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Questions and Answers
What physiological change occurs during alterations of digestive function?
What physiological change occurs during alterations of digestive function?
Which condition is most commonly associated with alterations in digestive function?
Which condition is most commonly associated with alterations in digestive function?
What is a potential consequence of prolonged alterations in digestive function?
What is a potential consequence of prolonged alterations in digestive function?
How do alterations in digestive function impact fluid balance in the body?
How do alterations in digestive function impact fluid balance in the body?
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What role do nurses play in managing patients with alterations of digestive function?
What role do nurses play in managing patients with alterations of digestive function?
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What symptom is commonly experienced by patients with alterations of digestive function?
What symptom is commonly experienced by patients with alterations of digestive function?
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Which of the following can lead to complications in patients with altered digestive function?
Which of the following can lead to complications in patients with altered digestive function?
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How can alterations in digestive function affect metabolism?
How can alterations in digestive function affect metabolism?
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What might be a psychological impact on patients experiencing alterations in digestive function?
What might be a psychological impact on patients experiencing alterations in digestive function?
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What is a possible long-term effect of untreated alterations in digestive function?
What is a possible long-term effect of untreated alterations in digestive function?
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What is a common challenge faced by healthcare professionals when managing patients with alterations of digestive function?
What is a common challenge faced by healthcare professionals when managing patients with alterations of digestive function?
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Which diagnostic test is often utilized to assess alterations in digestive function?
Which diagnostic test is often utilized to assess alterations in digestive function?
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In which way might alterations in digestive function affect a patient's overall health?
In which way might alterations in digestive function affect a patient's overall health?
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Which of the following factors is frequently associated with alterations in digestive function?
Which of the following factors is frequently associated with alterations in digestive function?
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How do health professionals often address the pain associated with alterations in digestive function?
How do health professionals often address the pain associated with alterations in digestive function?
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Which dietary change can be beneficial for patients experiencing alterations in digestive function?
Which dietary change can be beneficial for patients experiencing alterations in digestive function?
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Which of the following symptoms could indicate a severe alteration in digestive function?
Which of the following symptoms could indicate a severe alteration in digestive function?
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What is a common misconception about managing alterations in digestive function?
What is a common misconception about managing alterations in digestive function?
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Which psychological factor can exacerbate alterations in digestive function?
Which psychological factor can exacerbate alterations in digestive function?
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Which nursing intervention is important for patients with altered digestive function?
Which nursing intervention is important for patients with altered digestive function?
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What is a common dietary modification to improve digestive function?
What is a common dietary modification to improve digestive function?
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Which complication may arise from alterations of digestive function over time?
Which complication may arise from alterations of digestive function over time?
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What could be a critical sign indicating severe alterations in digestive function?
What could be a critical sign indicating severe alterations in digestive function?
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Which nursing intervention is essential for managing patients with altered digestive function?
Which nursing intervention is essential for managing patients with altered digestive function?
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Which factor is least likely to be associated with adjustments in digestive function?
Which factor is least likely to be associated with adjustments in digestive function?
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What is a common risk associated with prolonged alterations of digestive function?
What is a common risk associated with prolonged alterations of digestive function?
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Which of the following could be a potential indirect outcome of altered digestive function?
Which of the following could be a potential indirect outcome of altered digestive function?
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What type of dietary intervention is often recommended for patients with alterations in digestive function?
What type of dietary intervention is often recommended for patients with alterations in digestive function?
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How might alterations in digestive function affect dependency on medications?
How might alterations in digestive function affect dependency on medications?
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In patients experiencing alterations in digestive function, which of the following assessments is critical?
In patients experiencing alterations in digestive function, which of the following assessments is critical?
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Study Notes
Alterations of Digestive Function
- Learning Objectives: The learning objectives focus on factors influencing GI disorders, pathophysiology of upper/lower GI and digestive disorders, clinical manifestations, diagnostic tests, and medical treatment for GI tract disorders.
- Complications of GI disorders and nutritional disorders associated with GI function are also key areas of study, along with current evidence regarding obesity development and treatment.
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Clinical Manifestations of GI Dysfunction:
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Anorexia: Lack of desire to eat despite physiological stimuli that normally induce hunger.
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Vomiting: Forceful emptying of stomach and intestinal contents through the mouth. Symptoms include hypersalivation and tachycardia.
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Nausea: Subjective experience associated with various conditions.
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Retching: Non-productive vomiting.
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Projectile Vomiting: Spontaneous vomiting not preceded by nausea or retching.
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Constipation: Infrequent or difficult defecation. Symptoms include straining, hard stools, sensation of incomplete emptying, and fewer than three bowel movements per week.
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Diarrhea: Presence of loose, watery stools (acute or persistent). Major mechanisms include osmotic (laxatives, maldigestion), secretory (bacterial toxins), and motility issues. Systemic effects include dehydration, electrolyte imbalances, and weight loss.
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Abdominal Pain: Pain related to mechanical, inflammatory, or ischemic changes. Types include parietal (lining irritation) and visceral (organ pain).
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Gastrointestinal Bleeding (GIB): Bleeding at any GI tract point; may be visible or occult.
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Upper GI Bleeding: Bleeding from the esophagus, stomach, or duodenum.
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Lower GI Bleeding: Bleeding from the jejunum, ileum, colon, or rectum.
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Dysphagia: Difficulty swallowing related to mechanical obstructions or functional disorders. Symptoms include stabbing pain at the obstruction level, discomfort after swallowing, regurgitation, unpleasant taste, vomiting, aspiration and weight loss. Management involves eating small meals, adding fluids to meals, and raising the head during sleep.
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GERD (Gastroesophageal Reflux Disease): Reflux of acid and pepsin from the stomach to the esophagus. Causes include low resting tone of the lower esophageal sphincter, increased abdominal pressure, and delayed gastric emptying. Symptoms include heartburn, acid regurgitation, dysphagia, chronic cough, asthma attacks, laryngitis, and upper abdominal pain. Treatment includes proton pump inhibitors (PPIs).
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Hiatal Hernia: Diaphragmatic hernia with upper stomach protrusion. Treatment is conservative.
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Gastroparesis: Delayed gastric emptying without mechanical obstruction. Associated with diabetes mellitus, surgical vagotomy, or fundoplication. Symptoms include nausea, vomiting, abdominal pain, and postprandial fullness.
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Pyloric Obstruction: Blocking or narrowing of the stomach-duodenum opening. Symptoms include epigastric pain, fullness, nausea, a succussion splash sound, and vomiting. Prolonged obstruction may cause malnutrition, dehydration, and severe weakness. Treatment is usually conservative management.
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Intestinal Obstruction & Paralytic Ileus: Conditions preventing chyme movement through the intestinal lumen. Simple obstruction is mechanical blockage, while functional obstruction (paralytic ileus) is related to motility failure and can follow surgery, pancreatitis, or hypokalemia. Symptoms include small intestinal: colicky pain, nausea, vomiting, and large intestinal: hypogastric pain and abdominal distension.Treatment is surgical intervention.
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Gastritis: Inflammatory disorder of the gastric mucosa. Types include acute (caused by mucosal barrier injury) and chronic (caused by Helicobacter or immune-related issues). Symptoms are often vague.
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Peptic Ulcer Disease: Ulceration of the lower esophagus, stomach, or duodenum. Common causes include H. pylori infection, hypersecretion of stomach acid and pepsin, and NSAID use. Symptoms include intermittent epigastric pain that is often relieved by food or antacids. Treatment aims to relieve hyperacidity and potentially includes surgical options.
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Surgical Treatment for Ulcers: Surgical intervention is commonly used for recurrent or uncontrolled bleeding, and perforation (hole) in the stomach or duodenum. Objectives include reducing acid secretion stimuli, decreasing the acid-producing cells in the stomach, and correcting ulcer-related complications.
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Post-Gastrectomy Syndromes: A range of symptoms following gastric surgery, including dumping syndrome, alkaline reflux gastritis, afferent loop obstruction, diarrhea, weight loss, anemia, and bone/mineral disorders.
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Malabsorption Syndromes: Breakdown of chemical digestion processes or intestinal mucosal failure to absorb nutrients. Maldigestion and malabsorption often overlap.
- Pancreatic Exocrine Insufficiency: Insufficient pancreatic enzyme production. Causes include pancreatitis, pancreatic cancer, resection, and cystic fibrosis. Symptoms include fatty stools and weight loss.
- Lactase Deficiency: Inability to break down lactose. Results in lactose fermentation by bacteria, leading to flatulence, cramping pain, and osmotic diarrhea.
- Bile Salt Deficiency: Typically associated with liver disease or bile obstructions. Symptoms include fatty stools (steatorrhea), diarrhea, and loss of fat-soluble vitamins (A, D, E, K).
- Fat-Soluble Vitamin Deficiencies: Malabsorption can cause deficiencies in fat-soluble vitamins (A, D, K, and E), resulting in symptoms like night blindness, bone pain, osteoporosis, fractures, blood clotting issues, and petechiae (small reddish spots on the skin).
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Inflammatory Bowel Diseases (IBDs): Chronic inflammatory conditions such as ulcerative colitis (UC) and Crohn's disease (CD). IBDs involve multiple causes, including genetics, environmental factors, alterations in epithelial barrier functions, and altered immune reactions to intestinal flora.
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Ulcerative Colitis: Chronic inflammatory disease affecting the colon mucosa, often starting in the sigmoid colon and rectum, potentially extending to the entire colon. Symptoms include diarrhea (10-20 stools daily), urgency, bloody stools, and cramping. Mild to moderate disease is treated initially with 5-aminosalicyclates and potentially steroids. Severe disease may require immunomodulators or surgery.
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Crohn's Disease: Inflammatory condition that affects any part of the digestive tract, from mouth to anus. Characterized by "skip lesions", fissures into lymphatics, anemia due to malabsorption of B12 and folic acid. Symptoms and treatment are similar to ulcerative colitis.
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Irritable Bowel Syndrome (IBS): Symptoms include abdominal pain, and changes in bowel habits; common in females and associated with anxiety, depression, and reduced quality of life. Characterized by unknown causes (mechanisms proposed include visceral hypersensitivity, intestinal permeability, motility, microbiota, food allergies, and psychosocial factors). No cure, treatment is individualized.
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Diverticular Disease:
- Diverticula: Mucosal herniations through the colon wall, frequently in the sigmoid colon.
- Diverticulosis: Asymptomatic condition of diverticula presence.
- Diverticulitis: Inflammation of diverticula; complications may include abscesses, fistulas, obstructions, bleeding, and perforation. Symptoms of uncomplicated diverticular disease can be vague or absent.
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Appendicitis: Inflammation of the appendix. Common causes are obstruction, foreign bodies, and infections. Symptoms include abdominal pain that may initially appear periumbilical before localizing to the right lower quadrant, rebound tenderness, and possible fever/leukocytosis. Perforation, peritonitis, and abscesses are significant complications.
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Obesity: Increased body fat mass with a BMI >30 kg/m^2. It results from caloric intake exceeding expenditure There are alterations in interaction between peripheral and central pathways, along with multiple cytokines, hormones, and neurotransmitters. These interactions usually lead to signals at the hypothalamus and brainstem to determine hunger and satiety (control).
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Visceral Obesity: Body fat localized around the abdomen/upper body (Apple shape).
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Peripheral Obesity: Body fat around the thighs/buttocks (Pear shape).
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Normal Weight Obesity (NWO): Normal weight with body fat over 30%.
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Metabolically Healthy Obesity (MHO): Obese however with no typical metabolic issues. Weight loss surgery is the most substantial treatment.
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Malnutrition and Starvation: Malnutrition is lack of nutrition, including calories, protein, vitamins, and minerals. Starvation is decreased energy intake that leads to weight loss (cachexia).
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Liver Disorders:
- Portal Hypertension: High blood pressure in the portal vein system, often due to resistance.
- Varices: Enlarged and swollen veins, typically in the lower esophagus, stomach, abdominal wall, and rectum.
- Splenomegaly: Enlarged spleen due to blood pooling in portal veins.
- Ascites: Fluid accumulation in the peritoneal cavity, commonly caused by cirrhosis.
- Hepatic Encephalopathy: Neurological syndrome of impaired behavior and cognitive functions.
- Jaundice (Icterus): Yellow skin discoloration due to elevated bilirubin. Types include obstructive (extrahepatic or intrahepatic), and prehepatic (hemolytic) jaundice.
- Acute Liver Failure: Severe or necrotic liver cells without pre-existing liver disease or cirrhosis.
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Cirrhosis: Irreversible inflammatory condition and fibrosis of the liver; often caused by longstanding issues such as chronic alcohol abuse.
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Alcoholic Liver Disease: Liver damage related to alcohol toxicity; includes alcoholic fatty liver, alcoholic steatohepatitis, and alcoholic cirrhosis. Characterized by progressive liver damage.
- Nonalcoholic Fatty Liver Disease (NAFLD) / Nonalcoholic Steatohepatitis (NASH): Fat deposits in the liver, with inflammation in NASH cases. This can progress to liver cirrhosis and failure.
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Viral Hepatitis: Systemic viral infection affecting the liver; types A, B, C, D, and E exist as different etiologies and infections.
- Five types (A, B, C, D, E) and can range from asymptomatic to severe hepatitis with rapid onset of liver complications
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Disorders of the Gallbladder:
- Cholelithiasis: Gallstone formation. Increased risks are obesity, middle age, females, oral contraceptives, first nations ancestry, and genetic predispositions.
- Cholecystitis: Inflammation of the gallbladder often due to gallstones lodgement in cystic ducts. Symptoms include fever/leukocytosis, rebound tenderness, abdominal muscle guarding, and possible jaundice.
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Disorders of the Pancreas:
- Pancreatitis: Inflammation of the pancreas. Causes include blockage of pancreatic duct outflow by gallstones or pancreatic duct obstruction. Types include acute (usually resolves) and chronic (progressive fibrosis, destruction, and commonly linked to alcohol overuse). Continuous or intermittent abdominal pain with weight loss are common symptoms. A major concern related to pancreatitis is pancreatic cancer risk.
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Developmental Alterations of Digestive Function
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Cleft Lip and Cleft Palate: Congenital disruptions in facial development. Cleft Lip is formation issues occurring during embryonic development as one or both sides of the lip don't fully fuse. Cleft Palate issues often occurring during embryonic formation and the palate segment tissues don't completely fuse. - Nonsyndromic Cleft Lip and/or Palate: These cases aren't connected closely to major health issues.
- Evaluation and Treatment: Includes ultrasounds, postnatal imaging, surgical correction, speech, prosthetics and orthodontics follow-up.
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Infantile Hypertrophic Pyloric Stenosis: Narrowing of the pyloric sphincter in infants; likely genetic and environmental factors are to blame. Non-bilious vomiting commonly emerges between 2-8 weeks after birth with weight loss, dehydration, irritability, and possible electrolyte imbalance, often noted within the right upper quadrant. Management is usually surgical (pyloromyotomy).
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Malrotation: Small bowel problems, often needing surgical intervention because of twisting or improper positioning of the intestinal segments. Symptoms are intermittent/persistent bile-stained vomiting and include dehydration, electrolyte imbalance, pain, reduced stool output, or possibly bloody stools. Diagnostic approach is clinically based and often also requires abdominal x-rays.
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Meconium Syndromes:
- Meconium Ileus: Meconium plugging the ileum; caused by abnormal mucus. Obstruction may necessitate surgical procedures. Symptoms are abdominal distension, vomiting, and abdominal pain. Intestinal lavage and laxatives are often part of treatment.
- Meconium Plug Syndrome/Meconium Disease: Characterized by delayed/difficult meconium passage, frequently in premature/low birth weight infants. This condition presents with delayed passage of meconium, intestinal distension and in severe cases, it can be a surgical emergency.
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Hirschsprung's Disease (Aganglionic Megacolon): Lack of nerve cells in the colon causes impaired bowel function and distention. Characterized by chronic constipation; and can lead to complications like diarrhea, enterocolitis, sepsis, and potentially death. Treatment is surgical.
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Gastroesophageal Reflux: Dilation of the esophagus and stomach contents reflux into esophagus in infants. Often a minor component of normal infant development. Symptoms in newborns may include frequent vomiting, unexplained crying, choking and can be linked to sudden infant death.
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Intussusception: Telescoping of a proximal intestine segment into a distal segment. A common scenario is the ileum telescoping into the cecum and ascending colon, causing blockage. Intussusception can lead to dangerous complications if untreated (ischemia and necrosis). Reduction (with enema or surgery) is the core management strategy.
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Celiac Disease: Gluten intolerance; symptoms vary from mild to severe diarrhea and dehydration, malabsorption, and protein loss. Diagnosis uses serological autoantibodies measurements; Gluten-free diet for life with vitamin D, iron, and folic acid supplements are the typical treatment.
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Failure to Thrive (FTT): Characterized by inadequate physical development in infants or children. Causes are multifactorial; including biological, psychosocial, and environmental factors. Specific indicators include slower gaining weight, height, and head circumference ratio, compared to usual benchmarks or expected weight/height.
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Diarrhea: Prolonged diarrhea is dangerous in children due to fluid reserve limitations.
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Primary Lactose Intolerance: Inability to digest lactose due to inadequate lactase production. Symptoms often include osmotic diarrhea, abdominal pain, bloating, and flatulence.
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Neonatal Jaundice: High bilirubin in the blood. Physiological jaundice is common in healthy newborns; caused by immaturity and resolving without intervention in most cases. Pathological jaundice is more serious and linked to factors like maternal-fetal blood type incompatibility, premature birth, exclusive breast feeding, maternal age, gender, delayed meconium passage (difficulty in passing newborn stool), and excessive birth trauma.
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- Hepatitis: Hepatitis A, B, C, D, and E; range from mild to severe; some can occur as newborns and some can be passed from a mother; chronic hepatitis is possible.
- Chapter 37 (is likely the specific introductory chapter)
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Description
This quiz explores key concepts related to alterations in digestive function and their physiological effects. It addresses common conditions, potential consequences, fluid balance implications, and the nursing role in management. Test your knowledge on this important aspect of healthcare.