Heart Failure Overview
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Questions and Answers

What is heart failure (HF)?

The inability of the heart to pump enough blood to meet the needs of the tissues for oxygen and nutrients.

Heart failure is always a progressive, lifelong disorder.

False (B)

What are the two main types of heart failure?

  • Systolic failure (correct)
  • Left-sided heart failure
  • Diastolic failure (correct)
  • Right-sided heart failure

What is the pathophysiology of heart failure?

<p>The pathophysiology of heart failure is complex and involves multiple factors, including myocardial dysfunction, activation of the renin-angiotensin-aldosterone system, activation of the sympathetic nervous system, and ventricular remodeling.</p> Signup and view all the answers

What are some of the clinical manifestations of right-sided heart failure?

<p>Jugular venous distention (JVD) (A), Ascites (B), Weight gain (C), Dependent edema (D), Visceral and peripheral congestion (E), Hepatomegaly (F)</p> Signup and view all the answers

Flashcards

Heart Failure (HF)

Inability of the heart to pump enough blood to meet the body's needs for oxygen and nutrients.

Systolic Failure

Problem with heart's contraction, making it pump less forcefully.

Diastolic Failure

Problem with heart filling, making it hold less blood.

Right-Sided HF

Fluid buildup in the body's tissues outside the lungs.

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Left-Sided HF

Fluid buildup in the lungs.

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Jugular Venous Distention (JVD)

Swollen neck veins, a sign of fluid buildup.

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Peripheral Edema

Swelling in the extremities (legs and feet).

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Hepatomegaly

Enlarged liver due to fluid buildup.

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Ascites

Fluid buildup in the abdomen.

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Dyspnea on exertion (DOE)

Shortness of breath during physical activity.

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Orthopnea

Difficulty breathing when lying flat.

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Activity Intolerance

Inability to perform activities due to lack of energy.

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Excess Fluid Volume

Body holding too much fluid.

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ACE Inhibitors

Medications that reduce blood pressure and help the heart.

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Fluid Restriction

Limiting fluid intake to manage fluid overload.

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Daily Weights

Tracking weight changes to monitor fluid balance.

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Low-Sodium Diet

Reducing sodium intake to manage fluid.

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Diuretic Therapy

Using medications to increase urination and remove excess fluid.

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Cardiac Output (CO)

Amount of blood pumped by the heart per minute.

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Vital Signs

Measurements of heart rate, blood pressure, and respiratory rate.

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Pulse Oximetry

Measurement of oxygen saturation in the blood.

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Patient Education

Teaching the patient about their condition and management.

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Anxiety

Fear or worry related to the health condition.

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Study Notes

Chapter 29: Management of Patients With Complications From Heart Disease

  • This chapter covers the management of patients with heart disease complications, specifically focusing on heart failure (HF).
  • Heart failure (HF) is the inability of the heart to pump enough blood to meet the tissues' needs for oxygen and nutrients.
  • It's a syndrome characterized by fluid overload or inadequate tissue perfusion.
  • Heart failure indicates myocardial disease, a problem with heart contraction (systolic failure) or filling (diastolic failure).
  • Some cases of heart failure are reversible.
  • Most cases are progressive and lifelong, managed with lifestyle changes and medications.

Heart Failure (HF)

  • HF is a progressive, lifelong disorder.
  • It is managed with lifestyle changes and medications.

Pathophysiology of Heart Failure

  • Myocardial dysfunction can be caused by various factors including ischemic heart disease, hyperthyroidism, myocardial infarction, valve disease, alcohol and/or cocaine abuse, and hypertension.
  • Activation of renin-angiotensin-aldosterone system: This system leads to vasoconstriction, increasing afterload, blood pressure, and heart rate. This also causes sodium and water retention.
  • Activation of baroreceptors: This leads to stimulation of vasomotor centers in the medulla.
  • Activation of sympathetic nervous system: Catecholamines (epinephrine and norepinephrine) are released, leading to vasoconstriction.

Clinical Manifestations

  • Right-sided: Visceral and peripheral congestion, jugular venous distention (JVD), dependent edema, hepatomegaly, ascites, and weight gain.
  • Left-sided: Pulmonary congestion, crackles, S3 or "ventricular gallop," dyspnea on exertion (DOE), orthopnea, dry, nonproductive cough, and oliguria.

Nursing Process: The Care of the Patient With Heart Failure—Assessment

  • Focus: Effectiveness of therapy, patient self-management, symptoms if HF increases, emotional/psychosocial response, health history, physical exam.
  • Physical exam: Mental status, lung sounds (crackles, wheezes), heart sounds (S3), fluid status, daily weight, I&O, assess response to medications.

Nursing Process: The Care of the Patient With Heart Failure—Diagnoses

  • Activity intolerance: Related to decreased cardiac output (CO)
  • Excess fluid volume: Related to the HF syndrome
  • Anxiety-related symptoms: Related to the complexity of the therapeutic regimen.
  • Powerlessness: Related to chronic illness and hospitalizations.
  • Ineffective family therapeutic regimen management.

Collaborative Problems and Potential Complications

  • Hypotension, poor perfusion, cardiogenic shock
  • Dysrhythmias
  • Thromboembolism, pericardial effusion, cardiac tamponade

Nursing Process: The Care of the Patient With Heart Failure—Planning

  • Goals: Promote activity and reduce fatigue, relieve fluid overload symptoms, decrease anxiety, encourage patient to verbalize ability to make decisions and influence outcomes, educate patient and family about therapeutic regimen management.

Activity Intolerance

  • Bed rest for acute exacerbations.
  • Encourage regular physical activity (30-45 minutes daily).
  • Exercise training.
  • Pacing of activities.
  • Wait two hours after eating for physical activity.
  • Avoid activities in extreme temperatures.
  • Modify activities to conserve energy.
  • Positioning: elevate HOB to facilitate breathing, support arms.

Fluid Volume Excess

  • Assess for symptoms of fluid overload.
  • Daily weight.
  • Input and output (I&O).
  • Diuretic therapy: timing of medications.
  • Fluid intake, fluid restriction.
  • Maintenance of sodium restriction.

Patient Education

  • Medications.
  • Diet: low-sodium, fluid restriction.
  • Monitor for signs of excess fluid, hypotension, disease exacerbation, daily weight.
  • Exercise and activity program.
  • Stress management.
  • Infection prevention.
  • Know how and when to contact healthcare provider.
  • Include family in education.

Medications

  • ACE inhibitors: For vasodilation, diuresis, decreased afterload, monitor for hypotension, hyperkalemia, and altered renal function, and cough.
  • Angiotensin II receptor blockers: Alternative to ACE inhibitors, work similarly.
  • Hydralazine and isosorbide dinitrate: Alternative to ACE inhibitors.
  • Beta-blockers: Prescribed in addition to ACE inhibitors, may take several weeks before effects are seen. Use with caution in patients with asthma.
  • Diuretics: Decrease fluid volume; monitor serum electrolytes.
  • Digitalis: Improves contractility; monitor for digitalis toxicity, especially if patient is hypokalemic.
  • IV medications (e.g., milrinone): For hospitalized patients admitted for acute decompensated HF; decreases preload and afterload, causes hypotension, and increased risk of dysrhythmias.
  • Dobutamine: For patients with left ventricular dysfunction; increases cardiac contractility and renal perfusion.

Gerontologic Considerations

  • May present with atypical signs/symptoms (fatigue, weakness, somnolence).
  • Decreased renal function can make older patients resistant to diuretics and more sensitive to volume changes.
  • Administration of diuretics in older men requires surveillance for bladder distention caused by prostatic enlargement.

Sudden Cardiac Arrest/Death

  • Emergency management: Cardiopulmonary resuscitation (CPR); A: airway; B: breathing; C: circulation; D: defibrillation for VT & VF.

Pulmonary Edema

  • Acute event where the left ventricle cannot handle blood volume overload. This causes increased pressure in the pulmonary vasculature and fluid moves out of pulmonary capillaries into interstitial space of the lungs and alveoli.
  • Results in hypoxemia.
  • Symptoms include restlessness, anxiety, dyspnea, cool/clammy skin, cyanosis, weak and rapid pulse, cough, and noisy, moist respirations, increased sputum production, frothy/blood tinged sputum, and decreased level of consciousness.
  • Management: Monitor lung sounds, signs of activity intolerance, place patient upright/dangle legs, minimize exertion/stress, administer oxygen, and diuretics (e.g. furosemide), vasodilators (e.g. nitroglycerin).

Thromboembolism

  • Decreased mobility and decreased circulation increase the risk of thromboembolism, especially with cardiac disorders, including HF.
  • Pulmonary embolism: blood clot that originates in legs and travels to obstruct pulmonary vessels.
  • Management: Anticoagulant therapy.

Pericardial Effusion and Cardiac Tamponade

  • Pericardial effusion: Accumulation of fluid in the pericardial sac.
  • Cardiac tamponade: Restriction of heart function due to fluid in pericardial sac, resulting in decreased venous return and decreased CO.
  • Symptoms include ill-defined chest pain or fullness, pulsus paradoxus, engorged neck veins, and labile or low BP, shortness of breath. Falling systolic BP, narrowing pulse pressure, and rising venous pressure may also occur.
  • Management: Pericardiocentesis, Pericardiotomy.

Other Important Info

  • Heart Failure Question: The correct answer to identify optimal management of activity intolerance with decreased CO related to HF is maintaining heart rate, blood pressure, respiratory rate, and pulse oximetry within the targeted range.
  • Cardiac arrest: The most reliable sign of an adult or child's cardiac arrest is the absence of a carotid pulse. In an infant, the brachial pulse is assessed.

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Description

Explore the key aspects of heart failure, including its types, pathophysiology, and clinical manifestations. This quiz covers essential concepts related to the progressive nature of heart failure and its impact on health.

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