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Questions and Answers
What condition results from the abnormal production of glucose during burns?
What condition results from the abnormal production of glucose during burns?
Why is oxygen important for maintaining consciousness in patients?
Why is oxygen important for maintaining consciousness in patients?
What is typically involved in the assessment of a burn patient’s condition?
What is typically involved in the assessment of a burn patient’s condition?
What should be considered for monitoring in burn patients?
What should be considered for monitoring in burn patients?
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What is a consequence of decreased glucose uptake in burn patients?
What is a consequence of decreased glucose uptake in burn patients?
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What is the primary cause of burns?
What is the primary cause of burns?
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Which layer of skin is involved in thermoregulation?
Which layer of skin is involved in thermoregulation?
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What characterizes a 2nd degree superficial dermal burn?
What characterizes a 2nd degree superficial dermal burn?
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What is the zone of coagulation in burn injuries?
What is the zone of coagulation in burn injuries?
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What does the zone of stasis indicate after a burn?
What does the zone of stasis indicate after a burn?
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Which degree of burn is characterized by complete destruction of the epidermis and dermis?
Which degree of burn is characterized by complete destruction of the epidermis and dermis?
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What happens to nerves in a 3rd degree burn?
What happens to nerves in a 3rd degree burn?
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How does the epidermis respond to injury from burns?
How does the epidermis respond to injury from burns?
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What procedure should be considered when a patient presents with stridor, wheeze, and low oxygen saturation levels?
What procedure should be considered when a patient presents with stridor, wheeze, and low oxygen saturation levels?
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How does a 3rd degree burn around the chest affect breathing?
How does a 3rd degree burn around the chest affect breathing?
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What is the primary trigger for a patient to breathe when they are experiencing dyspnoea due to poor gas exchange?
What is the primary trigger for a patient to breathe when they are experiencing dyspnoea due to poor gas exchange?
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In the context of burns, what is the role of cytokines and inflammatory mediators on circulation?
In the context of burns, what is the role of cytokines and inflammatory mediators on circulation?
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What is a significant consequence of losing the dermis layer in burn patients?
What is a significant consequence of losing the dermis layer in burn patients?
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What is the recommended room temperature for a patient with a significant burn injury?
What is the recommended room temperature for a patient with a significant burn injury?
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What does the lethal triad of hypothermia, coagulation, and metabolic acidosis signify for burn patients?
What does the lethal triad of hypothermia, coagulation, and metabolic acidosis signify for burn patients?
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What should be included in the management of a burn patient besides fluid replacement?
What should be included in the management of a burn patient besides fluid replacement?
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What is the main aim of burns resuscitation?
What is the main aim of burns resuscitation?
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What effect do burns have on organ perfusion?
What effect do burns have on organ perfusion?
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What can convert the zone of stasis into an area of complete tissue loss?
What can convert the zone of stasis into an area of complete tissue loss?
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What condition can occur in severe burns affecting the lungs?
What condition can occur in severe burns affecting the lungs?
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In the zone of hyperaemia, what is the characteristic of blood flow?
In the zone of hyperaemia, what is the characteristic of blood flow?
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What systemic effect is observed when the burn reaches 30% of total body surface area?
What systemic effect is observed when the burn reaches 30% of total body surface area?
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How much can the basal metabolic rate increase in burn patients?
How much can the basal metabolic rate increase in burn patients?
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What happens to capillary permeability during a systemic response to burns?
What happens to capillary permeability during a systemic response to burns?
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What is a consequence of high insulin resistance in burn patients?
What is a consequence of high insulin resistance in burn patients?
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What is one of the first steps in assessing burns?
What is one of the first steps in assessing burns?
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What occurs as a result of peripheral vasoconstriction during burn injury?
What occurs as a result of peripheral vasoconstriction during burn injury?
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What is a potential consequence of increased vascular permeability in burn injuries?
What is a potential consequence of increased vascular permeability in burn injuries?
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What hormonal changes occur due to burns?
What hormonal changes occur due to burns?
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What does the release of cytokines mainly affect in a burn injury?
What does the release of cytokines mainly affect in a burn injury?
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What physiological process is increased to generate glucose in burn patients?
What physiological process is increased to generate glucose in burn patients?
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What should be considered if burns affect the airway areas?
What should be considered if burns affect the airway areas?
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Study Notes
Burns
- A burn is an injury to skin or other tissue caused by heat, radiation, electricity, friction, or chemicals.
- Understanding skin layers is crucial for managing burns (homeostasis).
Skin Layers
- Epidermis: Five layers, deeper layers constantly divide and migrate to the surface.
- Dermis: Crucial for thermoregulation (sweat, piloerection), nerve function, blood vessels and glands responsible for skin regeneration.
- Dermal Vascular Plexus: Important for sweating, piloerection, skin regulation, blood flow and skin regeneration.
Burn Classifications
- 1st Degree: Superficial epidermal, rash, sunburn (no dermis damage).
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2nd Degree:
- Superficial dermal: blisters, red, peeling skin, blanch when pressed, capillary refill present.
- Deep dermal: dry, mottled, red skin, may blister; no blanch on capillary refill.
- 3rd Degree: Full thickness (epidermis and dermis damaged, nerves destroyed, black leathery appearance).
Local Response (Pathophysiology)
- Zone of Coagulation: Maximum damage, tissue damaged at the point of injury due to clotting of proteins.
- Zone of Stasis: Surrounding zone; decreased tissue perfusion, potentially salvageable.
- Zone of Hyperaemia: Outmost zone; increased tissue perfusion, inflammation.
Systemic Response (Pathophysiology)
- The release of cytokines impacts the entire body.
- Systemic effect when burns are 30% or higher total body surface area.
- Cardiovascular changes, capillary permeability increases, vessels leak, (reduced blood flow to extremities).
- Release of hormones (adrenaline, noradrenaline, cortisol).
Metabolic Changes
- Basal metabolic rate increases up to three times normal.
- Gluconeogenesis: increased glucose production.
- Insulin resistance: reduced glucose uptake.
- Hyperglycemia: high blood sugar.
Treatment Steps
- Assess the burn's extent and severity.
- Classify burns (superficial, deep).
- Establish airway, breathing, and circulation.
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Description
This quiz focuses on understanding burns, their classifications, and the layers of skin involved in their management. Key concepts include the structure of the epidermis and dermis, as well as the various degrees of burn injuries. Test your knowledge on how burns affect skin and the body's response to injury.