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Questions and Answers
What are the direct detrimental effects of high levels of angiotensin II and aldosterone on cardiac muscle?
What are the direct detrimental effects of high levels of angiotensin II and aldosterone on cardiac muscle?
- Favor remodeling, fibrosis, and inflammatory changes (correct)
- Promote vasodilation and reduce natriuresis
- Inhibit renin and aldosterone release
- Increase myocardial contractility
Which natriuretic peptide is considered most important in heart failure?
Which natriuretic peptide is considered most important in heart failure?
- B-type natriuretic peptide (correct)
- D-type natriuretic peptide
- Atrial natriuretic peptide
- C-type natriuretic peptide
What does excessive elongation of heart muscle fibers lead to?
What does excessive elongation of heart muscle fibers lead to?
- Improved cardiac function
- Increased natriuresis
- Weaker contractions and diminished ejection ability (correct)
- Stronger contractions
Which term is used to describe the failure that occurs when the ventricle is unable to effectively pump blood?
Which term is used to describe the failure that occurs when the ventricle is unable to effectively pump blood?
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What is a characteristic of diastolic dysfunction in patients with heart failure?
What is a characteristic of diastolic dysfunction in patients with heart failure?
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Which response is activated by natriuretic peptides in heart failure?
Which response is activated by natriuretic peptides in heart failure?
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What role do atrial and B-type natriuretic peptides play in heart failure?
What role do atrial and B-type natriuretic peptides play in heart failure?
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