Podcast
Questions and Answers
Which cell type is the dominant cell type activated in chronic inflammation?
Which cell type is the dominant cell type activated in chronic inflammation?
- Macrophages (correct)
- Eosinophils
- Lymphocytes
- Mast cells
What is a characteristic feature of granuloma formation?
What is a characteristic feature of granuloma formation?
- Absence of lymphocytes
- Contains the injurious agent and attempts to eradicate it (correct)
- Presence of neutrophils
- Proliferation of fibroblasts
What type of granuloma is typically seen with tuberculosis (TB)?
What type of granuloma is typically seen with tuberculosis (TB)?
- Non-caseating granuloma
- Caseating granuloma (correct)
- Necrotizing granuloma
- Angiogenic granuloma
Which of the following is NOT a systemic sign associated with chronic inflammation?
Which of the following is NOT a systemic sign associated with chronic inflammation?
What is the main function of plasma cells in chronic inflammation?
What is the main function of plasma cells in chronic inflammation?
Which of the following is a feature of chronic inflammation that distinguishes it from acute inflammation?
Which of the following is a feature of chronic inflammation that distinguishes it from acute inflammation?
Which of the following best describes chronic inflammation?
Which of the following best describes chronic inflammation?
What are the main causes of chronic inflammation according to the text?
What are the main causes of chronic inflammation according to the text?
Which cells are involved in the infiltration during chronic inflammation?
Which cells are involved in the infiltration during chronic inflammation?
What is a key feature of the morphology of chronic inflammation?
What is a key feature of the morphology of chronic inflammation?
How does chronic inflammation differ from acute inflammation?
How does chronic inflammation differ from acute inflammation?
What are some systemic effects of chronic inflammation according to the text?
What are some systemic effects of chronic inflammation according to the text?
Chronic inflammation is always preceded by acute inflammation.
Chronic inflammation is always preceded by acute inflammation.
The morphology of chronic inflammation involves infiltration of eosinophils and neutrophils.
The morphology of chronic inflammation involves infiltration of eosinophils and neutrophils.
Chronic inflammation lasts for a short duration, typically days to weeks.
Chronic inflammation lasts for a short duration, typically days to weeks.
Angiogenesis and fibrosis are key features of the morphology of acute inflammation.
Angiogenesis and fibrosis are key features of the morphology of acute inflammation.
Chronic inflammation can only occur due to persistent infection by microorganisms.
Chronic inflammation can only occur due to persistent infection by microorganisms.
Chronic inflammation is always characterized by the 5 classic signs of acute inflammation.
Chronic inflammation is always characterized by the 5 classic signs of acute inflammation.
Eosinophils are the dominant cell type activated in chronic inflammation.
Eosinophils are the dominant cell type activated in chronic inflammation.
Granuloma formation can only be caused by infectious organisms such as TB and leprosy.
Granuloma formation can only be caused by infectious organisms such as TB and leprosy.
During chronic inflammation, the main function of lymphocytes is to directly kill injurious agents.
During chronic inflammation, the main function of lymphocytes is to directly kill injurious agents.
Tissue necrosis is not associated with chronic inflammation.
Tissue necrosis is not associated with chronic inflammation.
Angiogenesis is a common change observed in acute inflammation.
Angiogenesis is a common change observed in acute inflammation.
Macrophage-lymphocyte interactions are not involved in granuloma formation in chronic inflammation.
Macrophage-lymphocyte interactions are not involved in granuloma formation in chronic inflammation.
Chronic inflammation is always characterized by the 5 classic signs of acute inflammation.
Chronic inflammation is always characterized by the 5 classic signs of acute inflammation.
Granuloma formation can only be caused by infectious organisms such as TB and leprosy.
Granuloma formation can only be caused by infectious organisms such as TB and leprosy.
Macrophage-lymphocyte interactions are not involved in granuloma formation in chronic inflammation.
Macrophage-lymphocyte interactions are not involved in granuloma formation in chronic inflammation.
Angiogenesis is a common change observed in acute inflammation.
Angiogenesis is a common change observed in acute inflammation.
During chronic inflammation, the main function of lymphocytes is to directly kill injurious agents.
During chronic inflammation, the main function of lymphocytes is to directly kill injurious agents.
The morphology of chronic inflammation involves infiltration of eosinophils and neutrophils.
The morphology of chronic inflammation involves infiltration of eosinophils and neutrophils.
Macrophages are not the dominant cell type activated in chronic inflammation.
Macrophages are not the dominant cell type activated in chronic inflammation.
Granuloma formation is only associated with infectious organisms and not with autoimmune conditions.
Granuloma formation is only associated with infectious organisms and not with autoimmune conditions.
Eosinophils play a minor role in chronic inflammation compared to neutrophils.
Eosinophils play a minor role in chronic inflammation compared to neutrophils.
Tissue necrosis is not a common feature associated with chronic inflammation.
Tissue necrosis is not a common feature associated with chronic inflammation.
Angiogenesis is not observed as a change in chronic inflammation.
Angiogenesis is not observed as a change in chronic inflammation.
Plasma cells in chronic inflammation mainly act through direct killing of injurious agents.
Plasma cells in chronic inflammation mainly act through direct killing of injurious agents.
Study Notes
- Chronic inflammation is a condition where inflammation persists for months or years, either following acute inflammation or without it.
- Unlike acute inflammation, chronic inflammation is not characterized by the five signs of inflammation: redness, heat, swelling, pain, and loss of function.
- Chronic inflammation is marked by the infiltration of mononuclear cells, including macrophages, lymphocytes, plasma cells, and other immune cells, into the affected tissue.
- Macrophages are the dominant cell type in chronic inflammation, producing substances that can cause tissue damage and fibrosis.
- Lymphocytes produce cytokines and contribute to the immune response in chronic inflammation.
- In chronic inflammation, fibroblasts proliferate and cause fibrosis, leading to structural destruction and replacement of damaged tissue.
- Granulomas, a special type of chronic inflammation, form in response to persistent inflammatory stimuli and can contain the injurious agent and attempt to eradicate it.
- Granulomas can be caused by persistent infections, such as tuberculosis, leprosy, and syphilis, or exposure to exogenous materials like asbestos and silica.
- Chronic inflammation is associated with systemic signs, including low-grade fever, weight loss, and anemia.
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Test your knowledge on the concept and causes of chronic inflammation, processes involved, morphology, systemic effects, and main outcomes. Learn about chronic inflammation and its impact on health.
