Atherosclerosis Quiz

Which of the following is NOT a characteristic of an atheromatous plaque?

What is the primary theory used to explain the development of atherosclerosis?

What is one consequence of endothelial cell dysfunction?

Which of the following is NOT a potential cause of endothelial cell injury?

What happens to the blood vessel once endothelial cell damage occurs?

What is the first step in the response-to-injury hypothesis?

What is the role of macrophages in the development of atherosclerosis?

What is the potential consequence of atherosclerosis in the coronary arteries?

What is the consequence of plaque rupture in the arteries?

What does advanced atherosclerosis typically show?

Which region is commonly inspected for signs of atherosclerosis post-mortem?

What is one of the risks associated with advanced atherosclerotic plaques?

What can happen if blood flow is blocked by a clot formed from plaque rupture?

What is one visual characteristic of mild atherosclerosis?

What is often noted about the fibrous cap of the plaque during progression of atherosclerosis?

What is a characteristic feature of complicated plaques in advanced atherosclerosis?

What are the two predominant mechanical forces exerted by blood flow on blood vessel walls?

How does shear stress affect endothelial cells in blood vessels?

In what condition is blood flow considered laminar?

What happens to laminar flow when it transitions to turbulent flow?

Which factor influences the levels of shear stress in blood vessels?

What is a primary consequence of endothelial dysfunction?

Which of the following statements best describes turbulent blood flow?

What does pressure do to vascular smooth muscle (VSM) cells?

What caused the significant reduction of blood flow to Mr. Jones' heart?

What is the primary composition of the plaque observed in Mr. Jones' coronary artery?

How does the lumen of Mr. Jones' diseased coronary artery compare to a healthy artery?

What event likely led to Mr. Jones’ acute myocardial infarction?

Which of the following statements about Mr. Jones' coronary artery condition is incorrect?

What was a significant visual characteristic observed in the plaque during examination?

What likely contributed to the strain on Mr. Jones’ heart in addition to restricted blood flow?

Which pathway accurately describes the progression of Mr. Jones' cardiac condition?

What do LDLs and VLDLs undergo that is significant in the progression of atherosclerosis?

What happens to monocytes during the progression of atherosclerosis?

Which cells migrate into the tunica intima to contribute to foam cell formation?

At what stage does atherosclerosis begin to be clearly observable and can still be reversed?

What characterizes the final stage of atherosclerosis, specifically the atheromatous plaque?

What lifestyle changes can reverse the formation of fatty streaks in atherosclerosis?

Which layer of the artery wall do smooth muscle cells migrate into during the atherosclerotic process?

What is the primary factor that leads to the formation of fatty streaks?

What indicates a potential risk of atherosclerosis in Mr. Jones' son?

What is the triglyceride level recorded for Mr. Jones' son?

Which lipid level is considered high in Mr. Jones' son?

Which lipid marker shows the highest risk level in Mr. Jones' son?

Which statement reflects the findings from Mr. Jones’ son’s blood sample?

What does a high level of Low Density Lipoproteins (LDL) generally suggest?

What does the presence of a semi-transparent serum in a healthy patient sample indicate?

What is the significance of comparing Mr. Jones’ son’s blood results with reference tables?