Ascites Management in Cirrhosis: Complications and Strategies

What is the primary cause of ascites in cirrhosis patients?

Which physiological process leads to arterial underfilling and subsequent sodium and water retention in ascites?

In ascites patients, what is the role of Angiotensin II in fluid retention?

What physiological change occurs in the kidney in response to hypotension in ascites?

How does splanchnic vasodilation contribute to the development of ascites?

What is the primary consequence of activation of the RAAS system in ascites patients?

Which step in the pathophysiology of ascites involves NO and hypotension?

What does a SAAG value of ≥1.1 g/dL and Ascitic Protein <2.5 g/dL indicate in a patient with ascites due to portal hypertension?

Which diagnostic test is commonly used to assess the cause of fluid accumulation in the abdomen in a patient with cirrhosis?

What is the recommended daily intake of sodium for patients with ascites?

In the treatment of Grade 1 ascites, which non-pharmacologic intervention may be effective?

What adverse effect is commonly associated with spironolactone in patients with cirrhotic ascites?

In which situation should IV albumin be administered after therapeutic paracentesis for Grade 3 ascites?

Which medication type should be discontinued in patients with ascites due to cirrhosis?

What procedure may be considered for ascites not responsive to diuretics and sodium restriction?