Summary

These lecture notes from King Salman International University cover venous disorders, including deep venous thrombosis, chronic leg ulcers, and important notes on malignant ulcers. The document includes treatment options and complications.

Full Transcript

VENOUS DISORDERS PROFESSOR. SHERIEF MOHSEN DATE: 11/2024 DEEP VENOUS THROMBOSIS Etiology and PFs (Virchow triad) Decrease velocity (post.operative,prolonged recumbency,HF) Increase viscosity (polycythemia, burns, dehydration) Vessel wall injury (inflammation, trauma, previous D...

VENOUS DISORDERS PROFESSOR. SHERIEF MOHSEN DATE: 11/2024 DEEP VENOUS THROMBOSIS Etiology and PFs (Virchow triad) Decrease velocity (post.operative,prolonged recumbency,HF) Increase viscosity (polycythemia, burns, dehydration) Vessel wall injury (inflammation, trauma, previous DVT) Pathology Grey cluster (platelet adherence) Lines of Zahn (Fibrin and RBCs laminated deposition) Loose thrombus (very dangerous for pulmonary embolism) Tight thrombus and consolidation (organised thrombus) Recanalization and valve distruction (post phelebitic limb) Most important in managment of ulcer is three - four layered compression bandaging CHRONIC LEG ULCERS IMPORTANT NOTES Malignant ulcer: may complicate burn or other types of ulcers (malignant change = Marjolin ulcer) Squamous cell carcinoma: ccc by painless + raised everted edge + L.Ns Basal cell carcinoma: ccc by painless + rolled-in edge + No L.Ns T.B ulcer: ccc by Painful + undermined edge + cyanotic margin + matted L.Ns Syphlitic ulcer: Painful + punched-out edge + cyanotic margin + floor covered with wash leather slough

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