Muscle Fiber Membrane and TrPs Formation

What is one of the major hypotheses for the genesis of trigger points (TrPs) discussed in the chapter?

What cycle is related to the genesis of a trigger point according to the text?

What are the relationships examined in the chapter regarding trigger points?

What is discussed at the end of the chapter regarding trigger points treatment methods?

What is a trigger point?

What is the main characteristic of a latent trigger point?

Which term is used to describe trigger points that exist within skeletal muscle tissue or muscular fascia?

What are the two classifications into which all trigger points can be divided?

Which hypothesis links the sliding filament mechanism and the energy crisis to the genesis of a trigger point?

What is the main difference between an active and latent trigger point?

What is the term used to describe taut bands of skeletal muscle tissue where trigger points are located?

Which type of TrP does not cause pain unless compressed?

What term is used for TrPs located within skeletal muscle tissue or muscular fascia?

What is the key characteristic of an active trigger point?

What links the sliding filament mechanism, energy crisis, and genesis of a TrP?

What is the main difference between an active and latent trigger point?

What is the basic structural and functional unit of muscle tissue?

Where are the thin actin filaments attached in a sarcomere?

What causes the myosin-actin cross-bridges to form?

What happens when no ATP molecules are present?

What initiates the breakage of the cross-bridges between myosin and actin filaments?

Which component releases stored calcium ions into the sarcoplasm of a muscle fiber?

What is the role of T tubules in muscle contraction?

What causes a structural change that exposes the binding sites of actin filaments to myosin heads?

What happens when no ATP molecules are present during muscle contraction?

What occurs when ATP is present during muscle contraction?

What happens as long as ATP molecules are present during muscle contraction?

What contributes to the formation of the pain-spasm-pain cycle according to the text?

Why do trigger points (TrPs) not form more frequently in the body?

What is a reason that trigger points (TrPs) are often found in postural muscles?

What triggers the sustained partial depolarization of the motor endplate according to the text?

Why does local swelling in a muscle region contribute to trigger point (TrP) formation?

What is a key factor implicated in the formation of trigger points (TrPs) according to the text?

What is the role of ATP molecules in the sliding filament mechanism?

What initiates an electrical impulse that travels along the muscle fiber's membrane?

What is the function of T tubules in muscle fibers?

Which molecule is likened to a battery due to its role in storing energy in muscle fibers?

In a muscle fiber, what is the primary role of ATP molecules?

What happens if ATP molecules are not present during Step 11 of the sliding filament mechanism?

What is the consequence of not having neurotransmitters in the synapse?

Which structure absorbs calcium ions back into the sarcoplasmic reticulum after muscle contraction?

What initiates an electrical impulse that travels along the outer membrane of a muscle fiber?

What is responsible for transmitting an electrical impulse into the interior of a muscle fiber?

Which theory explains the genesis of trigger points (TrPs) based on energy availability?

What happens if ATP molecules are not present during reabsorption of calcium ions by the sarcoplasmic reticulum after sarcomere contraction?

What can initiate the pain-spasm-pain and contraction-ischemia cycle that leads to trigger point (TrP) formation?

What can result in the tightening of a muscle via the pain-spasm-pain cycle, predisposing it to trigger point (TrP) formation?

What protective mechanism causes muscles in a region to tighten up if there is any pain or damage to an adjacent tissue?

What happens to a muscle that remains in a shortened state for a prolonged period, predisposing it to trigger point (TrP) development?

What happens to muscle contraction when calcium ions are absent in the sarcoplasm?

How does the presence of calcium ions affect the exposure of active sites on actin molecules?

Why does a TrP continue to persist without therapeutic intervention?

What role does ATP play in the formation of TrPs?

Why do venous vessels being closed off worsen the condition of TrPs?

What initiates the contraction-ischemia cycle during muscle contraction?

How does the energy crisis hypothesis explain the formation of TrPs?

What distinguishes a TrP from a globally tight muscle?

"Globally tight muscles occur due to an excess of __________." Complete the statement based on the text.

What contributes to the lack of ATP molecules in the muscle tissue affected by tightness?

Why does a muscle that contracts lose its blood supply?

How does a deficiency of ATP molecules lead to persistent muscle contraction?

Why do sarcomeres located closest to the motor endplate tend to form TrPs more easily?

What term is used to describe the condition that results from constant irritation at the muscle's attachments?

What type of TrP is created by enthesopathy due to the pull of a central TrP?

How does excessive muscle contraction primarily contribute to the genesis of TrPs?

What happens if the pulling force of a central TrP is sufficiently strong according to the text?

What is a common circumstance that can lead to the formation of TrPs?

Why do central TrPs tend to create an enthesopathy?

What happens when a taut band is created within a muscle tissue?

What is the basic structural unit and the functional unit of a muscle?

Where are trigger points (TrPs) located within a muscle?

What is the role of Z-lines in a sarcomere?

What initiates muscle contractions in response to a message from the brain?

Where do motor neurons meet individual muscle fibers?

Which filament is located at the center of a sarcomere?

What floats across the synaptic cleft to bind to the motor endplate of a muscle fiber?

'Shortened active insufficiency' can lead to what condition mentioned in the text?

What structures run the entire length of a muscle?

What are some reasons why trigger points (TrPs) may form?

Which type of muscles do not have their fibers run from attachment to attachment?

What does effective clinical treatment of myofascial pain syndromes require an understanding of?

How does a muscle contract when ordered by the nervous system?

What is necessary to understand before discussing Trigger Points (TrPs)?