Inflammation Concepts and Mechanisms

<p>Tissue injury from trauma (D)</p> Signup and view all the answers

<p>Inflamma (D)</p> Signup and view all the answers

<p>Local and Systemic (B)</p> Signup and view all the answers

<p>Chemotaxis (C)</p> Signup and view all the answers

<p>Increased vascular permeability (A)</p> Signup and view all the answers

<p>Leukocytes (D)</p> Signup and view all the answers

<p>Increased blood flow, increased vascular permeability, leukocytic infiltration (C)</p> Signup and view all the answers

<p>Destruction of microbes (C)</p> Signup and view all the answers

<p>The process of leukocyte migration towards the site of inflammation (C)</p> Signup and view all the answers

<p>The microbe is not destroyed and the inflammation persists. (A)</p> Signup and view all the answers

<p>True (B)</p> Signup and view all the answers

<p>True (B)</p> Signup and view all the answers

<p>Infiltration with mononuclear cells (C)</p> Signup and view all the answers

<p>Infiltration with mononuclear cells (C)</p> Signup and view all the answers

<p>Both A and B (C)</p> Signup and view all the answers

<p>Both A and B (A)</p> Signup and view all the answers

<p>Tissue necrosis (B)</p> Signup and view all the answers

<p>All of the above (D)</p> Signup and view all the answers

<p>Regeneration of parenchymal cells (B)</p> Signup and view all the answers

<p>Tissue death (A)</p> Signup and view all the answers

<p>The formation of an abscess (C)</p> Signup and view all the answers

<p>Failure of acute inflammation to eliminate the inciting stimulus (B)</p> Signup and view all the answers

<p>Acute inflammation involves a rapid onset, while chronic inflammation develops slowly. (C)</p> Signup and view all the answers

<p>Neutrophils (A)</p> Signup and view all the answers

<p>Fibrin prevents the regeneration of damaged tissue (A)</p> Signup and view all the answers

<p>Rapid onset and short duration (B)</p> Signup and view all the answers

<p>It can persist for months or even years. (A)</p> Signup and view all the answers

<p>Macrophage activation (A)</p> Signup and view all the answers

<p>All of the above are correct. (D)</p> Signup and view all the answers

<p>Destruction of foreign materials or microbes (A)</p> Signup and view all the answers

<p>Mast cells (B), Eosinophils (D)</p> Signup and view all the answers

<p>Insulin (C)</p> Signup and view all the answers

<p>Fibrosis (C)</p> Signup and view all the answers

<p>Presence of pus (B)</p> Signup and view all the answers

<p>Interleukin 6 (D)</p> Signup and view all the answers

A complex protective reaction of the body to injury.

Common indicators include redness, heat, pain, swelling, and loss of function.

Caused by factors like infections (bacterial, viral, fungal) or injury.

Acute is short-term and quick; chronic lasts longer and can be harmful.

Dynamic responses involving vessels and immune cells to repair damage.

Rapid onset inflammation caused by harmful bacteria or tissue injury, lasting a few days.

Slow onset inflammation, often due to pathogens the body can't eliminate, lasting months to years.

White blood cells that primarily fight infections during acute inflammation.

Can improve, turn into an abscess, or become chronic if unresolved.

Results in tissue death, thickening, and scarring of connective tissue.

Factors like burns, cuts, and radiation causing injury.

Substances such as drugs and toxins that can cause harm.

Body's immune response causing conditions like rheumatoid arthritis.

Inflammatory responses occurring at the specific site of injury.

Widespread inflammatory responses affecting the entire body.

Process where cells engulf and digest microbes and debris.

Movement of white blood cells into the site of inflammation.

Substances released during inflammation that regulate the response.

A type of white blood cell involved in immune response by engulfing pathogens.

Proteins produced by immune cells that communicate and modulate immune responses.

Manifestations such as abscesses, rashes, and allergies caused by inflammatory responses.

Diseases where the immune system attacks the body's own cells, e.g., lupus or rheumatoid arthritis.

Begins within seconds to minutes of injury, lasts from minutes to days involving edema and neutrophils.

Inflammation ends when the damaging agent is eliminated and anti-inflammatory mechanisms activate.

Involves infiltration with mononuclear cells, destruction of tissue, and attempts at healing through fibrosis.

The process where inflammation leads to further injury, continuing the cycle of inflammation.

The process of replacing damaged tissues through regeneration or scarring.

A type of immune cell involved in chronic inflammation, aiding in tissue destruction and healing.

Acute inflammation where injury is limited, little tissue is destroyed, and parenchymal cells can regenerate.

A process where substantial tissue destruction occurs, involving tissue incapable of regeneration, leading to scar formation.

The process during inflammation where dead cells and microbes are cleared from the site of injury.

The absorption of excess fluid from tissues by lymphatics during inflammation.

Occurs when acute inflammation fails to resolve properly, potentially due to insufficient antibodies or persistence of stimuli.

Specialized cells formed during chronic inflammation, often associated with granulomatous conditions.

The process where a thrombus (blood clot) is replaced by granulation tissue during healing, restoring vascular integrity.

New connective tissue formed during the healing process, rich in capillaries, fibroblasts, and collagen.