Podcast
Questions and Answers
What is the underlying mechanism by which peritonitis leads to hypovolemic shock?
What is the underlying mechanism by which peritonitis leads to hypovolemic shock?
- Decreased renin production.
- Vasoconstriction in response to inflammation.
- Increased cardiac output due to fever.
- Fluid shift from intravascular spaces to the peritoneal cavity. (correct)
Which of the following assessment findings would be the MOST concerning in a patient with peritonitis?
Which of the following assessment findings would be the MOST concerning in a patient with peritonitis?
- Distended abdomen and altered bowel sounds.
- Abdominal pain and tenderness.
- Fever and tachycardia.
- Hypotension and altered mental status. (correct)
Why is it important to avoid administering morphine to patients who have gallstones contributing to acute pancreatitis?
Why is it important to avoid administering morphine to patients who have gallstones contributing to acute pancreatitis?
- Morphine can cause sphincter of Oddi spasm, increasing pancreatic duct pressure. (correct)
- Morphine can worsen nausea and vomiting.
- Morphine is contraindicated due to its potential to impair respiratory function.
- Morphine may cause hypotension.
A patient with acute pancreatitis develops tetany. Which electrolyte imbalance is MOST likely the cause?
A patient with acute pancreatitis develops tetany. Which electrolyte imbalance is MOST likely the cause?
What is the rationale for aggressive hydration in the initial management of acute pancreatitis?
What is the rationale for aggressive hydration in the initial management of acute pancreatitis?
Why are patients with acute pancreatitis, who are kept NPO (nothing by mouth), more susceptible to infections?
Why are patients with acute pancreatitis, who are kept NPO (nothing by mouth), more susceptible to infections?
What finding in a patient with acute cholecystitis would MOST strongly suggest the development of gangrenous cholecystitis?
What finding in a patient with acute cholecystitis would MOST strongly suggest the development of gangrenous cholecystitis?
Which statement best explains the rationale for using bile salts in the management of chronic cholecystitis?
Which statement best explains the rationale for using bile salts in the management of chronic cholecystitis?
What is the underlying reason why patients with cholelithiasis may experience referred pain to the shoulder?
What is the underlying reason why patients with cholelithiasis may experience referred pain to the shoulder?
A patient is scheduled for a laparoscopic cholecystectomy. What specific instruction should the nurse provide about post-operative dietary management?
A patient is scheduled for a laparoscopic cholecystectomy. What specific instruction should the nurse provide about post-operative dietary management?
Why is early intervention crucial for patients with a perforated bowel in the context of a large bowel obstruction (LBO)?
Why is early intervention crucial for patients with a perforated bowel in the context of a large bowel obstruction (LBO)?
In the management of a patient with a large bowel obstruction (LBO), what acid-base imbalance would you expect with persistent vomiting?
In the management of a patient with a large bowel obstruction (LBO), what acid-base imbalance would you expect with persistent vomiting?
Which of the following interventions is MOST critical in managing a patient diagnosed with a large bowel obstruction (LBO) with signs of strangulation?
Which of the following interventions is MOST critical in managing a patient diagnosed with a large bowel obstruction (LBO) with signs of strangulation?
How does the pathophysiology of a large bowel obstruction (LBO) contribute to hypovolemic shock?
How does the pathophysiology of a large bowel obstruction (LBO) contribute to hypovolemic shock?
How does the presence of adhesions contribute to the pathophysiology of peritonitis?
How does the presence of adhesions contribute to the pathophysiology of peritonitis?
Why is a ruptured appendix a common cause of secondary peritonitis?
Why is a ruptured appendix a common cause of secondary peritonitis?
A patient with suspected peritonitis is scheduled for a paracentesis. Why is this procedure performed?
A patient with suspected peritonitis is scheduled for a paracentesis. Why is this procedure performed?
Which of the following is the MOST accurate description of 'obstipation' in the context of large bowel obstruction (LBO)?
Which of the following is the MOST accurate description of 'obstipation' in the context of large bowel obstruction (LBO)?
How does reduced fluid absorption contribute to the pathophysiology of large bowel obstruction (LBO)?
How does reduced fluid absorption contribute to the pathophysiology of large bowel obstruction (LBO)?
What is the physiological basis for abdominal distension in large bowel obstruction (LBO)?
What is the physiological basis for abdominal distension in large bowel obstruction (LBO)?
What is the relationship between the serum amylase level and the serum lipase level in the diagnosis of acute pancreatitis?
What is the relationship between the serum amylase level and the serum lipase level in the diagnosis of acute pancreatitis?
What is the significance of Cullen's sign and Grey Turner's sign in cases of acute pancreatitis?
What is the significance of Cullen's sign and Grey Turner's sign in cases of acute pancreatitis?
Why is it essential to monitor blood glucose levels frequently in patients with acute pancreatitis?
Why is it essential to monitor blood glucose levels frequently in patients with acute pancreatitis?
How does hypovolemia contribute to the development of complications in patients with acute pancreatitis?
How does hypovolemia contribute to the development of complications in patients with acute pancreatitis?
What is the rationale for placing a patient with severe cholecystitis on NPO status?
What is the rationale for placing a patient with severe cholecystitis on NPO status?
Which of the following best describes the pathophysiology behind jaundice in a patient with cholelithiasis?
Which of the following best describes the pathophysiology behind jaundice in a patient with cholelithiasis?
What is the underlying mechanism of fat intolerance in patients with chronic cholecystitis?
What is the underlying mechanism of fat intolerance in patients with chronic cholecystitis?
Why is it important to manage N/V in patients with cholelithiasis?
Why is it important to manage N/V in patients with cholelithiasis?
What is the clinical significance of assessing the color of urine and stool in patients with suspected biliary obstruction due to cholelithiasis?
What is the clinical significance of assessing the color of urine and stool in patients with suspected biliary obstruction due to cholelithiasis?
What is the primary reason why patients with cholelithiasis who undergo laparoscopic cholecystectomy are instructed to follow a low-fat diet postoperatively?
What is the primary reason why patients with cholelithiasis who undergo laparoscopic cholecystectomy are instructed to follow a low-fat diet postoperatively?
What are the key differences between mechanical and non-mechanical causes of large bowel obstruction (LBO)?
What are the key differences between mechanical and non-mechanical causes of large bowel obstruction (LBO)?
What is the underlying mechanism by which a strangulated large bowel obstruction (LBO) can lead to sepsis?
What is the underlying mechanism by which a strangulated large bowel obstruction (LBO) can lead to sepsis?
Why is it crucial to assess the patient's vomitus for onset, amount, and odor in cases of suspected large bowel obstruction (LBO)?
Why is it crucial to assess the patient's vomitus for onset, amount, and odor in cases of suspected large bowel obstruction (LBO)?
What is the rationale for using a nasogastric (NG) tube for decompression in the management of a large bowel obstruction (LBO)?
What is the rationale for using a nasogastric (NG) tube for decompression in the management of a large bowel obstruction (LBO)?
In the context of peritonitis, what is the rationale for administering intravenous fluids?
In the context of peritonitis, what is the rationale for administering intravenous fluids?
What is the potential impact of increased intra-abdominal pressure (IAP) secondary to peritonitis on respiratory function?
What is the potential impact of increased intra-abdominal pressure (IAP) secondary to peritonitis on respiratory function?
In a patient with acute pancreatitis related to chronic alcohol use, what is the MOST likely mechanism by which alcohol contributes to pancreatic damage?
In a patient with acute pancreatitis related to chronic alcohol use, what is the MOST likely mechanism by which alcohol contributes to pancreatic damage?
A patient with a large bowel obstruction (LBO) suddenly develops a rigid abdomen and rebound tenderness. Which of the following is the MOST likely explanation for this change in condition?
A patient with a large bowel obstruction (LBO) suddenly develops a rigid abdomen and rebound tenderness. Which of the following is the MOST likely explanation for this change in condition?
A patient with cholelithiasis experiences referred pain in the right shoulder. What is the MOST accurate explanation for why this occurs?
A patient with cholelithiasis experiences referred pain in the right shoulder. What is the MOST accurate explanation for why this occurs?
In a patient with peritonitis, what is the MOST significant rationale for closely monitoring serum electrolyte levels?
In a patient with peritonitis, what is the MOST significant rationale for closely monitoring serum electrolyte levels?
In the management of acute cholecystitis, why are bile salts sometimes administered, and what is the underlying mechanism of their therapeutic effect?
In the management of acute cholecystitis, why are bile salts sometimes administered, and what is the underlying mechanism of their therapeutic effect?
Flashcards
Peritonitis
Peritonitis
Inflammation of the peritoneum, can be caused by contents irritating the peritoneum or WBCs moving to the area due to irritation.
Pathophysiology & Etiology of Peritonitis
Pathophysiology & Etiology of Peritonitis
Contents irritate the peritoneum, compliment system activates, WBC's move to the area, fluid moves to area, adhesions develop in Intravascular spaces.
Signs & Symptoms of Peritonitis
Signs & Symptoms of Peritonitis
Abdominal pain, fever, distended abdomen, altered bowel sounds, hypotension, tachycardia.
Complications of Peritonitis
Complications of Peritonitis
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Labs & Diagnostics for Peritonitis
Labs & Diagnostics for Peritonitis
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Nursing Interventions for Peritonitis
Nursing Interventions for Peritonitis
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Medical Interventions for Peritonitis
Medical Interventions for Peritonitis
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Secondary Peritonitis
Secondary Peritonitis
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Patient Education for Peritonitis
Patient Education for Peritonitis
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Large Bowel Obstruction (LBO)
Large Bowel Obstruction (LBO)
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Pathophysiology of LBO
Pathophysiology of LBO
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Complications of LBO
Complications of LBO
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Signs and symptoms of LBO
Signs and symptoms of LBO
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Labs & Diagnostics for LBO
Labs & Diagnostics for LBO
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Nursing Management of LBO
Nursing Management of LBO
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Nursing Interventions for LBO
Nursing Interventions for LBO
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Medical Interventions related to LBO
Medical Interventions related to LBO
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Acute Pancreatitis
Acute Pancreatitis
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Pathophysiology and Etiology of Acute Pancreatitis
Pathophysiology and Etiology of Acute Pancreatitis
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Common cause of Pancreatitis
Common cause of Pancreatitis
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Signs and Symptoms of Acute Pancreatitis
Signs and Symptoms of Acute Pancreatitis
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Complications of Acute Pancreatitis
Complications of Acute Pancreatitis
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Labs and Diagnostics for Acute Pancreatitis
Labs and Diagnostics for Acute Pancreatitis
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Nursing Interventions for Pancreatitis
Nursing Interventions for Pancreatitis
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Medical Interventions for Acute Pancreatitis
Medical Interventions for Acute Pancreatitis
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Cholelithiasis
Cholelithiasis
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Pathophysiology of Cholelithiasis
Pathophysiology of Cholelithiasis
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Signs and Symptoms of Cholelithiasis
Signs and Symptoms of Cholelithiasis
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Risk Factors for Cholelithiasis
Risk Factors for Cholelithiasis
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Labs and Diagnostics for Cholelithiasis
Labs and Diagnostics for Cholelithiasis
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Nutrition and Cholelithiasis
Nutrition and Cholelithiasis
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Medications for Cholelithiasis
Medications for Cholelithiasis
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Surgical Nursing Interventions
Surgical Nursing Interventions
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Post Op Nursing Interventions
Post Op Nursing Interventions
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Complications of Cholelithiasis
Complications of Cholelithiasis
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Study Notes
Peritonitis
- Peritonitis is a disease where contents irritate the peritoneum
- WBCs move toward the irritation
- Fluid moves to the area and into intravascular spaces
- Adhesions develop
Peritonitis Complications
- Sepsis
- ARDS
- Disseminated intravascular coagulation
- Hypovolemic shock
- Metabolic alkalosis
- Abscess
- Bowel obstruction
- Respiratory distress
Peritonitis Signs and Symptoms
- Abdominal pain
- Fever
- Distended abdomen
- Altered bowel sounds
- Hypotension
- Tachycardia
Labs and Diagnostics for Peritonitis
- False elevation may occur due to fluid shift
- Hemoglobin and hematocrit levels
- White blood cell count
- Peritoneoscopy
- Computed tomography scan
- Urinalysis
Nursing and Medical Interventions for Peritonitis
- Perform abdominal assessment
- Apply cold towels or remove blankets
- Manage risk for falls
- Monitor orthostatic vital signs
- Administer pain medication and Tylenol
- Nasogastric tube insertion
- Intravenous fluids
- Therapeutic paracentesis
Types of Peritonitis
- Primary peritonitis is often blood-borne, related to ascites and cirrhosis
- Secondary peritonitis happens where there's a perforation of an organ that spills contents into the internal cavity
Patient Education for Peritonitis
- Ruptured appendix, stab wound, and ulcer perforation causes peritonitis
Large Bowel Obstruction (LBO)
- A large bowel obstruction (LBO) can be nonmechanical or mechanical
LBO Pathophysiology and Etiology
- Fluid, gas, and intestinal contents back up proximally causing proximal bowel distention
- There is reduced fluid absorption and increased intestinal secretion
- Distal bowel empties and collapses
- There is increased pressure in the bowel lumen, leading to capillary permeability
- Extravasation of fluids and electrolytes occurs into the peritoneal cavity
LBO Can Result in
- Decreased circulating blood volume
- Hypotension and hypovolemic shock
Signs and Symptoms of LBO
- Can include metabolic alkalosis or metabolic acidosis
- Gradual onset
- Vomiting is rare
- Persistent cramping and lower abdominal pain
- No stool production
- Complete or partial Obstipation
- Abdominal distention becomes noticeable
Four Hallmark Symptoms of LBO
- Abdominal pain
- Nausea and vomiting
- Distention
- Constipation
LBO: Labs and Diagnostics Include
- Bleeding may increase or decrease hematocrit
- X-rays, CT scan, and contrast enema
- Sigmoidoscopy and colonoscopy may be performed
- White blood cell count may indicate strangulation or perforation
LBO: Nursing Interventions Include
- Goal is to regain internal patency
- Depending on the case: Emergency surgery, resection, partial or total colostomy or ileostomy, colonoscopy, NG decompression, IV hydration, Potential need for parenteral nutrition for long periods
Move These Medical Interventions from Nursing Interventions
- Colonoscopy removes polyps or dilates strictures
- NG Decompression to reduces bowel distention
- IV Hydration
- Parenteral Nutrition (PUN) if obstructed for long time
LBO: Complications
- Perforated bowel requires immediate treatment to avoid sepsis and death
Acute Pancreatitis
- Complication that can result in both pancreatic abscess and psuedocyst
Acute Pancreatitis Pathophysiology and Etiology
- Includes acute inflammation and gallbladder stones blocking bile potentially
- Spillage of pancreatic enzymes into surrounding pancreatic tissue causes autodigestion and severe pain
- Varies from mild edema to severe hemorrhagic issues
- There is less common a drug reaction that causes pancreacitits
- Less common for pancreatic cancer to cause it
Acute Pancreatitis Can Be Cause by
- Gallbladder disease
- Chronic alcohol use
- Hypertriglyceridemia (>1000)
Signs and Symptoms of Acute Pancreatitis
- Abdominal pain predominates in the lower left upper quadrant (LLUQ) and mid-epigastric regions
- Radiates to the back with sudden onset and deep, piercing, and constant pain
- Pain worsens after eating and stays when lying down
- Abdominal guarding
- Cyanosis, dyspnea, and crackles
More Symptoms of Acute Pancreatitis
- Nausea, vomiting
- Possible absent bowel sounds
- Abdominal guarding
- Flushing skin, low-grade fever
- Tachycardia and hypotension
- Jaundice and abdominal skin discoloration
- Grey Turner's or Cullen's sign
Acute Pancreatitis Labs and Diagnostics
- Serum amylase level (most sensitive, elevates within 24 hours, return to normal after 5-7 days)
- Serum lipase level (primary diagnostic marker, normal after 5-7 days), billburin, liver enzyme levels, triglyceride levels
Nursing Interventions for Acute Pancreatitis
- Flex trunk and knees to belly or pain medication
- Aggressive hydration at 250 mL/hr with intravenous lactated ringer (LR) or normal saline (NS)
- Management of metabolic complications
- Monitor blood glucose, minimizing stimmianon
NO Morphine
- Use only one-of morphine for patients with gallstone
Frequent Oral and Nasal Care
- Frequent oval and nasal care is necessary because patients is NPO
- More susceptible to infection
Biliary Tract Disease: Cholelithiasis
- Exact cause of gallstones unknown and they develop from cholesterol, bile salts bilirubin, Ca+, protein
- Stones may stay in gallbladder or migrate to cystic or common bile ducts
Cholelithiasis Pathophysiology
- Stones cause pain as pass through ducts
- Stones that lodge in ducts will cause obstructions and that can cause further problems
- Cholecystitis, obstructive jaundice, elevates lactic acid and pancreas depending on the location
Cholelithiasis: Choledocholithmiasis
- Choledocholithmiasis = gallbladder obstruction of common bile duct
Biliary Tract Disease: Varies from Severe to None: Signs and Symptoms Can Include
- Pain moves to severe when stones are moving or obstructing
- 3-6 hrs of tor & fat meal or pt lies down
- Steady excruciating
- Tachycardia, diaphoresis, prostration
- Pain shifts about Iwv, then residual tenderness in RUQ
- Refereed to shoulder, scapula
Biliary Tract Disease Additional Signs and Symptoms
- Indigestion
- Nausea and vomiting
- Abdominal distention and rigidity or guarding
- Diaphoresis with liver congestion
- Jaundice and pruritus
- Fat intolerance
- Dyspepsia
- Heartburn and flatulence when total obstruction occurs, bile builds up bilirubin is not excreted so there is hyperbilirubinemia
- If there is inflammation with fever and chills, indicates leukocytosis
Biliary Tract Disease: Risk Factors Include
- Female, multiparty, estrogen therapy
- 40
- Sedentary lifestyle, obesity, and familial tendency
- Diet is saturated fat, large, fatty meals or rapid weightloss
- Avoid large, fatty meals and take in fiber and Ca+
Small Frequent Meals
- Small frequent meals are always best avoid large meals and saturated fats
- Give intravenous liquids to regain normal bowel sounds
Risk Factors After Laparoscopic Intervention
- Light meals are needed for few days
- If patients over weight, calories should be restricted by being careful
- Liquids are needed
- May need to restrict fats for 4-6 wks
Nursing Interventions for Biliary Tract Disease
- Meds: Antibiotics, analgesics-opiods, anticholinergic or antimencl, bile salts, antihistamines
- Monitor for infection or bleeding
- Maintain fluid and electrolyte balance
- Manage surgical drains, ensure that ducts have potency to allow bile to drain
- Ensure patient is doing isometric exercises
Treat Biliary Tract Disease with Surgery
- Laparoscopic cholecystectomy or Incisional cholecystectomy
- T-tube insertion into common bile dou ensures parency and allows bile to drain
POST OP: VS: for Biliary Tract Disease
- Monitor the vitals , assess and treat pain, encourage I.S. coughing
Patient Comfort: Biliary Tract Disease
- Keep patient NPO and lower fat diet
- Maintain reference for pain from to shoulder from carbon dioxide
- Keep patient in Sims position
MORE Pathophysiology and Etiology
- Cholecystitis is inflammation of the gallbladder most often associated with obstruction from stones
- Can be acalculous
- Older adults with other clinicallly-ill conditions
Potential Biliary Tract Disease Changes:
- Involves only mucous lining or entire wall
- Gallbladder is edematous
- Hyperemic
- Contain bile or pus
- Cystic duck will become occulded
- Scarring and fibrosis after attacks
Complications Cholelithiasis Can Cause
- Cholelithiasis causes cholecystitis, subphrenic abscess, fistula, cholangitis and obstructive problems
- May also lead to pancreatitis, transaminitis, and biliary cirrhosis
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