember in the case of asthmatic patients when using β-blockers?
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Why are β-blockers without intrinsic sympathomimetic activity particularly useful in the treatment of patients with myocardial infarction?
Why are β-blockers without intrinsic sympathomimetic activity particularly useful in the treatment of patients with myocardial infarction?
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Why are metoprolol and atenolol preferred over propranolol for treating effort-induced angina?
Why are metoprolol and atenolol preferred over propranolol for treating effort-induced angina?
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