Human Sexual Development Quiz

<p>Ovaries will develop (C)</p> Signup and view all the answers

<p>Week 7 (B)</p> Signup and view all the answers

<p>Formation of anti-Mullerian hormone (D)</p> Signup and view all the answers

<p>Enduring attraction to others (B)</p> Signup and view all the answers

<p>Inability to convert Testosterone to DHT (D)</p> Signup and view all the answers

<p>Virilization due to high Testosterone levels (C)</p> Signup and view all the answers

<p>They undergo regression due to AMH (D)</p> Signup and view all the answers

<p>As completely female with undescended testes (B)</p> Signup and view all the answers

<p>Inguinal hernia (C)</p> Signup and view all the answers

<p>Oocytes or sperm (B)</p> Signup and view all the answers

<p>Sertoli cells (B)</p> Signup and view all the answers

<p>Become blood vessels and Leydig cells (D)</p> Signup and view all the answers

<p>At 3 weeks (D)</p> Signup and view all the answers

<p>Primitive sex cords condense in the cortex (D)</p> Signup and view all the answers

<p>Mullerian duct (D)</p> Signup and view all the answers

<p>SRY gene expression (A)</p> Signup and view all the answers

<p>They become ducts contributing to male reproductive structures. (A)</p> Signup and view all the answers

<p>They penetrate medullary mesenchyme to form testis cords. (A)</p> Signup and view all the answers

<p>An individual with some components of both male and female tracts or ambiguous genitalia (B)</p> Signup and view all the answers

<p>Presence of both ovarian and testicular tissue (D)</p> Signup and view all the answers

<p>It initiates the formation of male gonads (D)</p> Signup and view all the answers

<p>Disorder of sexual differentiation (DSD) (D)</p> Signup and view all the answers

<p>46, XY complete gonadal dysgenesis (D)</p> Signup and view all the answers

<p>An individual with male genotype but female external characteristics (A)</p> Signup and view all the answers

<p>Presence of both breasts and male features (A)</p> Signup and view all the answers

<p>Having a male phenotype with XX chromosome configuration (A)</p> Signup and view all the answers

<p>Formation of ambiguous genitalia (D)</p> Signup and view all the answers

<p>Leydig cells (C)</p> Signup and view all the answers

<p>AMH (B)</p> Signup and view all the answers

<p>Regress due to lack of stimulation by testosterone. (D)</p> Signup and view all the answers

<p>Growth of the penis, scrotum, and prostate (C)</p> Signup and view all the answers

<p>Involves the maturation of the Mullerian ducts (C), Results in regression of the Wolffian ducts (D)</p> Signup and view all the answers

<p>Contribute to the formation of Leydig cells (C)</p> Signup and view all the answers

<p>The Mullerian ducts will develop into female genitalia. (C)</p> Signup and view all the answers

<p>Anti-Mullerian Hormone, inhibits Mullerian duct development (A)</p> Signup and view all the answers

<p>Follows the regression of Wolffian structures (D)</p> Signup and view all the answers

<p>Sertoli cells and Leydig cells (C)</p> Signup and view all the answers

<p>Increased virilization due to high testosterone levels (D)</p> Signup and view all the answers

<p>Female external appearance with undescended testes (B)</p> Signup and view all the answers

<p>Development of female external genitalia (D)</p> Signup and view all the answers

<p>Development of ambiguous genitalia or predominantly female features (C)</p> Signup and view all the answers

<p>Primary amenorrhea and lack of body hair (D)</p> Signup and view all the answers

<p>The hormones produced by the gonads (B)</p> Signup and view all the answers

<p>SRY gene activation leads to testicular development followed by phenotypic differentiation. (A)</p> Signup and view all the answers

<p>Inhibits the development of female reproductive structures (C)</p> Signup and view all the answers

<p>They have the potential to become either testes or ovaries, depending on genetic cues. (C)</p> Signup and view all the answers

<p>Complete androgen insensitivity syndrome (B)</p> Signup and view all the answers

<p>Development of ovaries and female external genitalia (A)</p> Signup and view all the answers

<p>Testosterone (D)</p> Signup and view all the answers

<p>They develop into Sperm or Oocytes. (B)</p> Signup and view all the answers

<p>Sertoli cells and seminiferous tubules. (A)</p> Signup and view all the answers

<p>After the interaction with the aorta. (B)</p> Signup and view all the answers

<p>Leydig cells. (C)</p> Signup and view all the answers

<p>They surround primordial germ cells to form testis cords. (C)</p> Signup and view all the answers

<p>Their migration completes by 6 weeks. (B)</p> Signup and view all the answers

<p>Primitive sex cords condense in the cortex. (A)</p> Signup and view all the answers

<p>They regress due to the lack of male hormones. (C)</p> Signup and view all the answers

<p>They give rise to the testes or ovaries. (A)</p> Signup and view all the answers

<p>Testosterone (B)</p> Signup and view all the answers

<p>They regress and disappear (B)</p> Signup and view all the answers

<p>5-alpha reductase is involved in the conversion of testosterone into DHT (C)</p> Signup and view all the answers

<p>Convert androstenedione into estradiol (B)</p> Signup and view all the answers

<p>Continued development of Müllerian ducts (A)</p> Signup and view all the answers

<p>Transform into theca cells (D)</p> Signup and view all the answers

<p>Leydig cells (C)</p> Signup and view all the answers

<p>Development of the penis and scrotum (A)</p> Signup and view all the answers

<p>Wolffian ducts develop under testosterone influence (A)</p> Signup and view all the answers

<p>They lead to feminization of the external genitalia (B)</p> Signup and view all the answers

<p>True hermaphrodite (A)</p> Signup and view all the answers

<p>Ovotesticular DSD (A)</p> Signup and view all the answers

<p>Intersex (C)</p> Signup and view all the answers

<p>Complete Androgen Insensitivity Syndrome (B)</p> Signup and view all the answers

<p>XY sex reversal (C)</p> Signup and view all the answers

<p>Intersex (C)</p> Signup and view all the answers

<p>46, XX testicular DSD (C)</p> Signup and view all the answers

<p>46, XY DSD (B)</p> Signup and view all the answers

<p>True hermaphrodite (A)</p> Signup and view all the answers

<p>It represents the failure of gonadal differentiation. (C)</p> Signup and view all the answers

The genetically determined process that leads to male or female development based on the presence or absence of the Y chromosome.

The physical development of internal and external genitalia into either male or female characteristics.

A gene on the Y chromosome that is crucial for the development of testes in a male embryo.

A hormone produced by Sertoli cells in the testes.

A hormone produced by Leydig cells in the testes.

The stage in embryonic development where the gonads are capable of becoming either ovaries or testes.

The precursor of the female reproductive organs, also known as the paramesonephric duct.

Structures in the early embryo that will develop into either testes or ovaries.

Cells that migrate from the yolk sac to the genital ridges and become either sperm or oocytes.

Cells that develop into Sertoli cells in males and Granulosa cells in females. They surround primordial germ cells.

Cells that contribute to blood vessels and Leydig cells in males, and Theca cells in females. They surround primitive sex cords.

The process that determines whether an individual will develop as male or female, primarily influenced by the presence or absence of the SRY gene on the Y chromosome.

The physical development of internal and external genitalia into either male or female characteristics.

The development of testes in a male embryo, initiated by the SRY gene.

A hormone produced by Sertoli cells in the testes that causes regression of the Mullerian duct, preventing the development of female internal reproductive organs.

The precursor cells for ovarian follicles that develop into Granulosa cells.

They are the main internal reproductive organs in females, developing into the uterine tubes, uterus, and upper 1/3 of the vagina.

The process by which a sexually indifferent embryo differentiates into a male or female.

They are stimulated by testosterone and develop into male internal reproductive organs, including the epididymis, vas deferens, seminal vesicles, and ejaculatory ducts.

Testosterone produced by Leydig cells acts on the Wolffian ducts causing them to develop into the male reproductive structures.

They are key cells in the testes and are responsible for producing testosterone.

They develop into theca cells, which are responsible for producing androstenedione, a precursor to estrogen.

SRY, AMH, and Testosterone.

The physical development of the external genitalia, including the penis, scrotum, labia, and clitoris.

A genetic condition where a 46,XY male has incomplete masculinization due to a problem with testosterone production or its action.

This describes a situation where testosterone is produced, but the body can't convert it into dihydrotestosterone (DHT), leading to incomplete masculinization. It can also be caused by disorders of testosterone reception.

A condition where a 46,XY male has undescended testes and female external genitalia. This happens due to the lack of dihydrotestosterone (DHT), a crucial hormone for the development of male external genitalia.

A 46,XY male with 5-alpha reductase deficiency may appear largely female at birth, with some ambiguous genitalia. But, at puberty, a surge in testosterone can cause masculinization due to a 'mimicking' effect of DHT action.

A condition where a male with a functional SRY gene ('male') has an absence or lack of androgen receptor function, resulting in female external genitalia despite the production of testosterone.

A condition where an individual's sex chromosomes do not align with their physical sex characteristics, resulting in a mismatch between genotype and phenotype.

A broad term encompassing conditions where an individual's sexual development deviates from typical male or female patterns, leading to ambiguous genitalia or a combination of both sexual characteristics.

A rare condition where an individual possesses both ovarian and testicular tissue, resulting in a combination of male and female gonads.

A condition in which an individual with 46, XY chromosomes presents with female external genitalia due to a lack of or resistance to androgen hormones.

A condition in which an individual with 46, XX chromosomes presents with male external genitalia due to the presence of a functional SRY gene on the X chromosome.

A condition where an individual with 46, XY chromosomes has underdeveloped testes and may or may not have ambiguous genitalia, often due to mutations in the SRY gene or other genes involved in testicular development.

A condition where an individual with 46, XX chromosomes develops testes due to the translocation of the SRY gene from the Y chromosome to an X chromosome.

The genetically controlled process that decides if an embryo will develop male or female characteristics. It is determined by the presence or absence of the Y chromosome, which contains the SRY gene.

A gene located on the Y chromosome that plays a crucial role in the development of testes. It activates early in embryonic development, initiating male sexual development.

A hormone produced by Sertoli cells in the testes. It causes regression of the Mullerian duct, preventing the development of female internal reproductive organs.

A hormone produced by Leydig cells in the testes. It plays a key role in masculinization, stimulating the development of the Wolffian ducts into male internal reproductive organs.

The initial stage of gonad development, where the gonads are capable of becoming either ovaries or testes. This stage is before the influence of the SRY gene.

The precursor structure for female internal reproductive organs. The Mullerian duct develops into the fallopian tubes, uterus, and upper vagina.

The precursor of the male reproductive organs, also known as the mesonephric duct.

A hormone produced by Sertoli cells that inhibits the development of female internal reproductive organs.

In males, they develop into Sertoli cells and Leydig cells. In females, they become Granulosa cells and Theca cells.

Develop into the male internal reproductive organs, such as epididymis, vas deferens, seminal vesicles, and ejaculatory ducts.

Without the influence of testosterone, they regress in females.

The key hormone required for the differentiation of the Wolffian ducts into male internal reproductive organs.

They develop into the female internal reproductive organs, such as uterine tubes, uterus, and the upper 1/3 of the vagina.

The presence or absence of DHT dictates the development of male or female external genitalia.

A condition where a 46,XY male has incomplete masculinization due to a problem with testosterone production or its action.

A condition where a 46,XY male lacks the enzyme 5-alpha reductase, preventing the conversion of testosterone into DHT, causing incomplete masculinization.

A condition where a 46,XY male has undescended testes and female external genitalia due to the lack of dihydrotestosterone (DHT) caused by a deficiency in the enzyme 5α-reductase, which converts testosterone to DHT. DHT is crucial for the development of male external genitalia.

A condition where a 46,XY male has incomplete masculinization despite producing normal levels of testosterone, and instead has a female external appearance. This is due to a lack of action by the androgen receptor, which disrupts the proper use of testosterone and its derivatives.

The clinical presentation of a 46,XY male with 5α-reductase deficiency can vary. At birth, they may appear female or have ambiguous genitalia. However, at puberty, high testosterone levels can have a 'mimicking' effect of DHT, leading to masculinization (like deepening of voice, muscle growth) because DHT can act directly on certain tissues.

Testosterone is produced by the testes in males, but it can also be converted into dihydrotestosterone (DHT). DHT is a potent androgen that is critical for the development of male external genitalia. However, if there is a deficiency in the enzyme 5α-reductase, which converts testosterone to DHT, then DHT levels will be low, leading to incomplete masculinization.

A condition where a 46,XY male has normal testes that produce testosterone, but the androgen receptor is dysfunctional or absent. This prevents the body from responding to testosterone, leading to undifferentiated genitals, with a female external appearance. These individuals are often raised as females but may present later in life with a discrepancy between their assigned gender and their internal anatomy.

A rare disorder where an infant is born with gonads of both sexes (female ovaries and male testes). The gonads can be any combination of ovary, testes or a combined ovary and testes (ovotestes).

The appearance of male features (such as body hair, deepening of the voice, etc.) in a female during puberty due to excess androgens.