Podcast
Questions and Answers
What are the five classical signs of inflammation according to the text?
What are the five classical signs of inflammation according to the text?
- Redness, Heat, Swelling, Pain, Fever
- Redness, Dryness, Swelling, Pain, Heat
- Redness, Cold, Swelling, Pain, Numbness
- Redness, Heat, Swelling, Pain, Loss of function (correct)
Which classical sign of inflammation is described as 'Inflamed tissue appears red'?
Which classical sign of inflammation is described as 'Inflamed tissue appears red'?
- Swelling
- Redness (correct)
- Pain
- Heat
What causes the increase in temperature associated with the classical sign of 'Heat' in inflammation?
What causes the increase in temperature associated with the classical sign of 'Heat' in inflammation?
- Dilation of blood vessels and increased blood flow (correct)
- Loss of blood supply
- Decreased blood flow
- Absence of pain mediators
Which chemical mediators mentioned in the text are responsible for inducing pain in inflammation?
Which chemical mediators mentioned in the text are responsible for inducing pain in inflammation?
'Functio laesa,' a classical sign of inflammation, refers to:
'Functio laesa,' a classical sign of inflammation, refers to:
What triggers the vascular and cellular reactions of inflammation according to the text?
What triggers the vascular and cellular reactions of inflammation according to the text?
What is the purpose of inflammation according to Dr. C.Hand?
What is the purpose of inflammation according to Dr. C.Hand?
Which cellular infiltrate is mainly seen in acute inflammation?
Which cellular infiltrate is mainly seen in acute inflammation?
What distinguishes acute inflammation from chronic inflammation based on time course?
What distinguishes acute inflammation from chronic inflammation based on time course?
Which of the following is NOT a cause of acute inflammation?
Which of the following is NOT a cause of acute inflammation?
What type of tissue injury is usually associated with chronic inflammation?
What type of tissue injury is usually associated with chronic inflammation?
What is the main feature that differentiates acute from chronic inflammation based on cellular components involved?
What is the main feature that differentiates acute from chronic inflammation based on cellular components involved?
What is the main function of chemokines in the inflammatory response?
What is the main function of chemokines in the inflammatory response?
Which chemical mediator is responsible for smooth muscle contraction and pain during inflammation?
Which chemical mediator is responsible for smooth muscle contraction and pain during inflammation?
What is the mechanism of action of anti-inflammatory drugs like aspirin and NSAIDs?
What is the mechanism of action of anti-inflammatory drugs like aspirin and NSAIDs?
Which plasma factor directly targets killing of pathogens in the inflammatory response?
Which plasma factor directly targets killing of pathogens in the inflammatory response?
Stored in mast cells, basophils, and eosinophils, which early mediator causes immediate transient vascular permeability?
Stored in mast cells, basophils, and eosinophils, which early mediator causes immediate transient vascular permeability?
What role do prostaglandins and leukotrienes play in the inflammatory response?
What role do prostaglandins and leukotrienes play in the inflammatory response?
What is the main role of myeloperoxidase (MPO) in neutrophils?
What is the main role of myeloperoxidase (MPO) in neutrophils?
Which chemical mediators are responsible for increased vascular permeability and leukocyte adhesion?
Which chemical mediators are responsible for increased vascular permeability and leukocyte adhesion?
What is the role of Lysozyme in bacterial cells?
What is the role of Lysozyme in bacterial cells?
Which chemical mediator is responsible for causing vasodilation, pain, and fever?
Which chemical mediator is responsible for causing vasodilation, pain, and fever?
What does the presence of halide (Cl-) in neutrophils lead to?
What does the presence of halide (Cl-) in neutrophils lead to?
Which cells release cytokines like TNF, IL-1, and IL-6 during inflammation?
Which cells release cytokines like TNF, IL-1, and IL-6 during inflammation?
What is the hallmark of acute inflammation?
What is the hallmark of acute inflammation?
What happens to endothelial intracellular proteins under the influence of chemical inflammatory mediators?
What happens to endothelial intracellular proteins under the influence of chemical inflammatory mediators?
What type of fluid is characteristic of exudate in acute inflammation?
What type of fluid is characteristic of exudate in acute inflammation?
Which cellular event follows changes in vascular permeability during acute inflammation?
Which cellular event follows changes in vascular permeability during acute inflammation?
What is the purpose of fenestrations in endothelial cells?
What is the purpose of fenestrations in endothelial cells?
What leads to the escape of a protein-rich fluid into the interstitium during acute inflammation?
What leads to the escape of a protein-rich fluid into the interstitium during acute inflammation?
