Psychology Chapter 11: Physical Symptoms: Pain and Discomfort PDF
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Summary
This chapter explores the qualities and types of pain, differentiating between organic and psychogenic pain, acute and chronic pain. It covers different pain types, like chronic recurrent and intractable pain, and discusses the impact of chronic pain on individuals' well-being and daily functioning. The role and unique properties of pain perception are also detailed.
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CHAPTER 11 Physical Symptoms: Pain and Discomfort The Qualities and Dimensions of Pain The Qualities and Dimensions of Pain Definition of Pain Pain is an unpleasant sensory and emotional experience linked to actual or potential tissue damage, or described in terms of such damage. Qualiti...
CHAPTER 11 Physical Symptoms: Pain and Discomfort The Qualities and Dimensions of Pain The Qualities and Dimensions of Pain Definition of Pain Pain is an unpleasant sensory and emotional experience linked to actual or potential tissue damage, or described in terms of such damage. Qualities of Pain Pain experiences are highly varied and can be described with different qualities: ○ Sharp Pain: Includes stabbing or pricking sensations. ○ Dull Pain: Often described as aching or throbbing. ○ Burning Sensation: Feels like a burning on the affected area. ○ Cramping, Itching, or Aching: Each has a distinct sensation that indicates different types of irritation or damage. ○ Throbbing, Constant, Shooting, Pervasive, or Localized Pain: These descriptions relate to the intensity and spread of the pain. Dimensions of Pain 1. Organic vs. Psychogenic Pain: ○ Organic Pain: Directly linked to tissue damage or physical disorders. Examples include pain from burns or sprains. ○ Psychogenic Pain: No observable tissue damage; pain is believed to stem from psychological processes. It was once thought to be 'not real' but is now recognized as a genuine experience, similar to organic pain. 2. Acute vs. Chronic Pain: ○ Acute Pain: Short-term pain that lasts less than about three months. Often resolves with treatment and is not directly connected to past pain experiences. Example: pain from minor injuries or surgeries. ○ Chronic Pain: Long-lasting pain that persists beyond the usual course of an injury or illness, often without a clear end. Can lead to anxiety, depression, and significant disruption in life and work. Chronic Pain Types 1. Chronic-Recurrent Pain: Benign and episodic, with intense episodes followed by pain-free periods. Examples include migraine and tension-type headaches. 2. Chronic-Intractable-Benign Pain: Continuous pain of varying intensity without an underlying malignant condition. Example: chronic low back pain. 3. Chronic-Progressive Pain: Continuous and worsening pain associated with a malignant condition, increasing in intensity as the condition progresses. Examples include pain related to rheumatoid arthritis or cancer. Impact of Chronic Pain Chronic pain significantly affects individuals' psychological and physical health, influencing sleep, emotional well-being, and overall quality of life. It often results in diminished social interactions, reduced interests, and increased focus on pain management, leading to a restricted lifestyle centered around medical and pain management routines. Perceiving Pain Unique Properties of Pain Perception 1. Lack of Specific Receptor Cells: ○ Unlike vision or hearing, pain perception does not rely on specific receptor cells. ○ Nerve fibers detect and send signals of tissue damage. 2. Response to Diverse Noxious Stimuli: ○ Pain can result from physical pressure, lacerations, intense heat, or cold. 3. Emotional Component: ○ Pain perception is deeply tied to emotional experiences, making it a complex interplay of physiological and psychological processes. Neuropathic Pain Pain occurring without a detectable noxious stimulus. Often stems from past disease or nerve damage. Common Syndromes: 1. Neuralgia: ○ Characterized by recurrent episodes of intense shooting or stabbing pain along a nerve. ○ Example: Trigeminal Neuralgia (affects the facial trigeminal nerve). ○ Triggered by innocuous stimuli (e.g., cotton ball on the skin) rather than noxious stimuli (e.g., pinprick). 2. Causalgia (Complex Regional Pain Syndrome): ○ Severe burning pain in areas of prior severe injury (e.g., gunshot wound). ○ Triggered by minor stimuli (e.g., clothing, air puff). ○ Pain persists unpredictably and intensifies after wound healing. 3. Phantom Limb Pain: ○ Pain perceived in a missing or irreparably damaged limb. ○ Sensations of the limb's presence are common post-amputation. ○ Pain descriptors include shooting, burning, or cramping. ○ Example: A phantom hand feels "clenched," with nails digging into the palm. Unresolved Puzzle: Why only some individuals with neural damage develop these conditions remains unclear. Likely involves physiological (neural damage) and psychological factors. The Role of Pain’s Meaning 1. Masochism: ○ For some, pain is associated with pleasure, often in a sexual context. ○ Classical Conditioning Explanation: Pain paired with pleasurable outcomes (e.g., sexual stimulation) changes its perceived meaning. Example: Pavlov’s dogs approached aversive stimuli that signaled food. 2. Pain Perception Differences: ○ Beecher’s WWII Study: Soldiers perceived less pain from severe wounds than civilians with surgical wounds. Explanation: For soldiers, injuries meant escape from danger and returning home (positive meaning). For civilians, injuries symbolized disruption and personal problems (negative meaning). Psychological Influences on Pain Perception Cognitive and Emotional Factors: ○ Pain is less noticeable during distraction (e.g., competitive sports). ○ Optimism and context (e.g., relief from danger) significantly shape pain perception. Theories of Pain (Early Theories of Pain and The Gate-Control Theory of Pain) 1. Early Theories of Pain Early theories took a mechanistic view of pain, emphasizing sensory processes but neglecting psychological influences. 1. Specificity Theory: ○ Definition: Proposed a dedicated sensory system for pain, similar to vision or hearing. ○ Components: Special receptors, peripheral nerves, pathways to the brain, and brain areas for pain. ○ Criticism: Evidence shows no such specific sensory system exists for pain. 2. Pattern Theory: ○ Definition: Suggested that pain arises from certain patterns of neural activity shared with other senses. ○ Mechanism: Intense stimulation triggers unique patterns interpreted as pain (e.g., strong touch = pain, mild touch ≠ pain). ○ Criticism: Fails to explain why innocuous stimuli (e.g., light touch) can cause pain in conditions like causalgia. Overlooks psychological factors, such as attention, beliefs, or emotional states. Limitations of Early Theories: Did not account for psychological factors influencing pain perception. Misinterpreted hypnotized individuals, predicting they must feel pain but suppress their reaction. 2. Gate-Control Theory of Pain Developed in the 1960s by Ronald Melzack and Patrick Wall, this theory incorporates physiological and psychological processes in pain perception. Key Concepts: 1. Neural Gate Mechanism: ○ Located in the substantia gelatinosa of the spinal cord’s dorsal horn. ○ Modulates pain signals before they reach the brain. 2. Pain Pathway: ○ Noxious stimuli activate pain fibers (A-delta and C fibers). ○ Signals pass through the gate to transmission cells, which send impulses to the brain. ○ Pain is perceived when transmission cell output exceeds a critical threshold. 3. Gate Modulation Factors: ○ Activity in Pain Fibers: Pain fibers (A-delta and C) open the gate, increasing pain perception. ○ Activity in Peripheral Fibers: A-beta fibers (e.g., touch or rubbing stimuli) inhibit pain by closing the gate. Explains why rubbing an injured area or applying heat reduces pain. ○ Descending Messages from the Brain: Brain processes (e.g., distraction, anxiety, excitement) influence the gate. Example: Stress or anxiety can open the gate, while relaxation or distraction closes it. Application of Gate-Control Theory: 1. Psychological Factors: ○ Hypnosis, distraction, and positive emotions can reduce pain perception by closing the gate. ○ Stress or negative emotions may enhance pain by keeping the gate open. 2. Experimental Evidence: ○ David Reynolds’ Rat Study: Rats with electrodes in the periaqueductal gray area of the brainstem did not react to painful stimuli. Electrical stimulation induced analgesia, supporting the theory that brain impulses can inhibit pain perception. ○ Morphine activates this analgesic pathway, confirming the neural basis of pain modulation. 3. Stress-Induced Analgesia: ○ Pain perception reduces temporarily after a stressful event, explained by the gate-control mechanism. 3. Advancements: Neuromatrix Concept Proposed by Melzack: ○ A neuromatrix in the brain integrates sensory, emotional, and cognitive information. ○ Explains phenomena like phantom limb pain, where the brain perceives pain without noxious stimuli. Conclusion The gate-control theory provides a biopsychosocial framework for pain perception, accounting for sensory inputs, emotional states, and cognitive processes. Advances like the neuromatrix enhance understanding of complex pain phenomena. 1. Cold-Pressor Procedure Description: ○ Involves immersing a participant’s hand and forearm in ice water (maintained at 2°C or 35.6°F) to induce a continuous "aching" or "crushing" pain. ○ Water circulation ensures consistent cold exposure. Procedure: ○ Participant's arm is first immersed in room-temperature water for one minute. ○ They are informed about potential temporary discolouration and the rapid reduction of discomfort after removal. ○ Pain is assessed using: Self-ratings (e.g., pain scale). Endurance time (duration the participant can tolerate the pain). Experimental Findings: ○ Coping strategies, such as positive self-statements, reduce pain perception. ○ Study Example: Subjects trained to use self-statements ("I can handle it") reported less pain when these were explained as enhancing personal control. Without the explanation, pain ratings increased. 2. Muscle-Ischemia Procedure Description: ○ Reduces blood flow to arm muscles using a sphygmomanometer cuff inflated to 240 mm Hg. ○ Produces discomfort and temporary numbness or throbbing, simulating ischemic pain. Procedure: ○ Arm is raised to drain venous blood before inflating the cuff. ○ Participants are informed the procedure is safe but may cause temporary discomfort or discolouration. ○ Pain is measured through: Pain threshold: Pressure level at which discomfort begins. Self-ratings: Subjective reports of pain intensity. Experimental Findings: ○ Relaxation techniques or distractions (e.g., listening to comedy) significantly increase pain thresholds. ○ Pain threshold improvements (up to 50% higher) highlight the impact of psychological strategies on pain perception. Biopsychosocial Aspects of Pain 1. Neurochemical Basis of Pain and Psychosocial Links Electrical stimulation of the periaqueductal grey (PAG) area of the brainstem can produce analgesia (pain inhibition). Endogenous Opioids: ○ Naturally occurring neurochemical substances (e.g., endorphins) that reduce pain by inhibiting pain signal transmission. ○ These opioids are triggered by psychological factors like expectations or stress reduction. ○ Example: Placebos can stimulate endogenous opioid release, reducing pain perception. 2. Psychosocial Factors and Chronic Pain Psychosocial processes play a significant role in the development and maintenance of chronic pain. Classical Conditioning and Pain: Pain-related stimuli (e.g., words like "throbbing" or "stabbing") become conditioned stimuli, leading to distress and heightened pain perception. ○ Example: Migraine sufferers show stronger physiological reactions to pain-related words than non-sufferers. Repeated associations between pain and specific stimuli (e.g., movements) can condition pain-related behaviors and contribute to chronic pain. Pain Behaviors: Definition: Observable actions indicating pain (e.g., grimacing, limping). Types (Turk et al., 1985): ○ Facial or audible expressions (e.g., moaning, clenching teeth). ○ Distorted ambulation or posture (e.g., guarded movements, holding painful areas). ○ Negative affect (e.g., irritability). ○ Avoidance of activity (e.g., lying down, missing work). Reinforcement: ○ Pain behaviors are often maintained by operant conditioning (e.g., receiving attention or avoiding disliked activities). ○ Example: A person avoids lifting heavy objects after associating it with pain, reinforcing avoidance behavior. Secondary Gains: Benefits from pain (e.g., avoiding chores, receiving disability payments) can reinforce pain behaviors. Impact of Financial Compensation: ○ Patients receiving higher compensation report longer hospital stays, more chronic pain, and less success with treatments. 3. Social Reinforcement of Pain Behaviors Family and Social Reactions: ○ Attention from family or friends reinforces pain behaviors (e.g., children scratch more when parents pay attention to their scratching). ○ Distracting children from symptoms reduces pain behaviors and improves well-being. Spousal Influence: ○ Spouses' solicitousness (e.g., providing high levels of care) correlates with greater pain reporting and less activity. ○ Example: Patients report more pain when solicitous spouses observe them compared to when a neutral observer does. Impact of Family Dynamics: ○ Lack of family cohesion or excessive solicitousness promotes dependency and chronic pain behaviors. ○ Negative cycles of increased pain behaviors and solicitous responses may worsen physical and emotional outcomes. 4. Sociocultural and Gender Differences in Pain Gender Differences: ○ Men and women have similar pain thresholds but differ in pain intensity ratings: Women rate cold-pressor pain higher and report more interference with daily activities due to pain. Women have higher prevalence of migraines, back pain, and neck pain compared to men. Sociocultural Factors: ○ Lower socioeconomic classes report more pain and disability. ○ Indigenous populations in Canada experience higher rates of chronic pain and arthritis due to systemic barriers (e.g., limited healthcare access). ○ Experimental findings suggest cultural differences in pain perception (e.g., Chinese Canadians report higher cold-pressor pain than European Canadians). 5. Placebos and Pain Placebo Effect: ○ An inert substance or procedure that reduces pain due to the patient’s expectation of relief. ○ Effective for ~50% of patients, though effects diminish with repeated use. Mechanism: ○ Placebos reduce pain by: Decreasing stress. Triggering the release of endogenous opioids (e.g., endorphins). ○ Evidence: Double-blind studies show that blocking opioid activity (e.g., with naloxone) diminishes placebo-induced pain relief. Ethical Dilemmas: ○ Debate exists about using placebos in clinical practice, balancing patient benefit with informed consent and transparency. 6. Psychological Constructs and Pain Self-Efficacy: ○ Patients who believe they can control their pain experience less pain and rely less on medication. Social Injustice: ○ Beliefs about being misunderstood or mistreated can worsen pain outcomes and hinder recovery. Emotions, Coping Processes, and Pain Impact of Emotions on Pain Perception Positive Emotions: Positive emotions such as laughter have been shown to reduce the perception of pain, providing a natural form of pain relief. Negative Emotions: Negative emotions including depression, anxiety, and anger are prevalent in individuals with chronic pain and can exacerbate the pain experience. High levels of these emotions correlate with increased pain and disability. Research Findings: Chronic Pain and Emotion: Chronic pain is often accompanied by significant emotional distress. This relationship is bidirectional; emotional distress can exacerbate pain, and chronic pain can lead to increased stress and negative emotional states. Stress and Headaches: Studies indicate a strong correlation between stress and the onset of headaches, including migraines and tension-type headaches. This link is particularly pronounced in individuals with depressive symptoms. Stress-Induced Pain: A study involving arithmetic tasks under timed conditions showed that individuals with chronic headaches reported increased pain under stress, suggesting that stress can directly cause pain or significantly worsen existing pain conditions. Emotional Coping with Pain: Coping Mechanisms: Individuals coping with chronic pain frequently adopt emotion-focused coping strategies. These strategies might include rest, medication, relaxation techniques, or psychological methods such as distraction or positive self-talk. Effectiveness: These coping strategies, particularly covert ones like hoping or praying, are generally less effective in reducing chronic pain, indicating a need for more robust management techniques. Psychological Impact and Adjustment: MMPI Findings: The Minnesota Multiphasic Personality Inventory (MMPI) often shows that chronic pain patients have high scores on hypochondriasis, depression, and hysteria scales. These findings suggest significant emotional distress associated with chronic pain, regardless of the pain's organic or psychogenic origin. Catastrophizing: This involves an exaggerated negative mental state where individuals anticipate or perceive pain more intensely. Catastrophizing can worsen the pain experience and contribute to poorer outcomes in pain management. Biopsychosocial Model: Integrated Approach: Emphasizing a biopsychosocial approach is critical in pain management, acknowledging the complex interplay between biological, psychological, and social factors. Family Dynamics: The emotional state of family members, such as a spouse's depression, can also impact a patient's pain perception and management, highlighting the need for comprehensive family-inclusive treatment plans. Canadian Contributions: Psychosocial Perspectives on Pain Social Communication Model of Pain Proposed by: Ken Craig from the University of British Columbia. Focus: This model stresses the importance of social interactions in both acute and chronic pain experiences, diverging from traditional models that primarily focus on biophysical and psychological factors. Key Concept: Pain is not only an individual experience but also a social one, where the patient and caregiver interactions play a crucial role in shaping the pain experience. Dynamics: The model outlines that both patient and caregiver bring unique characteristics and histories that influence pain expression and management. The interaction between these factors determines the effectiveness of the pain treatment. Research Findings: Experimental studies, such as those involving the cold-pressor task, indicate that social presence can modify an individual's pain response, aligning it with those observed in others present. Communal Coping Model of Pain Catastrophizing Proposed by: Michael Sullivan from McGill University. Focus: This model delves into the interpersonal context of pain catastrophizing, where individuals may exaggerate pain responses to elicit help and empathy from others. Concept of Catastrophizing: Describes a response where individuals magnify and ruminate on pain, which can exacerbate the perceived intensity and lead to feelings of helplessness. Social Function: Catastrophizing is posited as a method to engage social support during acute pain episodes, although it may become maladaptive in chronic conditions, potentially leading to relationship imbalances and social conflicts. Empirical Support: High catastrophizers tend to show increased pain behavior when observed by others and may adopt less effective coping strategies when alone. Supportive interactions with close ones, like spouses, can mitigate the adverse effects of catastrophizing on pain outcomes. Implications for Treatment Biopsychosocial Approach: Both models advocate for a comprehensive approach to pain management that integrates biological, psychological, and social factors. Healthcare Application: These insights are crucial for healthcare practitioners and caregivers, suggesting that understanding and intervening in the social dynamics around patients can enhance pain management strategies. Family and Caregiver Role: The models underscore the importance of the support system around the patient, including family members and caregivers, in managing pain effectively. Assessing People’s Pain Self-Report Methods Interview Methods in Assessing Pain Purpose: Gather detailed qualitative information about the pain experience, especially for chronic pain patients. Key Areas of Focus: ○ History of Pain: When it began, progression over time, and past pain management efforts. ○ Emotional Adjustment: Psychological state before and after the onset of pain. ○ Lifestyle Impact: Changes in recreation, exercise, and diet due to the pain. ○ Pain's Social Context: Relationship dynamics during pain episodes and responses from family and coworkers. ○ Triggers and Coping Strategies: Events or factors worsening the pain and methods used to manage it. Pain Rating Scales and Diaries Pain Rating Scales: ○ Allow individuals to quantify pain intensity quickly and effectively. ○ Types of Scales: Visual Analogue Scale (VAS): A line with two endpoints (e.g., "No Pain" to "Worst Pain"). Suitable for children as young as 5 years. Box (Numeric) Scale: A numeric range (e.g., 0–10) representing increasing pain levels. Verbal Rating Scale: Descriptive phrases, such as "No Pain," "Considerable Pain," "Worst Pain Possible." ○ Advantages: Ease of use. Enables tracking of pain changes over time or during specific activities. Useful in identifying patterns (e.g., severity during specific times or activities). Pain Diaries: ○ A record of pain episodes over time, capturing: Details of Episodes: Intensity, timing, body location, and contextual factors (e.g., activity during onset). Medication Use: Doses and their impact. Comments: Observations about triggers, relief measures, and emotional state. ○ Clinical Use: Reveals pain trends, environmental influences, and helps improve patient-family dynamics by clarifying misconceptions. Pain Questionnaires Purpose: Capture the multidimensional nature of pain beyond just intensity. Key Example: McGill Pain Questionnaire (MPQ): ○ Development: Created by Ronald Melzack to account for sensory, affective, and evaluative dimensions of pain. ○ Structure: Pain Location: Patients mark areas of pain on a body diagram. Pain Descriptors: Select words from 20 subclasses that best describe their pain. Example: Subclass for "hot" includes "burning," "scalding," and "searing." Pain Patterns: Time-related variations (e.g., "continuous" or "intermittent"). Intensity Ratings: Five levels from "Mild" to "Excruciating." ○ Scoring: Pain Rating Index: Sum of subclass word scores. Present Pain Intensity (PPI): Based on current pain ratings. ○ Strengths: Effective for chronic pain assessment. Distinguishes between different pain types (e.g., arthritis vs. cancer). ○ Limitations: Requires strong English proficiency. Complex vocabulary (e.g., "lancinating," "taut") may confuse respondents with limited language skills or young children. Other Questionnaires: ○ Multidimensional Pain Inventory (MPI): Assesses the impact of pain on psychosocial factors like relationships and coping mechanisms. Behavioural Assessment Approaches 1. Assessing Pain Behavior in Structured Clinical Sessions Overview: ○ Structured sessions provide a controlled environment to assess pain-related behaviors systematically. ○ Typically conducted in hospital or clinical settings, these sessions focus on predefined pain behaviors and specific tasks. Key Methods: ○ UAB Pain Behavior Scale: A tool used by nurses during routine care (e.g., morning rounds). Patients perform several tasks, and 10 pain-related behaviors are rated on a 3-point scale: "None," "Occasional," "Frequent." Behaviors include mobility, medication use, and physical signs (e.g., grimacing or guarding). Scores are converted into numerical values for a total pain behavior score. ○ Standardized Activities: Patients perform a set of predetermined activities such as: Walking. Picking up an object. Removing shoes while sitting. Performing exercises like trunk rotations, toe touching, or sit-ups. These activities are videotaped, and trained assessors evaluate behaviors like: Guarded movement. Grimacing. Rubbing pain areas. Sighing. Results correlate well with self-reported pain levels, offering reliability and objectivity. 2. Assessing Pain Behavior in Everyday Activities Purpose: ○ Observe how pain impacts the patient’s daily life in their natural environment. ○ Identify how often specific pain behaviors occur and their effects on daily functioning. Key Methods: ○ Observation by Family Members or Key Others: Family members or caregivers, with proper training, track pain-related behaviors such as: Complaining of discomfort. Spending excessive time in bed. Requesting help for movement. Limping or avoiding physical activities. Observations are recorded systematically to ensure accuracy. ○ Fordyce Procedure: Developed by Wilbert Fordyce (1976), it involves: Compiling a list of 5–10 behaviors signaling pain (e.g., grimacing, vocal complaints). Tracking the frequency and duration of these behaviors. Monitoring how others respond to the pain behaviors, which may reinforce or discourage them. The procedure also examines the social context influencing pain behaviors. ○ Pain Diaries: Assessors maintain a detailed log of pain episodes, including: Details of Episodes: Date, time, and location (e.g., in the car, in bed). Observations: Specific behaviors suggesting pain. Thoughts and Feelings: Emotional responses during the episode. Actions Taken: Steps to alleviate the pain, rated for effectiveness on a scale from “did not help” to “stopped the pain completely.” Provides valuable data on pain patterns, triggers, and interpersonal influences on the pain experience. Psychophysiological Measures 1. Definition and Purpose Psychophysiology: The study of mental or emotional processes reflected in physiological changes. Pain has both sensory and emotional components that cause changes in bodily functions, which can be measured using psychophysiological tools. 2. Methods of Psychophysiological Pain Assessment A. Electromyograph (EMG) Definition: Measures electrical activity in muscles, reflecting their tension. Applications in Pain Assessment: ○ Muscle tension is associated with various pain states, such as: Headaches. Low back pain. ○ EMG activity increases during physical or psychological stress in pain patients compared to pain-free individuals. Correlation with Pain Intensity: ○ Extended Duration: EMG recordings over longer periods correlate reliably with pain intensity. ○ Short Duration: Brief assessments yield inconsistent results. Limitations: Muscle inactivity leads to no consistent differences between pain patients and controls. B. Measures of Autonomic Activity Definition: Evaluates physiological changes in the autonomic nervous system, such as: ○ Heart rate. ○ Skin conductance. Findings: ○ Autonomic changes relate more strongly to subjective pain ratings than to the actual strength of the pain stimulus. ○ Associations with chronic pain are inconsistent. ○ Autonomic responses can occur without pain (e.g., during stress). Limitations: Variability in autonomic activity makes interpretation difficult in reflecting pain. C. Electroencephalograph (EEG) and Evoked Potentials EEG: Records electrical activity of the brain. Evoked Potentials: ○ Definition: Electrical changes in the brain triggered by sensory stimuli (e.g., pain, sound). ○ Characteristics of Pain Stimuli: Evoked potentials increase in amplitude with stronger pain stimuli. Amplitudes decrease after administering analgesics. Strong correlation with subjective pain reports. Advantages: Provides objective evidence of pain intensity and response to treatment. Limitations: Influenced by other factors like attention and external conditions. 3. Clinical Implications Psychophysiological measures offer objective insights into pain responses but are not standalone diagnostic tools. Best Practices: ○ Use psychophysiological tools (e.g., EMG, EEG, autonomic measures) as supplements to: Self-reports. Behavioral assessments. ○ Helps to capture a more comprehensive understanding of the patient’s pain experience.