Intro to Spinal Cord Injury PDF
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Lincoln Memorial University
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This document is an introduction to spinal cord injury. It describes the damage to the spinal cord, reviews different spinal tracts, and details etiology, mechanisms of injury, and relevant clinical aspects.
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**Intro to Spinal Cord Injury** **What is a SCI?** - **Damage** to the **spinal cord** resulting in **symptoms below the level of injury** **Review of Spinal Tracts** - **DCML: sensory (ascends)** - functions: proprioception, vibration, fine touch, and pressure - crosses at the med...
**Intro to Spinal Cord Injury** **What is a SCI?** - **Damage** to the **spinal cord** resulting in **symptoms below the level of injury** **Review of Spinal Tracts** - **DCML: sensory (ascends)** - functions: proprioception, vibration, fine touch, and pressure - crosses at the medulla - located at the dorsal column of the spinal cord - **Spinal-Thalamic Tract: sensory (ascends)** - Functions: pain, temperature, crude touch - crosses immediately - located at the anterolateral portion of the spinal cord - **Corticospinal Tract: motor (descends)** - Functions: controls voluntary movement from the neck to the feet - Base of the pyramids (medulla) - located at the anterior and lateral (largest) portions of the spinal cord **Spinal Cord Cross Section** - **Cervical**: involves brachial plexus (arms affected\> legs) and located medially - **Sacral**: more lateral and affects legs\> arms A diagram of the spinal cord **Etiology** - 18,000 new cases annually in the USA - Currently between 300,000-2 million people with SCI in the US - Most common **between ages 16-30, 19 the most, why?** - Involves frontal lobe development and myelination - Can be traumatic or non-traumatic - Most traumatic incidents occur with accidents and risk-taking behavior - 80% male and 20% female **Mechanisms of Injury: Traumatic** - MVA- 38% - Falls (elderly)- 32% - Violence- 14% - Sports-related injuries- 9% **Mechanisms of Injury (Non-traumatic)** - Arterial venous malformation (AVM) - Thrombus, embolus, or hemorrhage to arterial supply of the spinal cord (stroke of a vessel) - Infection of the cord (common setting of IV drug abuse) - Tumor - MS with lesions to the spinal cord - ALS - Spinal stenosis: less space + fall will squish the space **Life Expectancy** - **Incomplete** (sensory and motor intact) longer than **complete** (no sensory or motor) - **Paraplegia** (2 limbs/trunk, legs, and pelvic organs) longer than **tetraplegia** (4 limb paralysis) - **Lower cervical tetraplegia** longer than **higher cervical tetraplegia,** why? - Diaphragm C3-C5 - **Mortality rate** is highest in the 1^st^ year after injury **Spinal Shock** - Happens immediately after SCI: **flaccid-full body** - A period of **areflexia** that last **24hrs** - Reflexes return **gradually over 1-3 days** - Can have **hyperreflexia for 1-4 weeks** after that - Should perform ASIA **within 72 hrs** after injury - **NO ASIA the first 24 hrs**, could show up as a complete SCI, but if too late could be hyperreflexia **Naming System** 1. Spinal level of injury 2. Anatomical location of injury in cord 3. Completeness of injury ![A diagram of a diagram Description automatically generated with medium confidence](media/image2.jpeg) Sensory LOI: DCML and STT A medical chart with a human body Description automatically generated\ **Sensory Scale** 2= intact 1= diminished 0= absent NT = Not Testable **Motor Scale** 0= total paralysis 1= palpable or visible contraction 2= active movement, gravity eliminated 3= active movement, against gravity 4= active movement, against some resistance 5= active movement, against full resistance 5\*= normal corrected for pain/disuse NT= Not Testable **Main Levels** **UE** C5: elbow flexors C6: wrist extensors: **\*\*\*Clinical importance**- \"tenodesis grip\" is where the fingers naturally flex and close when the wrist is extended, allowing individuals with limited hand function, to grasp objects by simply moving their wrist; essentially using the passive tension of the flexor tendons to achieve a grip without active finger flexion C7: elbow extensors C8: finger flexors T1: finger abductors (little finger) **LE** L2: hip flexors L3: knee extensors L4: Ankle DFs L5: Big Toe extensors S1: Ankle PFs - If there is no DAP or anal contraction = complete SCI ASIA A **ASIA Impairment Scale** A= **Complete**, Complete: **no motor or sensory** function is preserved in the sacral segments **S4-S5** B= **Incomplete**: **sensory but no motor** function is preserved **below** the **neurological level** and includes the sacral segments **S4-S5** C= **Incomplete**: **motor function** is preserved **below the neurological level**, and **more than ½** of key mm. below the neurological level have a **mm. grade less than 3 (2's & 1's)** - Maybe walker! D= **Incomplete**, **motor function** is preserved below the neurological level, and **at least ½** of key mm. below the neurological level have a **mm. grade of 3 or more (3's-5's)** - For sure walker! E= **Normal**: motor and sensory function is normal **Motor Level of Injury** - Determined by test 10 key mm. groups on L and R sides of body - "The lowest myotome that has a grade of at least a 3 if the one above it is a 5" - Example: **L2 Myotome (Hip Flexion)**: 5 (Normal strength) - **L3 Myotome (Knee Extension)**: 4 (Weak, but still able to move against gravity and some resistance) - **L4 Myotome (Ankle DF)**: 3 (Able to move against gravity, but not full resistance) In this example, the **L4 myotome** (Ankle DF) is the lowest myotome with a grade of **at least a 3**, and the myotome above it (L3) has a grade of **4**. This aligns with your criteria of wanting the lowest myotome with at least a 3, assuming the myotome above it is a 5. **Possible injury level: L3 or L4** - The injury is likely at or just above **L3**, because that\'s where strength starts to decrease below normal levels. - If the injury is at L3, the person would still have functional strength at L2, but L3 and below would be affected. - If the injury is at L4, it could be due to a loss of strength below that level, particularly affecting muscle groups below the L4 myotome. **Summary:** The injury level could be at **L3**, **L4**, or slightly above L3 (but below L2) based on the strength pattern. In practice, the injury would likely be classified as a **L3 spinal cord injury**, with some muscle groups (L2-L3) still functional, and others (L4 and below) weakened or paralyzed. - Graded on 1-5 scale (not +/- grades) - May differ from L to R - May differ from sensory level of injury **Sensory Level of Injury (less important for function)** - Determined by **light touch** and **pin prick** on both the R and L side of the body - Graded on a 3 pt. scale: 0=absent, 1=impaired, 2=normal - "The most caudal level with normal light tough and pinprick sensation" - May differ L to R - May differ from motor level of injury **Neurologic Level of Injury-** "the most caudal level of the spinal cord with normal motor and sensory function both the R and L sides of the body **Zone of Partial Preservation** - Used to apply only to complete injuries (ASIA) and defined as: "dermatomes and myotomes caudal to the sensory or motor level that remain **partially innervated**" - Revised in 2019 to included incomplete - **Motor ZPP's** can be included in incomplete with absent Voluntary Anal Contraction **(VAC)** - **Sensory ZPP's** can be included in incomplete with absent Deep Anal Pressure **(DAP)** **Types** ![A diagram of the brain Description automatically generated](media/image4.jpeg) - **Posterior Cord Syndrome**: "slapping feet" presentation and B DCML sensory loss, trauma most likely - **Anterior Cord Syndrome**: B corticospinal (motor) and sensory (STT), blood flow issue or stroke occurs here usually - **Central Cord Syndrome**: most likely caused by trauma (ex.whiplash), have arm deficits (UEs), varying degrees of sensory impairment, sacral sparing - **Cauda Equina Syndrome**: LMN, flaccid paresis, saddle anesthesia, usually incomplete due to spinal n. - **Brown-Sequard Syndrome**: Ipsilateral loss of DCML, and CST, CL loss of STT - **Conus Medullaris**: Mixed LMN and UMN **LMN** **vs. UMN Injuries** - UMN: Above Conus Medullaris - LMN: Below Conus Medullaris - Can have a mixed presentation - Generally, T12 and below is most likely LMN or mixed A table with numbers and percentages Description automatically generated **LMN** - Generally below T12 - Hyporeflexia- cannot control sphincters - Flaccidity - Decreased tone/spasticity - Negative UMN signs - Flaccid bladder and bowel - Psychogenic responses for sexual function **UMN** - Generally above T12 - Hyperreflexia - Increased tone/spasticity - Positive UMN signs - Spastic or hyperreflexive bladder and bowel - Reflexogenic arcs for sexual function **Types of Settings** ![A comparison of medical information Description automatically generated with medium confidence](media/image6.jpeg) Acute care: beginning stages Acute Rehab: these patients have to learn how to everything over again LTACH: for people with chronic wounds or vented **Secondary Complications: Cardiovascular Pulmonary** - Pneumonia (PNA) - Aspiration - Diaphragmatic/respiratory muscle impairment - PE/DVT - BP Management **\*\*Orthostatic Hypotension**: in T6 and above injures (autonomics that control BP are affected) **Secondary Complications: Autonomic** - **Autonomic Dysreflexia**- can be fatal (T6 and above) why?- BP keeps increasing, pts. can stroke out with this condition bc of no parasympathetic response - BP Management- you want to get this patient up not lying down to help decrease - Sweating response - Loss of descending control of ascending sympathetic reflexes - Lack of inhibition from higher centers **Symptoms of Autonomic Dysreflexia** - HTN (raise of 20-30 mmHg systolic) - Bradycardia - Headache (severe and pounding) - Profuse sweating - Increased spasticity - Vasodilation above level of injury (leading to flushing) - Constricted Pupils - Nasal congestion - Piloerection - Blurred vision - Dry pale skin due to vasoconstriction (below level of injury) **Secondary Complications: Neurologic** - Tone/spasticity changes: UMN or LMN - Neuropathic pain - Sensory loss **Secondary Complications: MSK** - Motor Loss - Osteoporosis - Secondary Overuse Injuries - Heterotopic Ossification- abnormal bone grows into soft tissue - Osteomyelitis (in setting of pressure injury): inflammation of bone **Secondary Complications: Psychological (grief and loss)** - Adjustment to trauma and or loss - Higher depression rates - Higher psychiatric illness diagnoses post-injury - Higher health care utilization for psychiatric diagnosis **Secondary Complications: GI/GU** - **UTI:** leading type of infection following SCI - **Reflexive** bladder/bowel (UMN): tone is too much so they void - **Flaccid** bladder/bowel (LMN): happens with gravity **\*Maturation reflex:** bladder **\*** **Bulbocavernosus reflex:** bowel **Secondary Complications: Integumentary** - High risk for **pressure injuries** due to: - Decreased sensation - Decreased mobility - Decreased blood flow - Increased potential for incontinence - Staging: **Stage 1**: intact skin, **non-blanchable** **Stage 2**: **partial thickness** looks like a blister or scrape **Stage 3**: full thickness into the **subcutaneous** fat layer **Stage 4**: full thickness involving **muscle or bone** **Deep Tissue Pressure Injury**: persistent **non-blanchable** discoloration with a **dark wound bed** due to **prolonged pressure or shear**. **May evolve rapidly** to a stage 3 or 4 level (within minutes to hours) A close-up of a skin condition Description automatically generated![A diagram of skin disease Description automatically generated](media/image8.jpeg) A close-up of a wound Description automatically generated![A close-up of several stages of a wound Description automatically generated](media/image10.jpeg)**LTACH** A close-up of a tissue injury Description automatically generated **PT Management: Acute Care** - ICU main ?: Can they sit up? - Early mobility once medically stable - Focus exam: sensory/motor function/respiratory function, skin integrity, PROM, BP fluctuations **Intervention:** - PROM/contracture prevention - Skin prevention - BP management w/change in position (Reverse Trendelenburg to prevent shear) - Respiratory function - Education - Upright positioning: Tilt every 15 mins - Basic Mobility **PT Management: Acute Rehab (avg. 32 days for SCI)** **PT Exam:** - ROM, strength, outcome measures, functional mobility level **Interventions:** - Aerobic Capacity - Skin integrity/management: prevent secondary injury - **ADL's/functional mobility** - Pain/spasticity management - Education - Strengthening - **DME, w/c, and bracing needs** **LTACH** - Mobility as able - Focus exam on: sensory/motor function, respiratory function, skin integrity, PROM, BP fluctuations - PROM/contracture prevention - Skin prevention or treatment - BP management with change in position - Respiratory function - Education - Upright positioning - Basic Mobility **PT Management: Out-patient** **PT Exam:** - MSK/Neuro/CV/Pulm/Integument integrity - Knowledge of SCI and level of independence - Community reintegration/navigation - Goal directed activities: return to sport, childcare, work, etc. - Prevent MSK repetitive use injuries - Overall strengthening - CV endurance - Pain management ![A close-up of several people holding their hands Description automatically generated](media/image12.jpeg) A medical equipment instructions for wheelchairs Description automatically generated with medium confidence ![A diagram of a health condition Description automatically generated](media/image14.jpeg)