Summary

This document discusses persistent somatic symptoms, including case studies and the biomedical model. It explores the relationship between symptoms and disease, and the psychological factors that may influence symptom perception. The concepts of attention and illness schemas are also examined, with the text providing a detailed overview for clinical contexts or students of healthcare courses.

Full Transcript

Persistent Somatic Symptoms Tuesday, 12 November 2024 1:58 pm What are physical symptoms? - Experienced 'in the body' - Subjective - May also present with objective 'signs' - Can be medically explained or unexplained ○ No identifiable disease or pathology Case study - Conversion Di...

Persistent Somatic Symptoms Tuesday, 12 November 2024 1:58 pm What are physical symptoms? - Experienced 'in the body' - Subjective - May also present with objective 'signs' - Can be medically explained or unexplained ○ No identifiable disease or pathology Case study - Conversion Disorder - An extreme case of PSS - Conversion disorder - neurology - Body's response to some underlying stress - power of mind over the body - Unconscious symptoms - Holly Longford - lost ability to speak, walk - suddenly after a collision in netball - Tests negative for everything - no one knew what it was - Challenge accepting the psychological nature of symptoms - - Wikipedia definition: A symptom is a departure from normal function or feeling, noticed by a patient - reflecting the presence of a physiological disturbance or a disease Biomedical model of illness… - Underlying disease (pathology) --> causes illness and symptoms of disease - Symptoms --> detection of disease - Sometimes its not that easy (symptoms with no diagnosed disease - then it's all in you head) - Following debunks this idea: Disease /=/ Symptoms - Studies on viral infections - look at biological markers vs self reports of symptoms - Influenza antibodies - found 75% of people with biological evidence of infection, didn't report having flu symptoms - You may have a disease but not realise it / show symptoms - Also found in : ur t Disease /=/ Symptoms - Studies on viral infections - look at biological markers vs self reports of symptoms - Influenza antibodies - found 75% of people with biological evidence of infection, didn't report having flu symptoms - You may have a disease but not realise it / show symptoms - Also found in : ○ Zika virus (80%) ○ Covid ○ HPV (80% contract, around 90% asymptomatic) ○ Genital herpes (25% US adults, 75-90% asymptomatic) ○ High blood pressure / cholesterol ○ Early stage cancers - Highlights need for screening!! May not have symptoms but need to catch disease early Symptoms also /=/ disease - Symptoms are more common than disease - Around 90% of people have 1+ symptom every week (across general population in NZ) - Psychological and social processes - role in perception of physical symptoms / experience Responding to everyday symptoms - What do people do when they have symptoms? ○ Self medicate t ) Responding to everyday symptoms - What do people do when they have symptoms? ○ Self medicate ○ Ask people/google - information seeking ○ Rest ○ Seek medical help Symptoms in medical care - Top reason for presenting to a GP - having symptoms (used as an index for our health status) - 1998 study - almost half of all visits were from 14 common symptoms - Only 10-15% found to be caused by organic illness over a year period (the rest are medically unexplained) - Organic - some biological marker of disease explaining the symptom Normal psychological Influences on symptom reporting - Attention (and beliefs/expectations) - Emotions 1. Attention to internal sensations - Competition Of Cues Hypothesis - Pennbaker & Lightner 1980 - Can pay attention to body, or the outside world - Natural state of attention is to be focused internally, but if the outside world is engaging - it pulls attention external - If paying attention to your internal state, more likely to notice symptoms - Pennbaker & Lightner 1980 - Can pay attention to body, or the outside world - Natural state of attention is to be focused internally, but if the outside world is engaging - it pulls attention external - If paying attention to your internal state, more likely to notice symptoms Study: - Half participants ran around an indoor track, others ran outdoors - Indoor track - reported significantly more exercise symptoms (fatigue, muscle pain, breathless etc) - Authors say due to the focus of attention (less attention left directed to body when outside - more to look at/focus on) - Another study - people more likely to cough during boring lectures or videos - Interesting environment --> fewer symptoms Selective search for attention - Missed in the Competition of Cues theory… - We can direct / control out attention towards certain things - Illness schemas - broad idea of what a particular illness involves ○ E.g. influenza - fever, aches, chills, tiredness, sore throat etc - Schemas can be formed by our experience, media portrayals, information - Schemas - generate a schema-guided search process for symptoms - - Schema activated - pay more attention to body looking for particular symptoms - then you may tend to experience them ! Experimentally tested this… Fictitious Enzyme Deficiency - Thiamine Acetylase (TAA) deficiency - Gave them an illness schema - Pancreatic enzyme that can cause headache, diarrhea, back pain - Diagnosed by saliva test - BUT - solution given was glucose and test strip wsa glucose test - Half told blue strip - positive for TAA deficiency - Other half told blye strip - negative for TAA deficiency - People with 'positive' test result experienced significantly more of those symptoms - Given schema - paying more attention to them - causes symptoms to occur Distress and Symptom reporting - People high in negative affect report more physical symptoms - True in people with trait negative affect AND experimentally manipulated negative affect n Distress and Symptom reporting - People high in negative affect report more physical symptoms - True in people with trait negative affect AND experimentally manipulated negative affect - Negative emotions - anxiety, depression, anger, distress - Doesn't seem to be that there is more disease Why? 1. Distress is experienced as symptoms ○ Somatisation - direct link from emotions to physical symptoms 2. Negative bias ○ Negative mood - negative attentional bias - focus on more physical symptoms which are generally negative ○ Memory - in a negative mood you're also more likely to remember negative memories/symptoms 3. Increased self focus ○ Negative mood - attention turns inwards to yourself - more likely to notice symptoms Somatic Symptom Disorder - Excessive thoughts, feelings or behaviours related to somatic symptoms or associated health concerns ○ Disproportionate and persistent thoughts about the seriousness of one's symptoms ○ Persistently high level of anxiety about health/symptoms ○ Excessive time and energy devoted to symptoms / health concerns - Need to be persistently symptomatic for 6+ months and cause distress - Symptoms are real and can be varied - Typically no apparent underlying cause or disease - Can also be diagnosed when someone has a disease/diagnosis - but focus on symptoms is excessive or unusual for that diagnosis - Somatisation - Alexithymia ○ Difficulty identifying feelings and distinguishing feelings from associated bodily arousal ○ Unable to link/explain physical symptoms to an emotion / psychological state (e.g. my heart is racing because I'm nervous) Impact of somatic symptom disorder - Diagnosis of exclusion - Need to run many tests - reinforces belief that something is wrong ○ Unable to link/explain physical symptoms to an emotion / psychological state (e.g. my heart is racing because I'm nervous) Impact of somatic symptom disorder - Diagnosis of exclusion - Need to run many tests - reinforces belief that something is wrong - Lack of reinforcement from negative results - Symptoms are still there - Expensive and distressing - Can be challenging to treat - belief that symptoms are coming froma disease - not psychological contributors Exam - Pain - Distinct physical symptom that often becomes persistent, after biological healing processes have occurred - "unpleasant sensory and emotional experience associated with actual or potential tissue damage, or described in terms of such damage" A life without pain? - Some people don't experience pain - Congenital Analgesia - Acute pain - pain in response to actual tissue damage is very important - warning that something is wrong and need to change (e.g. put hand on a hot stove) - People with congenital analgesia often die very young - injured / very ill but don't recognise it - Genetic mutation - Peripheral pain sensing neurons not working as they should - don't get pain signals something is wrong and need to change (e.g. put hand on a hot stove) - People with congenital analgesia often die very young - injured / very ill but don't recognise it - Genetic mutation - Peripheral pain sensing neurons not working as they should - don't get pain signals Pain processing - Nociception ○ Nervous systems process of encoding impending or actual tissue damage - Pain ○ The subjective experience of actual or impending harm - 4 stages: transduction, transmission, perception, modulation - 1. Transduction - Starts at nerve endings (nociceptors) - Primary afferent neurons - sensory neurons sending messages to the brain in 1. Transduction - Starts at nerve endings (nociceptors) - Primary afferent neurons - sensory neurons sending messages to the brain in peripheral nervous system - Respond to noxious stimuli - tissue damage and inflammation - C and A delta fibres ○ A - large diameter, myelinated, fast conducting - localised/sharp/stinging / pricking/ fast pain ○ C - small diameter, unmyelinated, slow conducting - dull / diffuse/ aching/ burning / slow 2. Transmission - Nociceptors send signal through dorsal horn of spinal cord to brainstem - Brainstem distributes signal across your brain - 3. Perception - Conscious multidimensional experience - Motivational - Behavioural - Sensory-discriminative - Emotional - This is where the biomedical view of pain stops… - It says: pain severity = amount of tissue damage - It says: same injury = same amount of pain - This doesn't do a great job of explaining the pain experience - Magnitude of injury doesn't always match the pain someone's experiencing Gate Control Theory (Melzack & Wall 1965) - Biopsychosocial view of pain - Hypothetical 'gate' that can open or shut (in dorsal horn of spinal cord) ○ Open - all the pain and suffering flows through Gate Control Theory (Melzack & Wall 1965) - Biopsychosocial view of pain - Hypothetical 'gate' that can open or shut (in dorsal horn of spinal cord) ○ Open - all the pain and suffering flows through ○ Shut - won't experience pain - 'Gate' receives in put from two directions: ○ Ascending messages - biological - bottom up information (maps onto biomedica model - periphery to brain) ○ Descending messages - psychological - top down modulation 4. Modulation - Extra step - only involved in biopsychosocial view (not in biomedical model) - Can be excitatory - increase pain (gate wide open) or inhibitory - decrease pain (close gate) - Psychological factors that influence pain - Emotions ○ Negative emotion (trait or induced) experience more pain - Thoughts about pain: ○ Beliefs about cause and effect, experiences, control, self-efficacy for managing - Meaning: ○ Study using blood pressure machine ○ 1 group - negative info (painful, blocking blood flow) other group - positive info (good for muscle cells) al - Thoughts about pain: ○ Beliefs about cause and effect, experiences, control, self-efficacy for managing - Meaning: ○ Study using blood pressure machine ○ 1 group - negative info (painful, blocking blood flow) other group - positive info (good for muscle cells) ○ Negative group could tolerate pain for much less time than positive group ○ Focusing on positive/beneficial effects - inhibited modulating effect - less pain signal sent to brain - Attention: ○ When distracted - experience significantly less pain than when paying attention to the pain experience Acute pain - Typically results from a specific injury or disease process - E.g. wound or infection - Short lived - resolves when tissue damage heals (but not always!) - Duration 3 months or less … - Psychological factors can influence the development of chronic pain from acute pain Chronic pain - Begins as acute pain - Lasts longer than 3 months - Beyond expected healing time - In excess of injury or disease Fear avoidance model - When an injury leads to pain experience - 2 pathways you may take 1. No fear - heal from injury 2. Pain catastrophising …. - - **know for exam: Biological processes of Pain Peripheral Sensitisation - Acute Pain - Nociceptor neurons have: ○ Reduced threshold - require less stimulation to send on a pain signal ○ Augmented response - send a more dramatic pain signal ○ ^Caused by inflammatory processes at the injury site, common after injury Central Sensitisation - Chronic Pain - Neurons in dorsal horn: ○ Amplify incoming information ○ 'wind up' - persistently high reactivity - strong signals to the brain ○ Lowered pain threshold Cortical Reorganisation - Chronic Pain - Somatosensory cortex: ○ Remodels in response to repeated input (neuroplasticity) ○ Greater and more widespread activation to nociceptive(painful) input - Second two are thought to be what maintains chronic pain and are influenced by psychological factors Treating chronic pain - CBT can be helpful - Second two are thought to be what maintains chronic pain and are influenced by psychological factors Treating chronic pain - CBT can be helpful - Educating people on the processes - Helping to tackle attention and affective mood - Restructure cognitions around pain Exam Info:

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