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Questions and Answers
What is considered the primary source of triglycerides (TGs) in the blood?
What is considered the primary source of triglycerides (TGs) in the blood?
- Muscle degradation
- Diet (correct)
- Liver synthesis
- Kidney filtration
Which lipoprotein type is expected to be elevated about 2 hours after a high-fat meal?
Which lipoprotein type is expected to be elevated about 2 hours after a high-fat meal?
- LDL
- Chylomicron (correct)
- HDL
- VLDL
Why should triglycerides (TGs) blood tests be performed in a fasting state?
Why should triglycerides (TGs) blood tests be performed in a fasting state?
- To avoid influence from liver metabolism
- To prevent false high readings from recent dietary intake (correct)
- To ensure hydration levels are constant
- To minimize hormonal fluctuations
How long after a meal will VLDL levels typically be elevated?
How long after a meal will VLDL levels typically be elevated?
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Which condition is NOT directly associated with hypertriglyceridemia?
Which condition is NOT directly associated with hypertriglyceridemia?
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Study Notes
Triacylglycerols (Triglycerides)
- Main source of triglycerides in the blood is from the diet
- Considered as an independent coronary heart disease risk factor
- Hypertriglyceridemia causes fatty liver disease and pancreatitis
Forms of Lipoproteins
- Triglycerides travel in the blood within two main forms of lipoproteins:
- VLDL (Very-Low-Density Lipoprotein)
- Chylomicron
Triglyceride Levels in Blood
- Value in the blood shows variation with:
- Age
- Sex
- Diet (elevated after a meal, especially a high-fat meal)
- Specific timing of elevation:
- Chylomicron: about 2 hours after a meal
- VLDL: about 4 hours after a meal
Laboratory Testing
- To obtain accurate results, test should be performed in a fasting state
- Single result interpretation is not dependable due to within-individual variation
Interpretation of Results
- Desirable: [no specific value mentioned]
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Description
Learn about the sources and effects of triglycerides in the blood, including their role in coronary heart disease, fatty liver disease, and pancreatitis.
