Types of Acute Inflammation

<p>The abscess may become chronic (B)</p> Signup and view all the answers

<p>Yellow color (D)</p> Signup and view all the answers

<p>They attract larger numbers of leukocytes (C)</p> Signup and view all the answers

<p>It transforms into a chronic abscess structure (C)</p> Signup and view all the answers

<p>Proteins and dead cell debris (C)</p> Signup and view all the answers

<p>Catarrhal inflammation (A)</p> Signup and view all the answers

<p>Fibrinous inflammation (A)</p> Signup and view all the answers

<p>Watery fluid (A)</p> Signup and view all the answers

<p>Membranous inflammation (A)</p> Signup and view all the answers

<p>Hemorrhagic inflammation (A)</p> Signup and view all the answers

<p>Marked tissue necrosis (A)</p> Signup and view all the answers

<p>Eosinophils (A)</p> Signup and view all the answers

<p>Shaggy appearance resembling bread and butter (A)</p> Signup and view all the answers

<p>Multiple communicating deep abscesses. (D)</p> Signup and view all the answers

<p>Thick skin areas like the back of the neck. (C)</p> Signup and view all the answers

<p>Streptococcus haemolyticus. (C)</p> Signup and view all the answers

<p>Abscesses contain localized pus whereas cellulitis has a diffuse distribution. (D)</p> Signup and view all the answers

<p>Hot, red, edematous, and shiny skin. (B)</p> Signup and view all the answers

<p>Secretion of streptokinase, hyaluronidase, and fibrinolysin. (A)</p> Signup and view all the answers

<p>Localized and often throbbing pain. (B)</p> Signup and view all the answers

<p>Cellulitis. (B)</p> Signup and view all the answers

<p>Initial accumulation of fibrin followed by serous fluid (A)</p> Signup and view all the answers

<p>Formation of fibrous adhesions if fibrin is not cleared (A)</p> Signup and view all the answers

<p>Formation of a thick, adherent membrane over the inflamed area (C)</p> Signup and view all the answers

<p>Fibrin network combined with necrotic tissue and pathogens (B)</p> Signup and view all the answers

<p>Presence of red blood cells due to capillary wall destruction (C)</p> Signup and view all the answers

<p>Necrotizing inflammation (D)</p> Signup and view all the answers

<p>Eosinophils (D)</p> Signup and view all the answers

<p>An antigen-antibody reaction specific to a sensitized individual (A)</p> Signup and view all the answers

<p>The abscess becomes chronic (B)</p> Signup and view all the answers

<p>A blind ended tract between the abscess cavity and the skin surface (C)</p> Signup and view all the answers

<p>Central area of necrosis and liquefaction (D)</p> Signup and view all the answers

<p>A large scar projecting on the surface (D)</p> Signup and view all the answers

<p>Serous inflammation (A)</p> Signup and view all the answers

<p>Fistula (C)</p> Signup and view all the answers

<p>An ongoing infection with thick pus (D)</p> Signup and view all the answers

<p>Pseudomembranous inflammation (B)</p> Signup and view all the answers

<p>Furuncle (C)</p> Signup and view all the answers

<p>Mild or prolonged irritant (D)</p> Signup and view all the answers

<p>Prolonged exposure to silica (C)</p> Signup and view all the answers

<p>Formation of a sinus (D)</p> Signup and view all the answers

<p>Lymphocytes and macrophages (A)</p> Signup and view all the answers

<p>Chronic inflammation features scanty fluid exudates. (B)</p> Signup and view all the answers

<p>Acute inflammatory response (B)</p> Signup and view all the answers

<p>Endarteritis obliterans with thick walls (C)</p> Signup and view all the answers

An accumulation of pus in a localized area, resulting from a bacterial infection.

Pus consists of bacteria, toxins, necrotic tissue, inflammatory fluid, and dead white blood cells.

An abscess has three zones: central necrosis (dead tissue), a middle zone of pus, and a peripheral zone of inflammation.

Pus is thick, creamy, alkaline, yellow, and odorless (unless caused by certain bacteria).

Abscesses grow by further liquefaction of necrotic tissue, increasing pressure inside.

Abscess healing involves the replacement of pus with granulation tissue.

If pus is not drained, the inflammation persists, becoming chronic and thick.

Complications include chronic infection, spread to other areas, and issues during healing.

Localized suppurative inflammation with multiple communicating deep abscesses, open on the surface by sinuses.

Usually Staphylococcus aureus, especially in diabetic patients.

Diffuse suppurative inflammation of loose subcutaneous tissues.

Streptococcus haemolyticus, which breaks down tissue to spread infection.

Localized collection of pus within a tissue cavity.

Abscess shows a well-defined swelling with redness and tenderness, while cellulitis has diffuse redness, swelling, warmth, and pain.

Abscess has a clear collection of pus, while cellulitis has thin, slowly formed pus.

Abscess has clear and localized borders, cellulitis has poorly defined, more diffuse borders.

The membrane of an abscess, composed of a central necrosis area, a liquefaction area, and a peripheral inflammation zone.

A blind-ended tract of infected granulation tissue between an abscess and the skin surface.

An infected granulation tissue tract between an abscess cavity and a hollow organ, or between two hollow organs.

A persistent loss of surface tissue continuity.

A large scar that projects from the skin surface, caused by excessive healing.

A small abscess related to hair follicles or sebaceous glands, commonly caused by Staphylococcus aureus.

Complications arising from abscesses such as bacteremia, septicemia, and pyemia

A type of inflammation where fibrin and fluid accumulate in the affected area, starting dry with more fibrin and becoming wet as more fluid collects.

Serofibrinous inflammation can either resolve (fibrin destroyed and fluid absorbed) or lead to fibrous adhesion (fibrin not removed).

A severe type of acute inflammation where a thick, dirty, adherent pseudomembrabe forms on the inflamed surface.

Membranous inflammation occurs when the causative organism secretes toxins, causing necrosis of the mucosa, leading to fibrin and cell accumulation forming a pseudomembrane.

Acute inflammation characterized by exudates rich in red blood cells due to damaged capillary walls.

Acute inflammation with severe tissue necrosis, characterized by marked cell death.

Inflammation triggered by an antigen-antibody reaction, causing excess fluid with high eosinophil count.

Examples of allergic inflammation include eczema (allergic dermatitis) and allergic rhinitis.

Inflammation where the exudate is rich in mucus, often seen in mucous membranes. It's usually mild.

Inflammation characterized by an exudate rich in fibrin. This is often seen in serous membranes like the pleura, pericardium, and peritoneum, as well as in the alveoli of the lungs.

Inflammation where the exudate is mainly watery fluid, seen in serous membranes and burns.

Inflammation where a pseudomembrane forms over the inflamed area. This membrane is made of fibrin, dead cells, and inflammatory cells. It is often seen in diphtheria and dysentery.

Burn blisters are an example of serous inflammation, characterized by fluid exudation from blood vessels.

Diphtheria is an example of pseudomembranous inflammation, characterized by a thick, yellowish-gray membrane forming on mucous membranes.

Chronic inflammation is a prolonged inflammatory process lasting for months or years, involving tissue destruction and healing attempts.

Chronic inflammation can be caused by persistent infections, prolonged exposure to toxins, or autoimmune reactions.

Chronic inflammation differs from acute inflammation by having a milder irritant, minimal tissue destruction, and scantly fluid exudates.

Chronic inflammation is characterized by lymphocytes, eosinophils, plasma cells, macrophages, fibroblasts, and foreign body giant cells.

Endarteritis obliterans (EAO) is a characteristic of chronic inflammation, involving thickened blood vessel walls and narrowed lumens.

In chronic inflammation, cells are often found around blood vessels (perivascular) or diffusely spread, which are lymphocytes, eosinophils, plasma cells, macrophages, fibroblasts, and foreign body giant cells