Electron Transport Chain and ATP Synthesis Quiz
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Questions and Answers

What is the ultimate 'electron sink' that drives the oxidation reactions in the electron transport chain?

  • FADH+
  • NADH
  • CO2
  • Molecular O2 (correct)

Which organelle is the main site of ATP synthesis?

  • Lysosome
  • Mitochondria (correct)
  • Endoplasmic Reticulum
  • Golgi Apparatus

What is the purpose of the Proton Motive Force (PMF) generated by the electron transport chain?

  • Enhancing the production of CO2
  • Promoting the formation of NADH
  • Facilitating the breakdown of glucose
  • Driving the synthesis of ATP (correct)

Which part of the mitochondria is impermeable to most molecules except those with specific transporters or channels?

<p>Inner membrane (B)</p> Signup and view all the answers

What are the high energy electrons carried by to generate a proton H+ gradient across the inner mitochondrial membrane?

<p>NADH, FADH+ (B)</p> Signup and view all the answers

What is the ultimate function of the Electron Transport Chain (ETC)?

<p>To generate a proton H+ gradient for ATP synthesis (D)</p> Signup and view all the answers

Which organelle contains an outer membrane permeable to most small molecules via porins?

<p>Mitochondria (D)</p> Signup and view all the answers

What is the role of molecular O2 in the electron transport chain?

<p>To act as the ultimate 'electron sink' driving the oxidation reactions (A)</p> Signup and view all the answers

What is the main function of the inner mitochondrial membrane?

<p>To house enzymes and ETC complexes (C)</p> Signup and view all the answers

What drives the synthesis of ATP in the mitochondria?

<p>Proton Motive Force (PMF) (D)</p> Signup and view all the answers

Flashcards

Electron Sink in ETC

Molecular O2 is the final electron acceptor in the electron transport chain (ETC), driving oxidation reactions.

ATP Synthesis Site

Mitochondria is the primary location for ATP production.

PMF Function

The Proton Motive Force (PMF) generated by the ETC drives ATP synthesis.

Inner Mitochondrial Membrane

Highly selective barrier in mitochondria, regulates passage of molecules.

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Electron Carriers in ETC

NADH and FADH2 carry high-energy electrons to the ETC.

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ETC Ultimate Function

The Electron Transport Chain generates a proton gradient to produce ATP.

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Mitochondrial Outer Membrane

Permeable to most small molecules.

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O2's role in ETC

O2 accepts electrons at the end of the ETC, driving the chain.

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Inner Mitochondrial Membrane Composition

Contains enzymes and ETC complexes essential for ATP production.

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ATP synthesis driver

The Proton Motive Force (PMF) powers ATP's synthesis in the mitochondria.

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Study Notes

Electron Transport Chain

  • Oxygen is the ultimate 'electron sink' that drives the oxidation reactions in the electron transport chain.
  • The purpose of the Proton Motive Force (PMF) generated by the electron transport chain is to drive ATP synthesis.

Mitochondria Structure

  • The inner mitochondrial membrane is impermeable to most molecules except those with specific transporters or channels.
  • The outer mitochondrial membrane is permeable to most small molecules via porins.

Electron Transport and ATP Synthesis

  • High-energy electrons are carried by electron carriers (such as NADH and FADH2) to generate a proton (H+) gradient across the inner mitochondrial membrane.
  • The ultimate function of the Electron Transport Chain (ETC) is to generate a proton gradient that drives ATP synthesis.
  • The main function of the inner mitochondrial membrane is to facilitate the electron transport chain and ATP synthesis.
  • The synthesis of ATP in the mitochondria is driven by the proton motive force generated by the electron transport chain.

Oxygen's Role

  • Molecular O2 is the final electron acceptor, which receives the electrons at the end of the electron transport chain.

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Description

Test your knowledge of biochemistry with this quiz on the purpose of the Electron Transport Chain (ETC) in generating ATP. Explore how high-energy electrons drive the synthesis of ATP and the complete oxidation of glucose.

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