Somatoform Disorders PPT PDF

Summary

This presentation provides an overview of somatoform disorders, a group of disorders involving physical symptoms without a detectable medical cause. It discusses various types of somatoform disorders, their causes, symptoms, diagnosis, treatment approaches, and differential diagnoses. The document includes details on somatization, conversion disorder, hypochondriasis, body dysmorphic disorder, and pain disorder.

Full Transcript

Somatoform disorder at glance.  Disorder characterized by recurring, multiple, clinically significant complaints about pain, gastrointestinal, sexual and pseudoneurological symptoms  Causes  Defense against psychological distress  Heightened sensitivity to physical sensations  Psych...

Somatoform disorder at glance.  Disorder characterized by recurring, multiple, clinically significant complaints about pain, gastrointestinal, sexual and pseudoneurological symptoms  Causes  Defense against psychological distress  Heightened sensitivity to physical sensations  Psychodynamic, cognitive & behavioural view  Symptoms  Pain  Gastrointestinal, neurological & sexual symptoms  Menstrual problems  Incidence  More common in women than men Glance cont’d…  Diagnosis  No Laboratory tests  Little evidence of definable medical illness  History of multiple physical complaints  Symptoms might cause significant impairment to social, occupational & other areas of functioning  Pain affecting atleast four differential body parts  Physical exam for non-organic pain  McBride's test  Hoover's test  Burn's test  Treatment  Cognitive behaviour therapy  Medications Somatoform disorders  Group of disorders that includes physical symptoms for which an adequate medical explanation cannot be found  Psychological factors --> symptom’s onset, severity, duration  Not malingering or factitious disorder  Psychosomatic complaints frequently involve:  Chronic pain  Problems with the digestive system, nervous system, and reproductive system  Typical onset – before age 30  Higher prevalence for women than men  Somatization  Physical complaints or impairments:  Without organic pathology  That are grossly in excess of what would be expected from the physical findings  Malingering  Intentionally feigning or grossly exaggerating illness or disability to derive benefit or secondary gain (e.g., to escape work, gain compensation, or obtain drugs)  Factitious disorder  Characterized by:  Physical or psychological symptoms that are intentionally produced or feigned in order to assume the sick role  Conscious fabrication of symptoms to gain attention 5 Specific somatoform disorders: 1. Somatization Disorder 2. Conversion Disorder 3. Hypochondriasis 4. Body Dysmorphic Disorder 5. Pain Disorder 6. Undifferentiated Somatoform Disorder 7. Somatoform Disorder Not Otherwise Specified  Hysteria, Briquet’s Syndrome  Many somatic symptoms  Multiple complaints and organ systems affected  Chronic & debilitating  Epidemiology  Lifetimeprevalence = 0.1-0.2%  F > M (5-20X) = 5:1 Diagnosis  Onset before the age of 30 years  Complain of at least 4 pain sxs, 2 GI sxs, 1 sexual sx, 1 pseudoneurological sx  No physical or laboratory explanation  Clinical features  Many somatic complaints; long complicated medical history  Psychological distress: anxiety, depression  Common suicidal threats  Medical history is circumstantial, vague, imprecise, inconsistent, disorganized  Patients are dependent, self-centered, hungry for admiration or praise  Common associated mental Disorders - MDD, PD, SRD, GAD, phobias Differential diagnosis 1. Non-psychiatric medical condition 2. Mental Disorder - MDD, GAD, schizophrenia 3. Other somatization Disorders  Treatment  Single identified MD  Visits: regular, avoid additional lab/diagnostic procedures  Somatic symptoms - emotional expressions  Psychotherapy: individual, group Conversion disorder  One or more neurological symptoms (paralysis, blindness, paresthesias)  Psychological factors --> onset, exacerbation  Epidemiology  F:M = 2:1 - 5:1  Onset is any age (common during adolescence and young adults)  Rural population, little educated, low IQ, low SE group, military personel  Comorbid with MDD, anxiety, schizophrenia Etiology 1. Psychoanalytic - repression of unconscious conflict/anxiety --> physical sx Nonverbal means of controlling and manipulating 2. Biological factors - hypometabolism of dominant hemisphere impaired hemispheric communication Diagnosis  Symptoms or deficits affecting neurological functions  Psychological factors --> onset, exacerbations  Not intentionally feigned or produced  Clinical features  Most common symptoms: paralysis, blindness, mutism  Most commonly associated with passive-aggressive, dependent, antisocial and histrionic PDs  Sensory Sxs: anesthesia and paresthesia, esp extremities  distribution usually inconsistent with central or peripheral neuro dse  hicharacteristic stocking and glove anesthesia or hemianesthesia (along the midline)  organs of special senses - deafness, blindness, tunnel vision --> N neuro exam Clinical features cont’d… 2. Motor Sxs: abnormal movements, gait disturbance, weakness, paralysis generally worsen by attention 3. Seizure Sxs: pseudoseizure 4. Mixed presentation  Other associated features:  Primary gain: represent an unconscious psychological conflict  Secondary gain: accrue tangible advantages & benefits  Le belle indifference: unconcerned about what appears to be a major impairment  Identification: unconsciously model their sxs on those someone important to them Differential diagnosis  Rule out medical disorder: thorough medical and neuro work-up  25-50% diagnosed with conversion DO --> neuro or non-psychiatric medical DO 1. Neuro DO - dementia, brain tumors, degenerative dse, basal ganglia dse 2. Psychiatric DO - schiz, deprssive DO, other somatoform, malingering, factitious DO Course & Prognosis  90-100% resolve in few days to less than a month  Good prognosis: sudden onset, easily identifiable stressor, good premorbid adjustment, no comorbid psychiatric or medical DO  25-50% --> neuro or non-psychiatric DO  Treatment  Spontaneously resolve  Insight-oriented supportive or behavioral therapy HYPOCHONDRIASIS  Unrealistic or inaccurate interpretations of physical symptoms or sensations --> preoccupation and fear that they have serious disease  Significant distress; impaired function  Epidemiology  F=M  Onset at any age Etiology  Misinterpretation of bodily symptoms  Social learning model  Variant form of other mental disorder - depression and anxiety DO (80%)  Aggressive and hostile wishes Diagnosis, Signs & Syp.  Preoccupied with false belief based misinterpretation of physical s/sxs  At least 6 months  Not a delusion or restricted to distress of appearance  Clinical features  Believe that they have a serious disease not yet detected  Conviction persist despite negative lab results, benign course, reassurances  Usually with depression and anxiety Differential diagnosis  Non-psychiatric medical condition  Other somatoform disorders  MDD, anxiety Disorders, schizophrenia, other psychotic Disorders  Course &Prognosis  Episodic, months to years  Good prognosis: high SE class, treatment- responsive anxiety or depression, sudden onset, (-) PD, (-) related non-psychiatric medical condition Treatment  Usually resistant to psychiatric treatment  Focus on stress reduction and education in coping with chronic illness  Group psychotherapy  Regular scheduled PE Body dysmorphic disorder  Preoccupation with an imagined bodily defect or an exaggerated distortion of a minimal or minor defect  Causes significant distress; impaired function  Epidemiology  Rare; poorly studied  Most common age of onset: 15-30 yo  F > M, unmarried  Commonly coexists with other mental Disorders: (MDD, anxiety, psychotic DOs) Etiology  Serotonin  Cultural and social effects  Psychodynamic models  Diagnosis  Preoccupied with an imagined defect in appearance or an overemphasis of a slight defect  Significant emotional distress; impaired functioning Clinical features  Most common concerns: facial flaws  Common associated symptoms: ideas of reference, attempts to hide deformity, excessive mirror checking or avoidance  Avoid social or occupational exposure  Housebound; attempt suicide  Traits: O-C, schizoid, narcissistic PD  Comorbid: depression, anxiety Disorders Differential diagnosis  Anorexia nervosa, gender identity DO, brain damage  Delusional DO, somatic type  Narcissistic PD, depressive DO, OCD, schizophrenia  Course & Prognosis  Gradual onset  Usually chronic  Treatment  Serotonin-specific drugs - clomipramine, fluoxetine  Treat coexisting mental DO PAIN DISORDER  Psychogenic pain DO  Pain in one or more sites --> no non-psychiatric medical or neurological condition  Emotional distress; functional impairment  Epidemiology  F>M  Peak onset on 4th to 5th decades  Blue-collar occupation, 1st degree relatives Etiology  Psychodynamic: expression of intrapsychic conflict  defense mechanism-displacement, substitution, repression  Behavioral: reinforced with reward and inhibited when ignored/punished  Interpersonal: manipulation and gaining advantages  Biological: 5HT and endorphins  Diagnosis  Significant complaints of pain  Emotional distress and functional impairment Clinical features  Collectionof different histories of various pains  Pain maybe post-traumatic, neuropathic, neurological, iatrogenic, musculoskeletal  (+) psychological factor  Long history of medical and surgical care, visits many MDs, requests many meds  Complicated by SRD  MDD: 25-50% of patients  Dysthymic or depressive DO sxs - 60-100% Differential diagnosis  Physical pain VS Psychogenic pain  Physical Pain: fluctuates in intensity, highly sensitive to emotional, cognitive, attentional and situational influence  Psychogenic Pain: does not vary, insensitive to any of above factors, does not wax or wane, not temporarily relieved by distraction  Other somatoform Disorders  Course & Prognosis  Abrupt onset and increases in severity Treatment  Address rehabilitation  PAIN IS REAL  Pharmacotherapy - antidepressant  Behavioral therapy  Psychotherapy  Pain control program Geneeral treatment of Psychosomatic disorders  CARE-MD treatment approach  Cognitive behavioral therapy (CBT)  Assess: rule out medical causes  Regular visits: Short frequent visits with focused exams  Explorestressors, promote healthy coping  Set boundaries  Empathy  Med-psych interface  Do no harm  Do not dispute the reality or severity of the patient’s physical complaints  Be aware that somatizing patients are often intellectually challenging NCP Psychosomatic disorders  Cognitive-behavioral treatment  Biofeedback  Relaxation training  Group therapy  Family therapy NCP Psychosomatic disorders NCP Psychosomatic disorders

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