How much do you know about Osteomyelitis?

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Which type of organisms can produce osteomyelitis?

Only parasites

Only viruses

Only bacteria

All of the above (correct)

What is the most common cause of osteomyelitis?

Parasites

Viruses

Fungi

Mycobacteria and pyogenic bacteria (correct)

Can osteomyelitis only manifest as a primary solitary focus of disease?

Yes

It depends on the type of infection

It depends on the type of organism

No (correct)

What is the most common cause of osteonecrosis?

Unknown causes Signup and view all the answers

What age range does osteonecrosis typically peak in?

30s to 50s Signup and view all the answers

What are the three suspected mechanisms causing osteonecrosis?

Mechanical disruption of vessels, thrombotic occlusion, and extravascular compression Signup and view all the answers

What is the main difference between medullary and subchondral infarcts?

Medullary infarcts involve the trabecular bone and marrow, while subchondral infarcts involve a triangular or wedge-shaped segment of tissue with subchondral bone plate as its base Signup and view all the answers

What happens to the overlying articular cartilage in subchondral infarcts?

It remains viable as it can access nutrients that are present in synovial fluid Signup and view all the answers

What happens to the necrotic trabeculae in the healing response of subchondral infarcts?

Osteoclasts resorb them Signup and view all the answers

How much do you know about Osteomyelitis?

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Questions and Answers

Which type of organisms can produce osteomyelitis?

What is the most common cause of osteomyelitis?

Can osteomyelitis only manifest as a primary solitary focus of disease?

What is the most common cause of osteonecrosis?

What age range does osteonecrosis typically peak in?

What are the three suspected mechanisms causing osteonecrosis?

What is the main difference between medullary and subchondral infarcts?

What happens to the overlying articular cartilage in subchondral infarcts?

What happens to the necrotic trabeculae in the healing response of subchondral infarcts?

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