Neuromuscular Blocking Agents and Adverse Effects
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Questions and Answers

What is the primary mechanism of action of small doses of non-depolarizing neuromuscular blocking agents (NMBs)?

  • Stimulation of histamine release
  • Direct blockage of ion channels of Nm receptors
  • Competitive antagonism of Ach at Nm receptors at the neuromuscular junction (NMJ) (correct)
  • Blockade of presynaptic Nn receptors

    • Which adverse effect is associated with tubocurarine, a prototype non-depolarizing NMB?

  • Increased acetylcholine release
  • Enhanced ganglion activity resulting in hypertension
  • Histamine release leading to bronchoconstriction (correct)
  • Stimulation of beta receptors

    • What effect does a large dose of non-depolarizing NMB have on the neuromuscular junction?

  • It blocks presynaptic Nn receptors, leading to decreased Ach release (correct)
  • It stimulates ganglion activity at the NMJ
  • It competitively antagonizes Ach at Nm receptors
  • It enhances Ach release from motor neurons

    • Which bacteria is most commonly associated with causing necrotizing fasciitis?

    <p>Streptococcus pyogenes</p> Signup and view all the answers

    What is the primary reason for calling the bacteria causing necrotizing fasciitis 'flesh-eating bacteria'?

    <p>Production of streptococcal pyrogenic exotoxins A, B &amp; C</p> Signup and view all the answers

    Which type of bacteria causing necrotizing fasciitis is commonly found in the oral or enteric environment?

    <p>Anaerobes</p> Signup and view all the answers

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