Classical and Polymicrobial Infections

A multi-species microbial community enclosed within a matrix that adheres to biotic and abiotic surfaces.

The process by which a mature multi-species biofilm develops from a single primary colonizer.

The process by which bacteria, specifically primary colonizers, interact with the conditioning film on a surface during biofilm development.

A single organism causing an infection.

The mechanism by which bacteria adhere to lung tissue in secondary infections.

An infection caused by a single organism.

A viral infection that triggers secondary infections.

The final stage of a classical infection, where the organism releases protein toxins.

An infection caused by multiple different microorganisms.

The process by which bacteria secrete signaling molecules to communicate with each other.

The ability of genetically distinct bacteria to adhere to each other.

The proteins on bacterial surfaces that help them attach to other bacteria.

A combination of Candida albicans and S. aureus causing denture stomatitis.

The molecule that inhibits filamentation of Candida albicans.

A group of microbes working together to acquire nutrients through a complex food web.

The ability of microbes to survive in harsh environments by forming polymicrobial communities.

The minimum number of species required for polymicrobial co-operation.

The limitation of 16S rRNA Next-Generation Sequencing because it can only identify the genus, not the species of bacteria.

The difficulty in predicting treatment outcomes for polymicrobial infections due to complex interactions and virulence factors.

The primary consequence of an overactive immune system in response to a viral infection.

The reason antibiotics are not used to treat viral infections.

The primary component of enamel that starts the development of plaque biofilm.

The benefits of bacteria growing in biofilms.

The primary consequence of changes in biofilm composition.

The process by which subtle changes in microbiota lead to inflammation in the gums, eventually progressing into periodontitis.

The primary consequence of high blood sugar in diabetic patients, affecting both blood flow and inflammation.