Podcast
Questions and Answers
During isovolumetric contraction and relaxation, what happens to the volume of blood in the ventricles?
During isovolumetric contraction and relaxation, what happens to the volume of blood in the ventricles?
- It increases due to ventricles contracting and building up pressure
- It fluctuates with the opening and closing of the AV and SL valves
- It does not change as all 4 valves are closed (correct)
- It decreases as the ventricles are relaxing and pressure falls
What is the term for the volume left in the ventricles at the end of the contraction?
What is the term for the volume left in the ventricles at the end of the contraction?
- End-Systolic Volume (ESV) (correct)
- End-Diastolic Volume (EDV)
- Cardiac Output (CO)
- Stroke Volume (SV)
What is the term for the volume ejected from the ventricles every beat?
What is the term for the volume ejected from the ventricles every beat?
- Cardiac Output (CO)
- End-Diastolic Volume (EDV)
- End-Systolic Volume (ESV)
- Stroke Volume (SV) (correct)
What is the formula for cardiac output (CO)?
What is the formula for cardiac output (CO)?
During isovolumetric relaxation, what happens to the ventricular pressure and the status of the valves?
During isovolumetric relaxation, what happens to the ventricular pressure and the status of the valves?
Which factor determines the resistance the heart has to pump against?
Which factor determines the resistance the heart has to pump against?
According to the Frank-Starling law, an increase in preload should result in:
According to the Frank-Starling law, an increase in preload should result in:
Which of the following is considered a negative inotrope?
Which of the following is considered a negative inotrope?
What is the primary determinant of cardiac output (CO) at rest?
What is the primary determinant of cardiac output (CO) at rest?
What causes the dicrotic notch in the arterial pressure waveform?
What causes the dicrotic notch in the arterial pressure waveform?
Which of the following is true about atrial systole?
Which of the following is true about atrial systole?
What does the term 'isovolumetric' mean in the context of ventricular contraction and relaxation?
What does the term 'isovolumetric' mean in the context of ventricular contraction and relaxation?
What is the primary determinant of afterload?
What is the primary determinant of afterload?
In the context of cardiac function, what does the term 'preload' refer to?
In the context of cardiac function, what does the term 'preload' refer to?
What is the formula for calculating stroke volume (SV)?
What is the formula for calculating stroke volume (SV)?
What is the primary impact of hypertension on the heart?
What is the primary impact of hypertension on the heart?
When does the majority of blood flow to the heart occur?
When does the majority of blood flow to the heart occur?
What is the impact of right coronary artery blockage?
What is the impact of right coronary artery blockage?
Which medication can influence the plateau phase of the action potential in cardiac muscle?
Which medication can influence the plateau phase of the action potential in cardiac muscle?
What is the role of troponin in cardiac physiology?
What is the role of troponin in cardiac physiology?
What is the function of the SA node in the heart?
What is the function of the SA node in the heart?
How does epinephrine (Epi) affect heart rate?
How does epinephrine (Epi) affect heart rate?
What is the primary impact of exercise on cardiac physiology?
What is the primary impact of exercise on cardiac physiology?
Through which vessel does venous circulation return to the heart?
Through which vessel does venous circulation return to the heart?
What is the impact of coronary circulation on the heart muscle?
What is the impact of coronary circulation on the heart muscle?
When is coronary blood flow fastest?
When is coronary blood flow fastest?
What is unique about the cellular structure of cardiac muscle?
What is unique about the cellular structure of cardiac muscle?
Study Notes
Cardiac Physiology Key Points
- Hypertension increases afterload and resistance, making the heart work harder
- Exercise improves cardiac output and oxygen utilization
- Coronary circulation is vital for providing nutrients and removing waste from the heart muscle
- Majority of blood flow to the heart occurs during the diastolic phase
- Coronary blood flow is fastest when the heart is contracting
- Right coronary artery blockage can impact pacemaker function
- Venous circulation returns to the heart through the coronary sinus
- Cardiac muscle has a unique cellular structure with intercalated discs and a high mitochondrial content
- Troponin is a cardiac marker for MI and is released into circulation in case of muscle damage
- The heart has its own pacemaker, the SA node, with an unstable resting membrane potential
- Epi increases heart rate by increasing the slope of action potentials
- Medications like CCB, BB, and digoxin can influence the plateau phase of the action potential in cardiac muscle
Studying That Suits You
Use AI to generate personalized quizzes and flashcards to suit your learning preferences.
Related Documents
Description
Test your knowledge of cardiac physiology with this quiz. Explore key points such as the impact of hypertension, the role of exercise in cardiac output, coronary circulation, cardiac markers for MI, and the influence of medications on the action potential in cardiac muscle.
