Pediatric Nursing: APGAR, High-Risk Newborns
31 Questions
0 Views

Choose a study mode

Play Quiz
Study Flashcards
Spaced Repetition
Chat to Lesson

Podcast

Play an AI-generated podcast conversation about this lesson

Questions and Answers

In a neonate diagnosed with Small for Gestational Age (SGA) exhibiting signs of polycythemia, which of the following pathophysiological mechanisms most directly contributes to the observed ruddy appearance?

  • Increased erythropoietin secretion due to chronic intrauterine hypoxia, leading to elevated red blood cell production and subsequent engorgement of dermal capillaries. (correct)
  • Increased capillary fragility secondary to vitamin K deficiency, leading to subcutaneous microhemorrhages that manifest as generalized erythema.
  • Compromised hepatic clearance of bilirubin as a result of immature liver function, causing bilirubin accumulation in the skin and imparting a reddish hue.
  • Peripheral vasoconstriction induced by hypothermia, resulting in pooling of blood in superficial vessels and a plethoric complexion.

An SGA neonate presents with a scaphoid abdomen. What physiological process is the MOST likely cause of this?

  • Increased intra-abdominal pressure displacing abdominal organs, creating a concave appearance.
  • Atrophy of abdominal musculature due to prolonged immobility, resulting in a sunken contour.
  • Congenital absence of abdominal muscles.
  • Diminished glycogen stores in the liver and reduced subcutaneous fat within the abdominal cavity. (correct)

An SGA infant exhibits persistent hypoglycemia despite early feedings and intravenous glucose administration. Which underlying metabolic adaptation common in SGA infants MOST likely contributes to this refractory hypoglycemia?

  • Impaired glycogenolysis and gluconeogenesis secondary to depleted hepatic glycogen stores and substrate deficiency. (correct)
  • Increased hepatic gluconeogenesis due to elevated cortisol levels.
  • Defective insulin secretion from pancreatic beta cells, hindering glucose utilization and promoting hyperglycemia.
  • Enhanced peripheral insulin receptor sensitivity, accelerating glucose uptake into peripheral tissues and exacerbating hypoglycemia.

In the context of Large for Gestational Age (LGA) neonates, which of the following physiological cascades best illustrates the pathogenesis of hypoglycemia, considering the interplay of hormonal regulation and metabolic demand?

<p>Exaggerated insulin secretion in response to chronic fetal hyperglycemia in utero, causing persistent suppression of hepatic glucose output and increased peripheral glucose utilization postnatally. (C)</p> Signup and view all the answers

For an SGA neonate exhibiting signs of meconium aspiration syndrome (MAS), what is the MOST critical initial nursing intervention to mitigate potential long-term respiratory sequelae?

<p>Immediate intubation and tracheal suctioning to remove meconium from the airways, followed by surfactant administration. (B)</p> Signup and view all the answers

An SGA neonate with a disproportionately large head and signs of asymmetric intrauterine growth restriction is at increased risk for which of the following long-term neurodevelopmental outcomes?

<p>Selective vulnerability of the brain to hypoxic-ischemic injury, predisposing to cerebral palsy and cognitive deficits. (B)</p> Signup and view all the answers

Considering the multifaceted risks associated with Large for Gestational Age (LGA) neonates, which of the following clinical presentations would necessitate the most immediate and aggressive intervention to prevent irreversible neurological sequelae?

<p>An LGA neonate displaying lethargy, hypotonia, and intermittent apneic episodes, accompanied by a blood glucose level of 25 mg/dL and arterial blood gas showing metabolic acidosis. (D)</p> Signup and view all the answers

What is the MOST appropriate target range for blood glucose in a 2-day-old neonate?

<p>50-90 mg/dL (C)</p> Signup and view all the answers

Given the increased risk of respiratory distress in LGA newborns, which of the following pathophysiological mechanisms is the most likely primary contributor to this complication?

<p>Relative deficiency of surfactant production due to delayed maturation of Type II pneumocytes, resulting in decreased alveolar stability and increased surface tension. (C)</p> Signup and view all the answers

In the context of managing hypoglycemia in an LGA neonate, which treatment strategy demonstrates the most comprehensive understanding of the underlying metabolic derangements and aims to provide sustained glycemic control?

<p>Early initiation of frequent feedings with expressed breast milk or formula, supplemented with a continuous intravenous infusion of D10W titrated to maintain euglycemia, while gradually weaning the infusion as tolerated. (C)</p> Signup and view all the answers

What is the primary rationale for closely monitoring SGA infants for apneic episodes during the initial days of life?

<p>Immature central respiratory drive and decreased chemoreceptor sensitivity, leading to periodic breathing and apnea. (A)</p> Signup and view all the answers

Considering the risk of hyperbilirubinemia in LGA newborns, which of the following factors is the least likely to contribute to the development of this condition?

<p>Decreased activity of heme oxygenase, the rate-limiting enzyme in the degradation of heme to biliverdin and carbon monoxide. (C)</p> Signup and view all the answers

An SGA neonate presents with tremors, cyanosis and an unstable temperature. Beyond immediate stabilization, what is the MOST crucial aspect of anticipatory guidance for parents regarding the long-term care of their child?

<p>Education on recognizing subtle signs of developmental delays and referral to early intervention programs to maximize developmental potential. (D)</p> Signup and view all the answers

Considering the maternal history factors associated with SGA infants, which of the following physiological mechanisms BEST explains how cardiovascular disease in the mother contributes to fetal growth restriction?

<p>Reduced maternal cardiac output and uteroplacental blood flow, impairing nutrient and oxygen delivery to the fetus. (D)</p> Signup and view all the answers

In the management of suspected clavicular fracture in a neonate, which diagnostic modality provides the most definitive confirmation while minimizing radiation exposure?

<p>Ultrasonography of the clavicle to visualize the fracture site and assess for periosteal hematoma formation. (C)</p> Signup and view all the answers

Which of the following interventions is least effective in preventing sepsis in LGA newborns?

<p>Prophylactic administration of broad-spectrum antibiotics to all LGA newborns immediately after birth, regardless of clinical signs of infection. (A)</p> Signup and view all the answers

What is the MOST likely reason why renal disease in the mother can lead to an SGA infant?

<p>Maternal hypertension and preeclampsia, leading to uteroplacental insufficiency and fetal growth restriction. (D)</p> Signup and view all the answers

Considering the long-term implications for LGA neonates, which of the following strategies demonstrates the most proactive approach to mitigating the risk of developing metabolic syndrome in adulthood?

<p>Provision of comprehensive education and support to parents, emphasizing the importance of a healthy diet, regular physical activity, and monitoring for early signs of insulin resistance and cardiovascular risk factors throughout the child's life. (A)</p> Signup and view all the answers

If a LGA newborn is noted to have shrill or intermittent cries and has difficulty being roused, which of the following conditions should be considered first?

<p>Hypoglycemia (D)</p> Signup and view all the answers

Given the potential for congenital anomalies in LGA neonates, what advanced diagnostic approach integrates genetic analysis with morphological assessment to provide a comprehensive evaluation of the newborn's condition?

<p>Whole-exome sequencing (WES) coupled with high-resolution fetal ultrasonography to detect subtle structural anomalies and identify underlying genetic mutations. (A)</p> Signup and view all the answers

In the context of Apgar scoring, which of the following statements MOST accurately reflects the clinical implications of a score of 6 at 5 minutes, and the subsequent management?

<p>The newborn is moderately depressed, necessitating immediate intervention, including positive pressure ventilation and potential pharmacological support, with continuous Apgar scoring every 5 minutes until a score of 7 or higher is achieved. (D)</p> Signup and view all the answers

A neonate, born large for gestational age (LGA) to a mother with poorly controlled gestational diabetes, exhibits tremors, cyanosis, and a blood glucose level of 30 mg/dL at one hour of age. Which of the following interventions is MOST critical in stabilizing this neonate, considering the interplay of potential complications?

<p>Administer a bolus of 10% dextrose intravenously, followed by a continuous infusion of 10% dextrose at a rate of 6 mg/kg/min, while simultaneously preparing for potential seizure management with phenobarbital. (D)</p> Signup and view all the answers

Considering the multifaceted risks associated with Large for Gestational Age (LGA) infants born to diabetic mothers, which of the following sets of clinical findings would warrant the MOST immediate and comprehensive diagnostic evaluation to differentiate between potential underlying etiologies?

<p>Persistent hypoglycemia despite dextrose infusion, hepatomegaly, and macroglossia, necessitating evaluation for Beckwith-Wiedemann syndrome and glycogen storage diseases. (A)</p> Signup and view all the answers

A preterm neonate born at 30 weeks gestation exhibits signs of respiratory distress syndrome. Given the understanding of potential complications in premature infants, which intervention would have the HIGHEST priority in the immediate management of this neonate?

<p>Providing continuous positive airway pressure (CPAP) to maintain alveolar patency and reduce the work of breathing, while carefully monitoring for signs of pneumothorax or air leak syndromes. (A)</p> Signup and view all the answers

An infant is diagnosed as Small for Gestational Age (SGA) due to placental insufficiency. Beyond immediate stabilization, which of the following long-term outcomes presents the GREATEST concern for this infant's developmental trajectory?

<p>Heightened risk of developing metabolic syndrome in adulthood, characterized by insulin resistance, dyslipidemia, and cardiovascular disease. (B)</p> Signup and view all the answers

A neonate born to a mother with gestational diabetes is exhibiting signs of hypocalcemia. Which of the following pathophysiological mechanisms BEST explains the etiology of hypocalcemia in this clinical context?

<p>Maternal hyperglycemia leads to fetal hyperinsulinemia, resulting in increased deposition of calcium in fetal bones and subsequent neonatal hypocalcemia due to abrupt cessation of maternal glucose supply. (A)</p> Signup and view all the answers

In managing a newborn diagnosed with a fractured humerus following a traumatic delivery, which of the following nursing interventions is MOST crucial in preventing long-term complications and promoting optimal healing?

<p>Immobilizing the affected limb with a splint or sling, ensuring proper alignment and preventing further injury, while closely monitoring for signs of compartment syndrome. (A)</p> Signup and view all the answers

Which of the following options BEST describes the complex interplay between genetic predispositions, maternal conditions, and neonatal outcomes in Large for Gestational Age (LGA) infants, warranting the most vigilant clinical monitoring?

<p>LGA infants with suspected genetic abnormalities like Beckwith-Wiedemann syndrome require monitoring for hypoglycemia, macroglossia, and hemihypertrophy, necessitating abdominal ultrasound to screen for Wilms tumor. (D)</p> Signup and view all the answers

A neonate presents with persistent respiratory distress, refractory to conventional oxygen therapy. The neonate was born via Cesarean section at 35 weeks gestation to a mother with poorly controlled gestational diabetes. Which of the following interventions should be implemented FIRST?

<p>Administer surfactant via endotracheal tube, followed by mechanical ventilation with permissive hypercapnia to minimize barotrauma and volutrauma. (A)</p> Signup and view all the answers

A Small for Gestational Age (SGA) neonate, born at term, exhibits signs of polycythemia. What chain of events culminates in this condition?

<p>Intrauterine growth restriction leads to chronic fetal hypoxia, stimulating erythropoietin production and resulting in increased red blood cell mass at birth. (D)</p> Signup and view all the answers

Signup and view all the answers

Flashcards

APGAR Scoring

Done at 1 & 5 minutes after birth. Assesses heart rate, respiratory effort, muscle tone, reflex irritability, and color. If <7 at 5 min, repeat at 10 min.

APGAR Score Interpretation

7-10 indicates a healthy newborn, 3-6 moderately depressed, and 0-2 severely depressed.

SGA (Small for Gestational Age)

Infants whose weight is below the 10th percentile for their gestational age.

LGA (Large for Gestational Age)

Infants whose weight is above the 90th percentile for their gestational age; often related to maternal diabetes.

Signup and view all the flashcards

Risks for LGA Infants

Birth trauma, respiratory distress, hypoglycemia, hyperbilirubinemia, and congenital anomalies are frequent complications.

Signup and view all the flashcards

Newborn of Diabetic Mother: Clinical Findings

Excessive size and weight, edema or puffiness in the face and cheeks, Hypocalcemia, Hyperbilirubinemia, Hypoglycemia, RDS

Signup and view all the flashcards

Fractured Humerus: Symptoms

Pain, tenderness, edema, and decreased movement.

Signup and view all the flashcards

Hypoglycemia in LGA Infants

LGA infants may develop hyperinsulinemia due to maternal hyperglycemia, leading to increased insulin production.

Signup and view all the flashcards

Premature Infant

Infants born before 37 weeks gestation.

Signup and view all the flashcards

High-Risk Newborn Categories

Includes SGA, LGA, premature infants, and those born to mothers with diabetes.

Signup and view all the flashcards

Broken Clavicle (Newborn)

Swelling, pain, tenderness, or reluctance to use the affected extremity in a newborn.

Signup and view all the flashcards

Nursing Care: LGA Newborn

Monitoring for signs of respiratory distress, shrill cries, lethargy, and poor muscle tone.

Signup and view all the flashcards

Large for Gestational Age (LGA)

A neonate whose weight is at or above the 90th percentile on the intrauterine growth curve.

Signup and view all the flashcards

Nursing Interventions for LGA

Monitor blood glucose levels, feeding, vital signs, and signs of hypoglycemia.

Signup and view all the flashcards

Hypoglycemia (Newborn)

An abnormally low level of glucose in the blood, < 30 mg/dl in the first 72 hours.

Signup and view all the flashcards

Monitor for Hypoglycemia

Monitor for tremors, seizures, apnea, and acidosis. Initiate early feeding and monitor for aspiration.

Signup and view all the flashcards

LGA Respiratory Distress

Monitor for signs of respiratory distress.

Signup and view all the flashcards

LGA Hyperbilirubinemia

Monitor bilirubin levels.

Signup and view all the flashcards

LGA Weight

Monitor weight.

Signup and view all the flashcards

LGA Feeding

Feed early with 10% glucose in water, breast milk, or formula as prescribed.

Signup and view all the flashcards

Hypoglycemia in SGA newborns

Blood glucose < 45 mg/dL in the first 3 days of life.

Signup and view all the flashcards

SGA newborn clinical findings

Fetal distress, temperature instability, hypoglycemia, polycythemia, infection signs, meconium aspiration.

Signup and view all the flashcards

Nursing interventions for Hypoglycemia

Provide early feedings, oral or IV glucose, monitor glucose levels, watch for feeding problems and apnea.

Signup and view all the flashcards

Physical signs of SGA

Lack of subcutaneous fat, scaphoid abdomen, disproportionately large head.

Signup and view all the flashcards

Signs of Polycythemia

Ruddy appearance, cyanosis, jaundice.

Signup and view all the flashcards

Maternal history risk factors for SGA

Cardiovascular disease, renal disease, diabetes.

Signup and view all the flashcards

Extremely Preterm Infant

Born earlier than 28 weeks.

Signup and view all the flashcards

Small for Gestational Age (SGA)

Smaller than expected weight based on gestational age.

Signup and view all the flashcards

Study Notes

  • These study notes cover pediatric nursing care for mothers and children at risk or with acute and chronic problems.

APGAR Scoring System

  • APGAR score assesses a newborn's condition at 1 and 5 minutes after birth.
  • A score of 7-10 indicates a healthy newborn.
  • A score of 3-6 indicates moderate depression.
  • A score of 0-2 indicates severe depression.
  • If the 5-minute APGAR score is less than 7, it should be performed at 10 minutes.

High-Risk Newborns

  • Categories include SGA (Small for Gestational Age), LGA (Large for Gestational Age), premature (<37 weeks gestation), and postmature (>42 weeks gestation).

LGA (Large for Gestational Age)

  • Can result from various obstetrical situations, including maternal diseases like diabetes or maternal genetic predispositions.
  • Genetic abnormalities such as Beckwith-Wiedemann Syndrome can lead to LGA infants.
  • Complications include birth trauma, hemorrhage, asphyxia, or even death.

Fractured Humerus (in LGA newborns)

  • Symptoms: pain, tenderness, edema, and decreased movement of the affected arm.

Hypoglycemia in LGA Infants

  • LGA infants can develop hyperinsulinemia due to maternal hyperglycemia.

SGA (Small for Gestational Age)

  • Infants whose weight is below the 10th percentile for gestational age.

Clinical Findings for LGA

  • Includes gestational age, birth trauma or injury, respiratory distress, hypoglycemia

Infants of Diabetic Mothers: Clinical Findings

  • Excessive size and weight.
  • Facial and cheek edema.
  • Hypocalcemia and hypoglycemia.
  • Respiratory distress signs: tachypnea, cyanosis, retractions, grunting, nasal flaring.
  • High incidence of congenital anomalies.
  • High incidences of hypoglycemia, respiratory distress, hypocalcemia, and hyperbilirubinemia.

Other Risk Injuries in LGA Newborns

  • Broken clavicle symptoms include swelling, pain, tenderness, or reluctance to use the affected extremity.

Nursing Interventions for High-Risk Newborns

  • Monitor for respiratory distress signs.
  • Monitor bilirubin and blood glucose levels.
  • Monitor weight.
  • Feed early: 10% glucose in water, breast milk, or formula.
  • Administer IV glucose for hypoglycemia if prescribed.
  • Monitor for edema, tremors, seizures, apnea, and acidosis.
  • Monitor for shrill or intermittent cries.
  • Evaluate lethargy.

LGA Specifics

  • LGA neonates plot at or above the 90th percentile on the intrauterine growth curve.

LGA: Nursing Interventions

  • Monitor vital and glucose levels.
  • Monitor for hypoglycemia signs.
  • Monitor infection signs.
  • Initiate measures to prevent sepsis or aspiration.
  • Provide stimulation through touching and cuddling.

Hypoglycemia

  • Abnormally low glucose levels in the blood: <30 mg/dl in the first 72 hours or <45 mg/dl after the first 3 days of life.
  • Normal glucose ranges: 40-60 mg/dl in a 1-day-old neonate or 50-90 mg/dl in a neonate older than 1 day.
  • Clinical findings: increased respiratory rate, twitching, nervousness, tremors, cyanosis, and unstable temperature.

Hypoglycemia: Nursing Interventions

  • Provide low blood glucose through early feedings.
  • Administer oral or IV glucose per orders.
  • Monitor blood glucose and for feeding problems.
  • Monitor for apneic episodes.

SGA: Clinical Findings

  • Fetal distress or temperature instability.
  • Hypoglycemia or polycythemia signs: ruddy appearance, cyanosis, jaundice.
  • Signs of infection or meconium aspiration.

SGA: Assessment

  • Physical signs: Lack of subcutaneous fat, scaphoid abdomen, disproportionately large head (asymmetric intrauterine growth retardation).
  • Maternal history: Cardiovascular or renal disease, diabetes, medications, substance use, or placental insufficiencies.
  • Neonatal causes: Inborn errors of metabolism, congenital anomalies, or multiple gestation.
  • Postnatal complications: Increased susceptibility to infection, respiratory compromise, neurological challenges, death, hypoglycemia, or risk of hypothermia.

SGA: Clinical Findings

  • Neonates plot at or below the 10th percentile on the intrauterine growth curve.

SGA: Nursing Interventions

  • Maintain airway and body temperature.
  • Observe for respiratory distress signs.
  • Monitor for infection and prevent sepsis.
  • Monitor glucose levels and signs of hypoglycemia.
  • Initiate feeding and watch for aspiration.
  • Provide stimulation via touching and cuddling.

Premature Infant

  • Born earlier than 37 weeks of gestation.
  • Extremely preterm: <28 weeks.
  • Very preterm: 28-32 weeks.
  • Moderate to late preterm: 32-37 weeks.

Premature Infant: Statistics

  • Globally 15 million babies are born too early each year.
  • Over 1 million children die annually from preterm complications.

Premature Infant: Risk Factors

  • Maternal hypertension or preeclampsia
  • Multiple gestation or renal/cardiac disease.
  • History of preterm labor.
  • Substance use and abuse
  • Late initiation of prenatal care.
  • Lower socioeconomic level

Premature Infant: Complications

  • Hypoglycemia or Respiratory instability.
  • Neurological problems and necrotizing enterocolitis.
  • Respiratory Distress Syndrome (RDS).

Premature Infant: Clinical Findings

  • Irregular respirations or temperature below normal.
  • Feeding: Poor suck and swallow reflexes.
  • Gastrointestinal: Diminished bowel sounds.
  • Urinary output: Increased or decreased.
  • Physical Appearance: Thin extremities, minimal creasing on soles and palms, skin thin with visible blood vessels, may appear jaundiced.
  • Genitalia: Undescended testes in boys or narrow labia in girls.

Premature Infant: Primary concerns

  • Immaturity of all body systems

Premature Infant: Interventions

  • Monitor vitals every 2 hours.
  • Maintain cardiopulmonary functions.
  • Administer oxygen and humidification.
  • Monitor intake and output and electrolyte balance.
  • Monitor daily weight or maintain warming device.
  • Position carefully.
  • Avoid exposure to infections; provide stimulation.

Post-mature Infant

  • Defined as a neonate born after 42 weeks of gestation
  • Placenta loses its ability to transport nutrients, oxygen, and waste products.
  • Problems: Meconium aspiration, malnutrition, asphyxia, death.
  • Assessment: Dry infant with crackling skin, absence of vernix and lanugo, long/thin extremities.
  • Clinical Findings: Hypoglycemia, parchment-like skin without lanugo, long fingernails and thin body, wasting of fat; meconium staining.

Post-mature Infant: Interventions

  • Provide normal newborn care and monitor temperature.
  • Monitor for hypoglycemia or meconium aspiration.
  • Monitor I&O and electrolyte balance.

Respiratory Distress Syndrome (RDS)

  • Respiratory dysfunction in neonates due to delayed lung maturation or lack of surfactant.
  • Surfactant reduces surface tension in alveoli, thereby aiding the lungs to remain open during exhalation.
  • RDS can also arise from non-pulmonary issues like sepsis, cardiac defects or airway obstruction.
  • Preterm infants born before lungs are ready for gas exchange are prone to RDS.
  • Impaired secretion of surfactant results in uneven expansion and alveolar collapse.

RDS: Contributing Factors

  • Includes immaturity of chest wall and lung, inadequate pulmonary surfactant, reduced blood flow, and interstitial or alveolar lung water.
  • Risk Factors: Prematurity, maternal diabetes, genetic factors, perinatal asphyxia, second-born twins, and cesarean section without labor

RDS: Clinical Manifestations

  • Tachypnea (80-100 breaths/min), dyspnea, retractions, fine crackles, expiratory grunt, cyanosis or metabolic acidosis, or elevated CO2

RDS: More Severe Manifestations

  • Pallor and cyanosis, poor muscle tone and response, or specific chest X-ray findings

RDS: Therapeutic Management

  • Maintain ventilation, acid-base balance, and a neutral thermal environment; ensure tissue perfusion, and avoid hypotension.
  • Maintain hydration and electrolyte balance or administer betamethasone to enhance the production of surfactant.

RDS: Medical Management

  • Includes ventilatory and antibiotic support, surfactant administration, and provision of intravenous fluids as needed for nutrition or hydration

RDS: Complications

  • Includes Pneumothorax, Bronchopulmonary Dysplasia (BPD), Retinopathy of Prematurity (ROP), and Hyperbilirubinemia
  • Inhaled Nitric Oxide (INO) supports pulmonary function.

RDS: Nursing Diagnoses

  • Ineffective Airway Clearance or Breathing Pattern or Impaired Gas Exchange or Risk for Trauma.
  • Interventions: Providing appropriate stimulation, avoiding infection or oxygen administration.

Post-mature Infant

  • This is a neonate born after 42 weeks of gestation.
  • The placenta loses its ability to transport nutrients, oxygen, and waste products.
  • Problems: Meconium aspiration, malnutrition, asphyxia, death
  • Signs: Dry and cracked skin, absence of vernix and lanugo, long and thin extremities.
  • Clinical Findings: Hypoglycemia, parchment-like skin without lanugo and long fingernails, muscle wasting.

Post-mature Infant: Nursing Interventions

  • Provide normal newborn care
  • Monitor for hypoglycemia or meconium aspiration.
  • Monitor temperature and I&O.

Sudden Infant Death Syndrome (SIDS)

  • Sudden and unexplained death of child younger than 1 year.
  • Happens even after post-mortem investigation, death scene investigation, and case history review.
  • The cause is unknown.
  • Risk Factors: Prone sleeping position, maternal smoking, brainstem abnormality, and socioeconomic factors.
  • Pathologic Findings: Pulmonary edema and intrathoracic hemorrhage.

SIDS: Interventions

  • Monitor jaundice and skin color in natural light.
  • Promote hydration to maintain blood volume and encourage frequent feeding.
  • Educate families using supine sleeping position and a safe sleep environment and discouraging smoking and bed-sharing.

Hyperbilirubinemia Interventions

  • Inspect skin in natural light
  • Encourage frequent feedings to get rid of meconium

Retrolental Fibroplasia (ROP)

  • Condition affecting retina of premature infants.

Necrotizing Enterocolitis (NEC)

  • Serious gastrointestinal condition affecting preterm infants.

Preterm Newborn: Interventions

  • Monitor respiratory rate and color, support respirations, and monitor blood gases.
  • Schedule eye exams, suction, and administer surfactant.

Preterm Newborn: Nursing Interventions

  • Position the infant for stability or provide nutrition.

Common complications of RDS

  • Pneumothorax
  • Bronchopulmonary Dysplasia (BPD)
  • Retinopathy of Prematurity (ROP)

Erythroblastosis Fetalis

  • This is a condition characterized by the destruction of red blood cells.

Erythroblastosis Fetalis: Clinical Findings

  • Anemia, jaundice, enlarged placenta, edema, and ascites.

Erythroblastosis Fetalis: Nursing Interventions

  • Perform early blood typing and Rh testing to determine the mother's blood type and administer Coombs' test during pregnancy.

Erythroblastosis Fetalis: Treatment

  • Provide phototherapy
  • Administer Rho(D) immune globulin.

Neonatal Sepsis

  • Invasive infection that primarily involves bacteria invading the bloodstream, and can have severe consequences for newborns.
  • May develop either early(within 24 hours) or late (after 24 hours).

Neonatal Sepsis: Factors

  • Prematurity and Low Birth Weight or Maternal Infections.
  • Early Onset Sepsis: Occurs within the first 24 hours after birth, due to infections passed down from mother.
  • Late Onset Sepsis: Develops after 24 hrs, due to hospital-acquired infections.
  • Treatment includes antibiotic therapy or supportive therapy.

Common Pathogens causing Neonatal Sepsis

  • Bacterial Infections
  • Viral Infections
  • Fungal Infections

Nursing Interventions

  • Monitor Vital Signs and respiratory function closely.
  • Fluid and Electrolyte Management.
  • Monitor Organ Function.

Statistics & Global Impact

  • Sepsis is a leading cause of death among children under the age of five across the globe.

Therapeutic Management

  • Maintain ventilation and hygiene
  • Administer Antibiotics

Symptoms of Neonatal Sepsis

  • Body temperature changes.(fever or hypothermia).
  • Breathing problems. (apnea, tachypnea)
  • Jaundice (yellow skin and whites of the eyes).

Neonatal Sepsis:: Prevention and Early Recognition:

  • Prenatal care and infection control.

Late-Onset Sepsis: Prevention

  • Premature delivery and prolonged Catheters use

Supportive Therapy

  • Oxygen and Respiratory and Hemodynamic management

Diagnosis of Sepsis in Children

  • Requires clinical and Lab assessments

Known GBS statues

  • Antibiotics should be given

If the following are present the treatment for sepsis in children:

  1. Oxygen therapy
  2. IV fluid resuscitation

Hypotheses/Proposed Causes of SIDS

  1. Brainstem Abnormality
  2. Maternal Smoking
  3. Poor Prenatal Care and Low Maternal Age

Monitor for Jaundice: Nursing Interventions

  • Examine newborn's skin color in natural light.

SIDS Pathologic Findings

  • Pulmonary Edema and Intrathoracic Hemorrhage are common findings at autopsy

Rh Incompatibility and Rhogam Administration

  • Key Mechanisms: The Mother’s immune system produces antibodies which attack fetal red blood cells

Anti - Rh antibodies

  • cause hemolysis.

Nursing consideration for eye examination

  • Schedule eye exams before discharge for premature newborns who required (02) support to assess retinal damage

Nursing Interventions

  • Monitor for signs of infection and hyperbilirubinemia
  • Initiate feeding and assist with nutrition

Sepsis in Older Children:: Causes and Risks

  • Wound infections.
  • Other infections (MRSA)
  • High Fever
  • Low or no urine output
  • Rapid breathing, shortness of breath and Heart Rate(tachycardia)

Early on set Sepsis occurs

  • In the first 24 hrs. after birth

Prevention SIDS

  • Avoid prone (stomach) position
  • Use safe environment for sleep
  • Encourage, support, and teach breastfeeding

SIDS risk Factors: to avoid

  • Vary the infants head position
  • avoid overheating
  • Discourage bed sharing

SIDS Definition:

  • Sudden and unexplained death for reasons that are not known and cannot explained

Inhaled Nitric Oxide (INO) or Oxygen is used in case/Treatment

  • In severe Cases of SIDS

Meconium Aspiration Syndrome (MAS):

  • Occurs in new borns when a new born breathes in mixed fluid around the the lungs mixed

Complications of Meconium Aspiration Syndromes (MMAS):

  • Air Syndrome Leaks
  1. Causes of Asphyxia and Seizures

Meconium Aspiration syndrome treatment

  • Barrel Shaped Chest and heart rate may be seen on the new borns.

Oxygen Support.

  • Oxygen A face Mask can be can be used with for the baby`s lungs or ECMO

Phototherapy – Nursing Interventions

  1. Newborn Expose skin to lights
  2. Monitor the lights for Intensity
  3. Hydration and Fluid management

Monitor bilirubin levels. Nursing consideration

  • In the first 24- 48 hrs. can be given
  • The therapy is given is when the baby has Kernicterus, which can cause damage to the new borns

ABO Incompatibility

  • Occurs from Mother has A or B antigen
  • Can also affect first baby if the mother has A or B antigen
  • Can be less serve because they Typically do not cross the placenta
  • Check for a poor feeding pattern(sucking reflex), weak , and signs of infection

If mom is Rh + positive

  • Will give Rhogam during the pregnancy

Management for labor

Nursing intervention Support or Provide oxygen therapy

Late onset Sepsis

  • caused by Prolonged or Premature delivery

Premature On set Sepsis:

  • Ampicillin and gentamicin

Management of Sepsis in Older children

  • Antibiotics is to provide nutrition support

Signs of Deterioration in New Born

  • Monitor breathing ( oxygen) and temperature
  • Isolation- is provided to prevent additional infection
  • Maintain body temperature to avoid hypothermia

Treatment for SIDS in children:

  • Oxygen thereapy 02
  • IV fluids resuscitation is needed

GBS

  • All pregnant Women should be Screen using both the Rectal and vaginal Cultures

Baby is crying and active after birth, in order to manage : MAS

  • Do not suction

If the baby shows : MAS the treatment would be

Suctioning: A tube may be placed in the baby's trachea

Causes of RH incompatibility

  • Mother negative and the baby is positive negative Mother with a positive Fetus: The Mother may sensitized positive blood cells to Rh producing the anti -Rh antibodies anti - Rh, These attack to fetus red blood cells causing hemolysis The Mother

What should happen the mother is unsensitized and what test need to be done: for RH incompatibility

  • Rhogam afterbirth, week of gestation and perform procedure

How do you reduce, maintain normal and avoid metabolic conditions in the hospital as part of treatment - SIDS:

  • Use warmth measures ( Maintain body temperature)

Studying That Suits You

Use AI to generate personalized quizzes and flashcards to suit your learning preferences.

Quiz Team

Related Documents

Description

Study notes on pediatric nursing care, including APGAR scoring and management of high-risk newborns such as SGA, LGA, premature, and postmature infants. Covers complications and special considerations for each category. Focuses on conditions like fractured humerus in LGA newborns.

More Like This

Use Quizgecko on...
Browser
Browser