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Questions and Answers
What is the primary mechanism of action of small doses of non-depolarizing neuromuscular blocking agents (NMBs)?
What is the primary mechanism of action of small doses of non-depolarizing neuromuscular blocking agents (NMBs)?
- Stimulation of histamine release
- Direct blockage of ion channels of Nm receptors
- Competitive antagonism of Ach at Nm receptors at the neuromuscular junction (NMJ) (correct)
- Blockade of presynaptic Nn receptors
Which adverse effect is associated with tubocurarine, a prototype non-depolarizing NMB?
Which adverse effect is associated with tubocurarine, a prototype non-depolarizing NMB?
- Increased acetylcholine release
- Enhanced ganglion activity resulting in hypertension
- Histamine release leading to bronchoconstriction (correct)
- Stimulation of beta receptors
What effect does a large dose of non-depolarizing NMB have on the neuromuscular junction?
What effect does a large dose of non-depolarizing NMB have on the neuromuscular junction?
- It blocks presynaptic Nn receptors, leading to decreased Ach release (correct)
- It stimulates ganglion activity at the NMJ
- It competitively antagonizes Ach at Nm receptors
- It enhances Ach release from motor neurons
Which bacteria is most commonly associated with causing necrotizing fasciitis?
Which bacteria is most commonly associated with causing necrotizing fasciitis?
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What is the primary reason for calling the bacteria causing necrotizing fasciitis 'flesh-eating bacteria'?
What is the primary reason for calling the bacteria causing necrotizing fasciitis 'flesh-eating bacteria'?
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Which type of bacteria causing necrotizing fasciitis is commonly found in the oral or enteric environment?
Which type of bacteria causing necrotizing fasciitis is commonly found in the oral or enteric environment?
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