Mechanism of Action of Cardiac Glycosides

What is the primary mechanism of action of cardiac glycosides like Digoxin in increasing contractility in the heart?

What effect do calcium channel blockers (Dihydropyridines) have on contractility in the heart?

Which type of drugs act on Autonomic Neural Control by being Cardio-Selective β-Blockers?

What is the primary mechanism of action of drugs modulating the Renin-Angiotensin-Aldosterone system (RAAS) in managing heart failure?

Which of the following is NOT a characteristic of inotropic drugs used in acute heart failure?

What role do non-dihydropyridine calcium channel blockers play in managing heart conditions?

Which class of CCBs has greater selectivity for vascular smooth muscle cells than for cardiac myocytes?

Which class of CCBs is primarily used to treat hypertension due to their high vascular selectivity?

Which CCB class can lead to reflex cardiac stimulation (tachycardia and increased inotropy) due to their powerful systemic vasodilator effects?

Which class of CCBs is relatively selective for the myocardium and less effective as a systemic vasodilator drug?

Which drug is an intermediate between verapamil and dihydropyridines, reducing arterial pressure without causing significant reflex cardiac stimulation?

What is the primary mechanism of action of Cardiac Glycosides like Digoxin?

What is the primary mechanism of action of Calcium Channel Blockers (CCBs) like Dihydropyridines?

Which effect is NOT associated with the action of Dihydropyridines?

What is the primary pharmacological action of Cardiac Glycosides such as Digoxin?

Which of the following is a characteristic of Non-Dihydropyridine Calcium Channel Blockers?

Which process is NOT modulated by Autonomic Neural Control in the cardiovascular system?

Inotropic drugs primarily affect which aspect of cardiac function?