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What leads to a significant reduction in the permeability of sclerotic dentin compared to normal dentin?
What leads to a significant reduction in the permeability of sclerotic dentin compared to normal dentin?
What is a consequence of intense caries activity in dentin?
What is a consequence of intense caries activity in dentin?
What can lead to the formation of replacement odontoblasts?
What can lead to the formation of replacement odontoblasts?
What is the role of reparative dentin in response to dental caries?
What is the role of reparative dentin in response to dental caries?
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What can result from acute, rapidly advancing caries?
What can result from acute, rapidly advancing caries?
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What characterizes infected dentin?
What characterizes infected dentin?
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Which type of dentin responds to long-term, low-level acid demineralization?
Which type of dentin responds to long-term, low-level acid demineralization?
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Which color of disclosing dyes can indicate affected dentin?
Which color of disclosing dyes can indicate affected dentin?
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What is sclerotic dentin known for?
What is sclerotic dentin known for?
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What response can a vital pulp initiate to counter slow caries progression?
What response can a vital pulp initiate to counter slow caries progression?
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Which of the following describes the appearance of infected dentin during visual inspection?
Which of the following describes the appearance of infected dentin during visual inspection?
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What indicates a reaction to severe, rapidly advancing caries?
What indicates a reaction to severe, rapidly advancing caries?
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Which layer of dentin is marked by the highest level of bacterial invasion?
Which layer of dentin is marked by the highest level of bacterial invasion?
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What primarily contributes to the susceptibility of pits and fissures to caries?
What primarily contributes to the susceptibility of pits and fissures to caries?
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What happens to enamel caries when it reaches the dentinoenamel junction (DEJ)?
What happens to enamel caries when it reaches the dentinoenamel junction (DEJ)?
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What is the first clinical site for caries initiation mentioned?
What is the first clinical site for caries initiation mentioned?
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Why is clinical diagnosis of pit-and-fissure caries often challenging?
Why is clinical diagnosis of pit-and-fissure caries often challenging?
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What type of bacteria is primarily found in carious pits and fissures?
What type of bacteria is primarily found in carious pits and fissures?
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What is the geometric shape of a pit-and-fissure caries lesion in cross-section?
What is the geometric shape of a pit-and-fissure caries lesion in cross-section?
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What characterizes the environment within a carious lesion?
What characterizes the environment within a carious lesion?
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Which type of caries initiation site is described as having smooth enamel surfaces that shelter biofilm?
Which type of caries initiation site is described as having smooth enamel surfaces that shelter biofilm?
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What is the main driving force for the remineralization process?
What is the main driving force for the remineralization process?
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What characteristic is observed in remineralized lesions commonly described as arrested caries?
What characteristic is observed in remineralized lesions commonly described as arrested caries?
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When should discolored, remineralized areas not be restored?
When should discolored, remineralized areas not be restored?
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Which zone is identified as the advancing edge of the enamel lesion?
Which zone is identified as the advancing edge of the enamel lesion?
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What is the appearance of non-cavitated enamel caries lesions on dry teeth?
What is the appearance of non-cavitated enamel caries lesions on dry teeth?
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How many zones have been identified in the microscopic features of non-cavitated enamel?
How many zones have been identified in the microscopic features of non-cavitated enamel?
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In which part of the enamel caries lesion is subsurface porosity primarily caused by demineralization?
In which part of the enamel caries lesion is subsurface porosity primarily caused by demineralization?
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What indicates that an area of enamel is demineralized?
What indicates that an area of enamel is demineralized?
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What shape do smooth-surface lesions typically present in cross-section?
What shape do smooth-surface lesions typically present in cross-section?
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Which of the following areas is most susceptible to the development of cariogenic biofilms?
Which of the following areas is most susceptible to the development of cariogenic biofilms?
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What is the estimated time for the progression from non-cavitated enamel caries to cavitation?
What is the estimated time for the progression from non-cavitated enamel caries to cavitation?
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How do root surface caries lesions typically appear in terms of shape?
How do root surface caries lesions typically appear in terms of shape?
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What characteristic is observed in more advanced enamel lesions?
What characteristic is observed in more advanced enamel lesions?
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What factor contributes to the rapid progression of root surface caries?
What factor contributes to the rapid progression of root surface caries?
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What sign indicates active caries in affected enamel?
What sign indicates active caries in affected enamel?
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Which surface of the teeth provides a less favorable site for cariogenic biofilm attachment?
Which surface of the teeth provides a less favorable site for cariogenic biofilm attachment?
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What is the primary characteristic of the body of the lesion in terms of mineralization?
What is the primary characteristic of the body of the lesion in terms of mineralization?
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Which zone is described as almost unaffected and remains heavily mineralized?
Which zone is described as almost unaffected and remains heavily mineralized?
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What is the shape of dentinal caries in cross-section?
What is the shape of dentinal caries in cross-section?
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What characterizes infected dentin in relation to dental lesions?
What characterizes infected dentin in relation to dental lesions?
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Which zone is also referred to as inner carious dentin?
Which zone is also referred to as inner carious dentin?
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What role do tubules in dentin play concerning bacterial infection?
What role do tubules in dentin play concerning bacterial infection?
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Which of the following statements regarding affected dentin is correct?
Which of the following statements regarding affected dentin is correct?
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What is true concerning the Zone 1 in carious dentin?
What is true concerning the Zone 1 in carious dentin?
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Study Notes
Dental Cariology
- Dental caries is the result of disequilibrium between demineralization and remineralization.
- Cavitated tooth surfaces provide more retention for biofilm, accelerating tooth structure destruction.
- When enamel caries reaches the dentin-enamel junction (DEJ), rapid lateral expansion occurs due to dentin's lower acid resistance.
- This sheltered, acidic, and anaerobic environment creates an ideal environment for cariogenic bacteria.
Clinical Sites for Caries Initiation
- Pits and fissures of enamel are the most susceptible areas.
- Smooth enamel surfaces harboring cariogenic biofilm are another common initiation site.
- Root surfaces are also susceptible due to thin cementum and lack of enamel protection.
Pits and Fissures of Enamel
- Bacteria rapidly colonize pits and fissures of newly erupted teeth.
- S. sanguis is prevalent in healthy pits and fissures, while Mutans Streptococci are more common in carious areas.
- The long, narrow shape of pits and fissures hinders biofilm removal, increasing susceptibility to caries.
- Pit-and-fissure caries expands as it penetrates enamel, making the initial lesion appear smaller than the actual extent of the carious lesion.
Smooth Enamel Surfaces
- Cariogenic biofilm generally develops near the gingival or under proximal contacts.
- Smooth surfaces are less favorable for biofilm attachment than pits and fissures.
- Lesions on smooth surfaces show a V-shape cross-section, with the apex towards the dentin-enamel junction (DEJ).
Root Surface Caries
- Root surfaces, being rougher than enamel, readily allow cariogenic biofilm formation, especially in the absence of good oral hygiene.
- Cementum, the covering of root surfaces, is thin and provides minimal resistance to caries.
- Root caries lesions have irregular margins, tend to be U-shaped in cross-section, and progress rapidly due to the lack of enamel protection.
Progression of Enamel Caries
- The time for progression from non-cavitated enamel caries to cavitation on smooth surfaces is approximately 18 months, plus or minus 6 months.
- Stages include white spot lesions, followed by a softened, chalky enamel that chips away with an explorer, and, finally, cavitation.
- Remineralized (arrested) caries lesions are discolored (usually brown or black) but remain intact and are more resistant to future caries attack.
Classification of Dental Caries
- Classification of dental caries is based on the clinical characteristics of normal and altered enamel. Key characteristics used in classification include tooth hydration level, desiccation level, surface texture, and surface hardness.
- Different stages are related to varying degrees of demineralization, with descriptions including "hydrating," "desiccated," "smooth," and "roughened," to "hard," "very soft," and "hard."
Microscopic Features of Non-Cavitated Enamel
- Four zones are identified pre-cavitation: translucent zone (advancing edge), dark zone, body (greatest demineralization), and surface zone (least affected).
Microscopic Features of Non-Cavitated Dentin
- Three zones are distinguishable in carious dentin in slowly advancing lesions: normal (smooth, bacteria free), affected (demineralization of intertubular dentin), and infected (outermost carious layer, bacterial invasion).
- The "affected" dentin can be further sub-categorized as subtransparent, transparent, or turbid.
2- Dentin Caries
- Dentin contains less mineral and has microscopic tubules allowing bacterial ingress/egress of mineral.
- Caries spreads laterally along the dentin-enamel junction (DEJ).
- Dentinal caries is V-shaped, with a wide base at the DEJ and a narrow apex towards the pulp.
- Caries in dentin advances faster than in enamel due to dentin's lower resistance to acid attack.
- Tubules in the infected dentin of dentin lesions provide an easy pathway for bacteria.
- Dentin reacts to caries in three ways: short-term, low-level acid demineralization, moderate-intensity and high-intensity acid demineralization.
- In slowly advancing caries, a vital pulp can repair demineralized dentin via remineralizing intertubular dentin and apposition of the peritubular dentin.
- Sclerotic dentin has a higher mineral content, appearing shiny and darker, but is harder against the explorer and seals off lesions by preventing the passage of bacteria into tubules.
- More intense caries activity results in significant inflammation of the pulp, causing regeneration of replacement (secondary) odontoblasts.
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Description
Test your knowledge on dental caries and their initiation sites with this quiz. Explore how demineralization and remineralization affect tooth structure, and identify where caries commonly begin. Challenge yourself to understand the relationship between bacteria and tooth decay.