Understanding Peritonitis

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Questions and Answers

What is the underlying mechanism by which peritonitis leads to hypovolemic shock?

  • Decreased renin production.
  • Vasoconstriction in response to inflammation.
  • Increased cardiac output due to fever.
  • Fluid shift from intravascular spaces to the peritoneal cavity. (correct)

Which of the following assessment findings would be the MOST concerning in a patient with peritonitis?

  • Distended abdomen and altered bowel sounds.
  • Abdominal pain and tenderness.
  • Fever and tachycardia.
  • Hypotension and altered mental status. (correct)

Why is it important to avoid administering morphine to patients who have gallstones contributing to acute pancreatitis?

  • Morphine can cause sphincter of Oddi spasm, increasing pancreatic duct pressure. (correct)
  • Morphine can worsen nausea and vomiting.
  • Morphine is contraindicated due to its potential to impair respiratory function.
  • Morphine may cause hypotension.

A patient with acute pancreatitis develops tetany. Which electrolyte imbalance is MOST likely the cause?

<p>Hypocalcemia. (D)</p> Signup and view all the answers

What is the rationale for aggressive hydration in the initial management of acute pancreatitis?

<p>To improve renal function and prevent acute kidney injury. (B)</p> Signup and view all the answers

Why are patients with acute pancreatitis, who are kept NPO (nothing by mouth), more susceptible to infections?

<p>Lack of enteral nutrition impairs gut barrier function and increases the risk of bacterial translocation. (D)</p> Signup and view all the answers

What finding in a patient with acute cholecystitis would MOST strongly suggest the development of gangrenous cholecystitis?

<p>Sudden decrease in pain with signs of systemic toxicity. (B)</p> Signup and view all the answers

Which statement best explains the rationale for using bile salts in the management of chronic cholecystitis?

<p>Bile salts assist with the digestion and absorption of fat-soluble vitamins (A, D, E, K). (C)</p> Signup and view all the answers

What is the underlying reason why patients with cholelithiasis may experience referred pain to the shoulder?

<p>Irritation of the phrenic nerve due to gallbladder inflammation. (A)</p> Signup and view all the answers

A patient is scheduled for a laparoscopic cholecystectomy. What specific instruction should the nurse provide about post-operative dietary management?

<p>Adhere to a low-fat diet and gradually introduce fatty foods as tolerated. (A)</p> Signup and view all the answers

Why is early intervention crucial for patients with a perforated bowel in the context of a large bowel obstruction (LBO)?

<p>To avoid sepsis and potentially fatal complications. (D)</p> Signup and view all the answers

In the management of a patient with a large bowel obstruction (LBO), what acid-base imbalance would you expect with persistent vomiting?

<p>Metabolic alkalosis due to loss of gastric acid. (B)</p> Signup and view all the answers

Which of the following interventions is MOST critical in managing a patient diagnosed with a large bowel obstruction (LBO) with signs of strangulation?

<p>Initiating broad-spectrum antibiotics and preparing the patient for immediate surgical intervention. (A)</p> Signup and view all the answers

How does the pathophysiology of a large bowel obstruction (LBO) contribute to hypovolemic shock?

<p>Fluid and electrolytes shifting into the peritoneal cavity reducing circulating blood volume. (A)</p> Signup and view all the answers

How does the presence of adhesions contribute to the pathophysiology of peritonitis?

<p>Adhesions can cause bowel obstruction and compromise blood supply, increasing the risk of perforation and peritonitis. (C)</p> Signup and view all the answers

Why is a ruptured appendix a common cause of secondary peritonitis?

<p>The appendix contains a high concentration of bacteria, which can contaminate the peritoneal cavity upon rupture. (A)</p> Signup and view all the answers

A patient with suspected peritonitis is scheduled for a paracentesis. Why is this procedure performed?

<p>To obtain a fluid sample for analysis and identify the causative agent of the infection. (B)</p> Signup and view all the answers

Which of the following is the MOST accurate description of 'obstipation' in the context of large bowel obstruction (LBO)?

<p>Complete or partial inability to pass stool or flatus. (B)</p> Signup and view all the answers

How does reduced fluid absorption contribute to the pathophysiology of large bowel obstruction (LBO)?

<p>It causes dehydration and electrolyte imbalances. (B)</p> Signup and view all the answers

What is the physiological basis for abdominal distension in large bowel obstruction (LBO)?

<p>Accumulation of fluid, gas, and intestinal contents proximal to the obstruction. (C)</p> Signup and view all the answers

What is the relationship between the serum amylase level and the serum lipase level in the diagnosis of acute pancreatitis?

<p>Serum lipase remains elevated longer while serum amylase is more specific. (A)</p> Signup and view all the answers

What is the significance of Cullen's sign and Grey Turner's sign in cases of acute pancreatitis?

<p>They indicate pancreatic necrosis and hemorrhage. (D)</p> Signup and view all the answers

Why is it essential to monitor blood glucose levels frequently in patients with acute pancreatitis?

<p>Pancreatitis leads to insulin resistance and can cause hyperglycemia. (A)</p> Signup and view all the answers

How does hypovolemia contribute to the development of complications in patients with acute pancreatitis?

<p>It reduces perfusion to the pancreas and other organs, increasing the risk of necrosis and organ failure. (D)</p> Signup and view all the answers

What is the rationale for placing a patient with severe cholecystitis on NPO status?

<p>To reduce stimulation of gallbladder contraction, thereby minimizing pain and preventing further inflammation. (A)</p> Signup and view all the answers

Which of the following best describes the pathophysiology behind jaundice in a patient with cholelithiasis?

<p>Obstruction of the bile ducts by gallstones, leading to the accumulation of bilirubin in the bloodstream. (C)</p> Signup and view all the answers

What is the underlying mechanism of fat intolerance in patients with chronic cholecystitis?

<p>Reduced bile secretion from the gallbladder, impairing fat emulsification and digestion. (B)</p> Signup and view all the answers

Why is it important to manage N/V in patients with cholelithiasis?

<p>To prevent fluid and electrolyte imbalances. (A)</p> Signup and view all the answers

What is the clinical significance of assessing the color of urine and stool in patients with suspected biliary obstruction due to cholelithiasis?

<p>To evaluate the patency of the bile ducts and assess the elimination of bilirubin. (A)</p> Signup and view all the answers

What is the primary reason why patients with cholelithiasis who undergo laparoscopic cholecystectomy are instructed to follow a low-fat diet postoperatively?

<p>To allow the digestive system to adjust to the absence of the gallbladder. (A)</p> Signup and view all the answers

What are the key differences between mechanical and non-mechanical causes of large bowel obstruction (LBO)?

<p>Mechanical obstructions involve a physical barrier, while non-mechanical obstructions result from impaired peristalsis. (C)</p> Signup and view all the answers

What is the underlying mechanism by which a strangulated large bowel obstruction (LBO) can lead to sepsis?

<p>Ischemia and necrosis of the bowel wall, leading to bacterial translocation into the bloodstream. (C)</p> Signup and view all the answers

Why is it crucial to assess the patient's vomitus for onset, amount, and odor in cases of suspected large bowel obstruction (LBO)?

<p>To identify the level and severity of the obstruction, as well as signs of bowel ischemia or necrosis. (B)</p> Signup and view all the answers

What is the rationale for using a nasogastric (NG) tube for decompression in the management of a large bowel obstruction (LBO)?

<p>To drain accumulated fluids and gas from the stomach and bowel, relieving pressure and distension. (B)</p> Signup and view all the answers

In the context of peritonitis, what is the rationale for administering intravenous fluids?

<p>To correct fluid deficits and maintain adequate perfusion due to fluid shifts and losses. (C)</p> Signup and view all the answers

What is the potential impact of increased intra-abdominal pressure (IAP) secondary to peritonitis on respiratory function?

<p>Restriction of diaphragmatic excursion, leading to decreased lung expansion and potential respiratory distress. (C)</p> Signup and view all the answers

In a patient with acute pancreatitis related to chronic alcohol use, what is the MOST likely mechanism by which alcohol contributes to pancreatic damage?

<p>Causes the formation of protein plugs and fibrosis in pancreatic ducts, obstructing enzyme outflow and leading to autodigestion. (C)</p> Signup and view all the answers

A patient with a large bowel obstruction (LBO) suddenly develops a rigid abdomen and rebound tenderness. Which of the following is the MOST likely explanation for this change in condition?

<p>The patient has developed peritonitis due to bowel perforation. (C)</p> Signup and view all the answers

A patient with cholelithiasis experiences referred pain in the right shoulder. What is the MOST accurate explanation for why this occurs?

<p>Gallstones are irritating the diaphragm, which shares a nerve pathway (phrenic nerve) with the shoulder. (C)</p> Signup and view all the answers

In a patient with peritonitis, what is the MOST significant rationale for closely monitoring serum electrolyte levels?

<p>To identify and correct imbalances caused by fluid shifts and third spacing. (D)</p> Signup and view all the answers

In the management of acute cholecystitis, why are bile salts sometimes administered, and what is the underlying mechanism of their therapeutic effect?

<p>Bile salts improve fat emulsification and absorption in the small intestine, reducing the workload on the inflamed gallbladder. (C)</p> Signup and view all the answers

Flashcards

Peritonitis

Inflammation of the peritoneum, can be caused by contents irritating the peritoneum or WBCs moving to the area due to irritation.

Pathophysiology & Etiology of Peritonitis

Contents irritate the peritoneum, compliment system activates, WBC's move to the area, fluid moves to area, adhesions develop in Intravascular spaces.

Signs & Symptoms of Peritonitis

Abdominal pain, fever, distended abdomen, altered bowel sounds, hypotension, tachycardia.

Complications of Peritonitis

Sepsis, ARDS, DIC, hypovolemic shock, metabolic alkalosis, abscess, bowel obstruction, respiratory distress.

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Labs & Diagnostics for Peritonitis

H&P, WBC (CBC), H&H, ACT, Peritoneoscopy, CT, US.

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Nursing Interventions for Peritonitis

Abdominal assessment, cold towel/remove blankets, high fall risk, orthostatics.

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Medical Interventions for Peritonitis

Pain meds & Tylenol, NG tube, IV fluids, Paracentesis.

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Secondary Peritonitis

Contents spill into the intestinal cavity.

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Patient Education for Peritonitis

Ruptured appendix, stab, ulcer perforation.

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Large Bowel Obstruction (LBO)

A blockage in the large intestine that prevents the passage of stool and gas.

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Pathophysiology of LBO

Fluid, gas, intestinal contents back up proximally, proximal bowel distention reduces fluid absorption & increases intestinal secretions, distal bowel empties & collapses.

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Complications of LBO

Fluid and electrolytes extravasate into the peritoneal cavity, decreasing circulating blood volume, causing hypotension & hypovolemic shock.

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Signs and symptoms of LBO

Abdominal pain, nausea/vomiting, distention, constipation.

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Labs & Diagnostics for LBO

H&P, X-rays, CT scan with contrast enema, LBO: sigmoidoscopy + colonoscopy, increased WBC-strangulation + perforation, increased HCT, serum electrolyte, BUN + creatinine.

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Nursing Management of LBO

Vomitus: onset, amount, odor, bowel function, strict I's & O's, ALID base imbalance.

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Nursing Interventions for LBO

Restore internal patency, emergency surgery, resection, partial & total colostomy or ileostomy, colonoscopy.

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Medical Interventions related to LBO

Remove polyps, dilates strictures, NG decompression, reduces bowel distention, IV hydration, PUN if obstructed for long time.

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Acute Pancreatitis

Inflammation of the pancreas, caused by spillage of pancreatic enzymes into surrounding pancreatic tissue, varies from mild edema to severe hemorrhagic.

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Pathophysiology and Etiology of Acute Pancreatitis

Spillage of pancreatic enzymes into surrounding pancreatic tissue, causing auto digestion + severe pain, varies from mild edema to severe hemorrhagic.

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Common cause of Pancreatitis

Gallbladder disease and chronic alcohol use.

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Signs and Symptoms of Acute Pancreatitis

Abdominal pain, radiates to back, sudden onset, deep, piercing, continuous, eating makes pain worse, starts when recumbent, not relieved with vomiting.

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Complications of Acute Pancreatitis

Pancreatic pseudocyst, pancreatic abscess, pulmonary complications, cardiovascular complications, hypocalcemia.

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Labs and Diagnostics for Acute Pancreatitis

Increased serum amylase level, increased serum lipase level, increased bilirubin & liver enzymes (AST & ALT), increased triglyceride level, increased glucose, decreased calcium.

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Nursing Interventions for Pancreatitis

Flex trunk + knees to belly, pain meds, morphine-NO.

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Medical Interventions for Acute Pancreatitis

Aggressive hydration, NPO, management of metabolic complications, minimizing pancreatic stimulation

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Cholelithiasis

Cholelithiasis is the formation of gallstones in the gallbladder.

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Pathophysiology of Cholelithiasis

Stones may stay in the gallbladder or migrate to the cystic or common bile duct, causing pain or obstruction.

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Signs and Symptoms of Cholelithiasis

Severe pain in the RUQ radiating to the shoulder, scapula, nausea, vomiting, indigestion, diaphoresis, rigidity, abdominal guarding.

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Risk Factors for Cholelithiasis

Female, multiparity, estrogen therapy, 40+, sedentary lifestyle, obesity, familial tendency, Native American ancestry, diet in saturated fat.

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Labs and Diagnostics for Cholelithiasis

Change in WBC, serum bilirubin level, urinary bilirubin, liver enzyme levels, elevated serum amylase of lipase level.

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Nutrition and Cholelithiasis

Small frequent meals with low fat intake.

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Medications for Cholelithiasis

Antibiotics, analgesics, anticholinergics, antiemetics, bile salts -assist with digestion and vit absorption cholestyramine or antihistamines.

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Surgical Nursing Interventions

Laparoscopic or Incisional cholecystectomy, t-tube.

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Post Op Nursing Interventions

Liquids to regular, assess and treat pain, encourage IS, PT comfort

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Complications of Cholelithiasis

Cholecystitis, gangrenous cholecystitis, gallbladder rupture, cholangitis and obstructive jaundice, pancreatitis, transaminitis, biliary cirrhosis.

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Study Notes

Peritonitis

  • Peritonitis is a disease where contents irritate the peritoneum
  • WBCs move toward the irritation
  • Fluid moves to the area and into intravascular spaces
  • Adhesions develop

Peritonitis Complications

  • Sepsis
  • ARDS
  • Disseminated intravascular coagulation
  • Hypovolemic shock
  • Metabolic alkalosis
  • Abscess
  • Bowel obstruction
  • Respiratory distress

Peritonitis Signs and Symptoms

  • Abdominal pain
  • Fever
  • Distended abdomen
  • Altered bowel sounds
  • Hypotension
  • Tachycardia

Labs and Diagnostics for Peritonitis

  • False elevation may occur due to fluid shift
  • Hemoglobin and hematocrit levels
  • White blood cell count
  • Peritoneoscopy
  • Computed tomography scan
  • Urinalysis

Nursing and Medical Interventions for Peritonitis

  • Perform abdominal assessment
  • Apply cold towels or remove blankets
  • Manage risk for falls
  • Monitor orthostatic vital signs
  • Administer pain medication and Tylenol
  • Nasogastric tube insertion
  • Intravenous fluids
  • Therapeutic paracentesis

Types of Peritonitis

  • Primary peritonitis is often blood-borne, related to ascites and cirrhosis
  • Secondary peritonitis happens where there's a perforation of an organ that spills contents into the internal cavity

Patient Education for Peritonitis

  • Ruptured appendix, stab wound, and ulcer perforation causes peritonitis

Large Bowel Obstruction (LBO)

  • A large bowel obstruction (LBO) can be nonmechanical or mechanical

LBO Pathophysiology and Etiology

  • Fluid, gas, and intestinal contents back up proximally causing proximal bowel distention
  • There is reduced fluid absorption and increased intestinal secretion
  • Distal bowel empties and collapses
  • There is increased pressure in the bowel lumen, leading to capillary permeability
  • Extravasation of fluids and electrolytes occurs into the peritoneal cavity

LBO Can Result in

  • Decreased circulating blood volume
  • Hypotension and hypovolemic shock

Signs and Symptoms of LBO

  • Can include metabolic alkalosis or metabolic acidosis
  • Gradual onset
  • Vomiting is rare
  • Persistent cramping and lower abdominal pain
  • No stool production
  • Complete or partial Obstipation
  • Abdominal distention becomes noticeable

Four Hallmark Symptoms of LBO

  • Abdominal pain
  • Nausea and vomiting
  • Distention
  • Constipation

LBO: Labs and Diagnostics Include

  • Bleeding may increase or decrease hematocrit
  • X-rays, CT scan, and contrast enema
  • Sigmoidoscopy and colonoscopy may be performed
  • White blood cell count may indicate strangulation or perforation

LBO: Nursing Interventions Include

  • Goal is to regain internal patency
  • Depending on the case: Emergency surgery, resection, partial or total colostomy or ileostomy, colonoscopy, NG decompression, IV hydration, Potential need for parenteral nutrition for long periods

Move These Medical Interventions from Nursing Interventions

  • Colonoscopy removes polyps or dilates strictures
  • NG Decompression to reduces bowel distention
  • IV Hydration
  • Parenteral Nutrition (PUN) if obstructed for long time

LBO: Complications

  • Perforated bowel requires immediate treatment to avoid sepsis and death

Acute Pancreatitis

  • Complication that can result in both pancreatic abscess and psuedocyst

Acute Pancreatitis Pathophysiology and Etiology

  • Includes acute inflammation and gallbladder stones blocking bile potentially
  • Spillage of pancreatic enzymes into surrounding pancreatic tissue causes autodigestion and severe pain
  • Varies from mild edema to severe hemorrhagic issues
  • There is less common a drug reaction that causes pancreacitits
  • Less common for pancreatic cancer to cause it

Acute Pancreatitis Can Be Cause by

  • Gallbladder disease
  • Chronic alcohol use
  • Hypertriglyceridemia (>1000)

Signs and Symptoms of Acute Pancreatitis

  • Abdominal pain predominates in the lower left upper quadrant (LLUQ) and mid-epigastric regions
  • Radiates to the back with sudden onset and deep, piercing, and constant pain
  • Pain worsens after eating and stays when lying down
  • Abdominal guarding
  • Cyanosis, dyspnea, and crackles

More Symptoms of Acute Pancreatitis

  • Nausea, vomiting
  • Possible absent bowel sounds
  • Abdominal guarding
  • Flushing skin, low-grade fever
  • Tachycardia and hypotension
  • Jaundice and abdominal skin discoloration
  • Grey Turner's or Cullen's sign

Acute Pancreatitis Labs and Diagnostics

  • Serum amylase level (most sensitive, elevates within 24 hours, return to normal after 5-7 days)
  • Serum lipase level (primary diagnostic marker, normal after 5-7 days), billburin, liver enzyme levels, triglyceride levels

Nursing Interventions for Acute Pancreatitis

  • Flex trunk and knees to belly or pain medication
  • Aggressive hydration at 250 mL/hr with intravenous lactated ringer (LR) or normal saline (NS)
  • Management of metabolic complications
  • Monitor blood glucose, minimizing stimmianon

NO Morphine

  • Use only one-of morphine for patients with gallstone

Frequent Oral and Nasal Care

  • Frequent oval and nasal care is necessary because patients is NPO
  • More susceptible to infection

Biliary Tract Disease: Cholelithiasis

  • Exact cause of gallstones unknown and they develop from cholesterol, bile salts bilirubin, Ca+, protein
  • Stones may stay in gallbladder or migrate to cystic or common bile ducts

Cholelithiasis Pathophysiology

  • Stones cause pain as pass through ducts
  • Stones that lodge in ducts will cause obstructions and that can cause further problems
  • Cholecystitis, obstructive jaundice, elevates lactic acid and pancreas depending on the location

Cholelithiasis: Choledocholithmiasis

  • Choledocholithmiasis = gallbladder obstruction of common bile duct

Biliary Tract Disease: Varies from Severe to None: Signs and Symptoms Can Include

  • Pain moves to severe when stones are moving or obstructing
  • 3-6 hrs of tor & fat meal or pt lies down
  • Steady excruciating
  • Tachycardia, diaphoresis, prostration
  • Pain shifts about Iwv, then residual tenderness in RUQ
  • Refereed to shoulder, scapula

Biliary Tract Disease Additional Signs and Symptoms

  • Indigestion
  • Nausea and vomiting
  • Abdominal distention and rigidity or guarding
  • Diaphoresis with liver congestion
  • Jaundice and pruritus
  • Fat intolerance
  • Dyspepsia
  • Heartburn and flatulence when total obstruction occurs, bile builds up bilirubin is not excreted so there is hyperbilirubinemia
  • If there is inflammation with fever and chills, indicates leukocytosis

Biliary Tract Disease: Risk Factors Include

  • Female, multiparty, estrogen therapy
  • 40
  • Sedentary lifestyle, obesity, and familial tendency
  • Diet is saturated fat, large, fatty meals or rapid weightloss
  • Avoid large, fatty meals and take in fiber and Ca+

Small Frequent Meals

  • Small frequent meals are always best avoid large meals and saturated fats
  • Give intravenous liquids to regain normal bowel sounds

Risk Factors After Laparoscopic Intervention

  • Light meals are needed for few days
  • If patients over weight, calories should be restricted by being careful
  • Liquids are needed
  • May need to restrict fats for 4-6 wks

Nursing Interventions for Biliary Tract Disease

  • Meds: Antibiotics, analgesics-opiods, anticholinergic or antimencl, bile salts, antihistamines
  • Monitor for infection or bleeding
  • Maintain fluid and electrolyte balance
  • Manage surgical drains, ensure that ducts have potency to allow bile to drain
  • Ensure patient is doing isometric exercises

Treat Biliary Tract Disease with Surgery

  • Laparoscopic cholecystectomy or Incisional cholecystectomy
  • T-tube insertion into common bile dou ensures parency and allows bile to drain

POST OP: VS: for Biliary Tract Disease

  • Monitor the vitals , assess and treat pain, encourage I.S. coughing

Patient Comfort: Biliary Tract Disease

  • Keep patient NPO and lower fat diet
  • Maintain reference for pain from to shoulder from carbon dioxide
  • Keep patient in Sims position

MORE Pathophysiology and Etiology

  • Cholecystitis is inflammation of the gallbladder most often associated with obstruction from stones
  • Can be acalculous
  • Older adults with other clinicallly-ill conditions

Potential Biliary Tract Disease Changes:

  • Involves only mucous lining or entire wall
  • Gallbladder is edematous
  • Hyperemic
  • Contain bile or pus
  • Cystic duck will become occulded
  • Scarring and fibrosis after attacks

Complications Cholelithiasis Can Cause

  • Cholelithiasis causes cholecystitis, subphrenic abscess, fistula, cholangitis and obstructive problems
  • May also lead to pancreatitis, transaminitis, and biliary cirrhosis

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