Questions and Answers
What is the basis for diagnosing aflatoxicosis in livestock?
Clinical signs and liver lesions
In the context of treating aflatoxicosis, what should be done if animals are immunosuppressed?
Consider antimicrobial therapy for infectious processes
Why is it important to avoid aflatoxin-contaminated feed during drought or widespread contamination?
To reduce contamination of aflatoxin M1 in milk
Which mycotoxin binder is generally recognized to be effective in binding aflatoxin in feed?
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What type of metabolites are aflatoxins?
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At what temperature is mold growth typically sufficient for the production of aflatoxins?
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What is the role of HSCAs in reducing aflatoxin effects in animals?
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Why should aflatoxin-contaminated feed be avoided in lactating animals?
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In what way do sodium bentonites and polymeric glucomannans affect aflatoxin residues in poultry and dairy cattle?
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What is a common effect of aflatoxins on different species?
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Why are mycotoxin binders like hydrated sodium calcium aluminosilicates used in animal feed?
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What is the primary reason for not using aflatoxin-contaminated feed in lactating animals?
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Which organ is the major target of aflatoxin toxicity?
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What is a potential consequence of prolonged exposure to low concentrations of aflatoxins?
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How are acutely affected animals by aflatoxin toxicity identified?
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What can be detected in the urine, liver, kidney, or milk of lactating animals with high aflatoxin intakes?
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What is one method to reduce aflatoxin contamination in grain?
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What is NOT currently approved by the US FDA for use in food animals to reduce aflatoxin contamination?
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Which type of aflatoxin is recognized as the predominant toxin found in crops?
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In which organ are aflatoxins metabolized to an epoxide that binds to macromolecules?
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What is the acceptable regulatory concentration range for aflatoxins in milk in different countries?
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Which animals are relatively resistant to the acute form of aflatoxicosis?
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What specific laboratory change is associated with liver damage due to aflatoxins?
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At what dietary concentration do aflatoxins generally cause acute clinical disease, including mortality?
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What is one of the primary effects of reduced protein synthesis due to aflatoxins?
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Which animals are susceptible if toxic diets containing aflatoxins are fed over long periods?
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What type of outbreaks are more usual in animals suffering from aflatoxicosis?
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What is the primary organ affected by high dosages of aflatoxins?
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