Podcast
Questions and Answers
Stress adaptation allows bacterial cells to grow faster in changed conditions.
Stress adaptation allows bacterial cells to grow faster in changed conditions.
False (B)
Sub-lethal injury causes a permanent physiological change in bacterial cells.
Sub-lethal injury causes a permanent physiological change in bacterial cells.
False (B)
Lethal stress always causes the death of all bacterial cells in the population.
Lethal stress always causes the death of all bacterial cells in the population.
False (B)
Stress adaptation enables bacterial cells to resist subsequent exposure to harsher treatment.
Stress adaptation enables bacterial cells to resist subsequent exposure to harsher treatment.
Experiencing lethality by a fraction of the bacterial population may lead to improved survival of the overall population.
Experiencing lethality by a fraction of the bacterial population may lead to improved survival of the overall population.
Stress adaptation enables bacterial cells to resist subsequent exposure to harsher treatment.
Stress adaptation enables bacterial cells to resist subsequent exposure to harsher treatment.
Experiencing lethality by a fraction of the bacterial population may lead to improved survival of the overall population.
Experiencing lethality by a fraction of the bacterial population may lead to improved survival of the overall population.
Sub-lethal injury causes a permanent physiological change in bacterial cells.
Sub-lethal injury causes a permanent physiological change in bacterial cells.
Bacterial cells adapt completely to the changed conditions, and growth rate is not affected.
Bacterial cells adapt completely to the changed conditions, and growth rate is not affected.
A brief exposure to a suboptimal environment triggers cellular mechanisms that enable bacterial cells to resist subsequent exposure to harsher treatment.
A brief exposure to a suboptimal environment triggers cellular mechanisms that enable bacterial cells to resist subsequent exposure to harsher treatment.
What is the main cause of staphylococcal food poisoning?
What is the main cause of staphylococcal food poisoning?
Why is staphylococcal food poisoning considered as 'intoxication'?
Why is staphylococcal food poisoning considered as 'intoxication'?
What distinguishes staphylococcal toxin from other toxins in terms of its resistance to heat?
What distinguishes staphylococcal toxin from other toxins in terms of its resistance to heat?
Which compound excreted by S. aureus is considered a virulence factor?
Which compound excreted by S. aureus is considered a virulence factor?
In 1914, how was the link between staphylococci and foodborne illness established?
In 1914, how was the link between staphylococci and foodborne illness established?
Study Notes
Stress Adaptation in Bacterial Cells
- Stress adaptation enables bacterial cells to grow faster in changed conditions and resist subsequent exposure to harsher treatment.
- Sub-lethal injury causes a permanent physiological change in bacterial cells, allowing them to adapt to new conditions.
- Experiencing lethality by a fraction of the bacterial population may lead to improved survival of the overall population.
- Brief exposure to a suboptimal environment triggers cellular mechanisms that enable bacterial cells to resist subsequent exposure to harsher treatment.
Staphylococcal Food Poisoning
- The main cause of staphylococcal food poisoning is the ingestion of enterotoxins produced by Staphylococcus aureus.
- Staphylococcal food poisoning is considered an 'intoxication' because it is caused by the ingestion of pre-formed toxins.
- Staphylococcal toxin is resistant to heat, distinguishing it from other toxins.
- The compound excreted by S. aureus considered a virulence factor is alpha-toxin.
- The link between staphylococci and foodborne illness was established in 1914.
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Description
Explore the concept of microbial stress response in food environments and the degrees of bacterial injury. Learn about stress adaptation, sub-lethal injury, and lethal stress, and their impact on bacterial cells.
