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Questions and Answers
What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
In type III hypersensitivity, if immune complexes are not eliminated, where do they mostly deposit and trigger inflammation?
In type III hypersensitivity, if immune complexes are not eliminated, where do they mostly deposit and trigger inflammation?
Which disease is commonly associated with the deposition of immune complexes with anti-nuclear autoantibodies in multiple organs such as heart, joints, skin, kidneys, and lungs?
Which disease is commonly associated with the deposition of immune complexes with anti-nuclear autoantibodies in multiple organs such as heart, joints, skin, kidneys, and lungs?
Which type of hypersensitivity involves complement and neutrophils as mechanisms of tissue damage?
Which type of hypersensitivity involves complement and neutrophils as mechanisms of tissue damage?
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What is the mechanism of tissue injury in type IV hypersensitivity?
What is the mechanism of tissue injury in type IV hypersensitivity?
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Which mechanism of tissue damage is common to both type III hypersensitivity and type IV hypersensitivity?
Which mechanism of tissue damage is common to both type III hypersensitivity and type IV hypersensitivity?
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In which disease do immune complexes with anti-nuclear autoantibodies most commonly deposit in multiple organs such as heart, joints, skin, kidneys, and lungs?
In which disease do immune complexes with anti-nuclear autoantibodies most commonly deposit in multiple organs such as heart, joints, skin, kidneys, and lungs?
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What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
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Where do immune complexes mostly deposit and trigger inflammation if not eliminated in type III hypersensitivity?
Where do immune complexes mostly deposit and trigger inflammation if not eliminated in type III hypersensitivity?
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Which immune response is initiated when an antigen like purified protein derivative (PPD) of mycobacterium is injected intradermally in an individual?
Which immune response is initiated when an antigen like purified protein derivative (PPD) of mycobacterium is injected intradermally in an individual?
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What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
What is the classic clinical demonstration of the function of delayed-type hypersensitivity (DTH) response?
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Where do immune complexes mostly deposit and trigger inflammation if not eliminated in type III hypersensitivity?
Where do immune complexes mostly deposit and trigger inflammation if not eliminated in type III hypersensitivity?
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Which immune response is initiated when an antigen like protein purified derivative (PPD) of mycobacterium is injected intradermally in an individual?
Which immune response is initiated when an antigen like protein purified derivative (PPD) of mycobacterium is injected intradermally in an individual?
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In which disease do immune complexes with anti-nuclear autoantibodies most commonly deposit in multiple organs such as heart, joints, skin, kidneys, and lungs?
In which disease do immune complexes with anti-nuclear autoantibodies most commonly deposit in multiple organs such as heart, joints, skin, kidneys, and lungs?
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What is the mechanism of tissue injury in type IV hypersensitivity?
What is the mechanism of tissue injury in type IV hypersensitivity?
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