15 Questions
What are the possible routes of transmission for the microbe causing chronic gastritis and peptic ulcers?
Oral-oral, fecal-oral, environmental
Which virulence factor of the microbe plays a role in its ability to survive in the gastric environment?
Motility via flagella
Which type of inflammatory cells are typically found in the lamina propria in chronic gastritis?
Neutrophils
What is the pattern of gastritis associated with duodenal ulcers?
Antral with high acid production
What is the main risk factor for adenocarcinoma in patients with chronic gastritis?
Intestinal metaplasia
What is a potential cause of acute gastritis based on the provided information?
Increased acid secretion
Which factor is NOT associated with the pathogenesis of acute gastritis?
Increased bicarbonate secretion
What may frequently accompany acute gastritis according to the text?
Peptic ulcer disease
Which of the following is NOT a potential cause of acute gastritis based on the text?
Enhanced blood flow
What is a possible consequence of direct epithelial damage in acute gastritis?
Disruption of adherent mucus layer
What is the pathogenesis of pernicious anemia in autoimmune gastritis?
Intrinsic factor deficiency due to hyperplasia of antral gastrin-producing G cells
Which cell type is primarily targeted by CD4+ T cells in autoimmune gastritis?
Parietal cells
What is a characteristic endoscopic feature of autoimmune gastritis?
Coarser rugae than normal in the affected regions
Which condition is associated with hypergastrinemia as a consequence of autoimmune gastritis?
Peptic ulcer disease
In autoimmune gastritis, what happens due to the lack of intrinsic factor?
Onset of pernicious anemia
Test your knowledge on the pathogenesis of gastric diseases involving CD4+ T cells attacking parietal cells, resulting in lack of gastric acid release and hypergastrinemia. Explore the development of pernicious anemia due to intrinsic factor deficiency. Identify endoscopic features of affected mucosa.
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