G6PD Deficiency and Heart Disorders Quiz

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18 Questions

What is a common symptom of G6PD Deficiency?

Dark colored urine

Which enzyme deficiency can lead to premature destruction of red blood cells?

G6PD Deficiency

Which congenital heart disorder causes left to right shunting of blood?

Atrial Septal Defect

What is a common complication of Ventricular Septal Defect?

Risk for infection

What is a management strategy for individuals with G6PD Deficiency?

Avoiding oxidative stress triggers

What is the main characteristic of Acyanotic Heart Disorders?

Increased pulmonary blood flow

Which symptom is characteristic of St. Vitus Dance (Sydenham's Chorea)?

Involuntary facial grimaces

Which condition presents with mucocutaneous lymph node syndrome and acute systemic vasculitis in children under 5 years old?

Kawasaki Disease

What is the hallmark symptom of Kawasaki Disease?

Irritability

Which treatment option is indicated for acute streptococcal pharyngitis to prevent complications like rheumatic fever?

Antibiotics

Which condition can lead to damage to heart valves and is the most common complication of rheumatic fever?

Acquired heart disease

What is the drug of choice for the prevention of rheumatic heart disease during the acute phase of rheumatic fever?

Penicillin G

What is the major diagnostic criteria for Rheumatic Heart Disease?

Tachycardia out of proportion to fever

Which medication is used for decreasing pulmonary blood flow in congenital heart defects?

Indomethacin

What is the correct management approach for Tetralogy of Fallot?

Blalock-Taussig shunt

What is the primary symptom of Tetralogy of Fallot?

Increasing cyanosis with crying or activity

Which condition has a combination of four congenital heart defects?

Tetralogy of Fallot

What characterizes Acquired Heart Disorders like Rheumatic Heart Disease?

Self-limiting condition

Test your knowledge on G6PD deficiency, its symptoms, complications, and management, along with congenital heart disorders. Learn about the lack of enzyme leading to hemolytic anemia, the effects of oxidative stress on RBCs, and the implications of untreated conditions.

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