10 Equine Degenerative Myelopathy

What is the primary role of Vitamin E in preventing Equine Degenerative Myelopathy (EDM)?

Which of the following best describes the characteristic clinical presentation of Equine Protozoal Myeloencephalitis (EPM)?

What is the significance of the N752/D752 mutation in the context of Equine Herpes Myeloencephalitis (EHM)?

Which diagnostic approach offers the most accurate assessment of disease likelihood in cases of Equine Protozoal Myeloencephalitis (EPM)?

What is the most critical factor to consider when collecting cerebrospinal fluid (CSF) for diagnosing Equine Protozoal Myeloencephalitis (EPM)?

A horse presents with clinical signs suggestive of Equine Degenerative Myelopathy (EDM). Serum vitamin E levels are marginal. What additional information would be most useful in supporting a diagnosis of EDM?

Which statement accurately articulates the current understanding of the role of vaccines in managing Equine Herpes Myeloencephalitis (EHM)?

What is the primary reason for the guarded prognosis associated with Equine Degenerative Myelopathy (EDM), even with early and consistent treatment?

What is the primary rationale behind administering nonsteroidal anti-inflammatory drugs (NSAIDs) in the early stages of managing Equine Herpes Myeloencephalitis (EHM)?

Which key element in the life cycle of Sarcocystis neurona is most critical to target when implementing preventative measures against Equine Protozoal Myeloencephalitis (EPM)?

What is the primary mechanism of action of ponazuril and diclazuril, the FDA-approved medications for treating Equine Protozoal Myeloencephalitis (EPM)?

In cases of Equine Herpes Myeloencephalitis (EHM), what is the significance of xanthochromia upon cerebrospinal fluid (CSF) analysis?

A horse is suspected of having Equine Degenerative Myelopathy (EDM) based on clinical signs. If postmortem necropsy is performed; where would be the most important region to examine for characteristic lesions?

A horse displays asymmetric ataxia and muscle atrophy. If Equine Protozoal Myeloencephalitis (EPM) is suspected; where should you collect CSF to increase test sentitivity?

When assessing a horse with suspected Equine Herpes Myeloencephalitis (EHM) during an outbreak, what is the most appropriate protocol regarding isolation?

A horse is diagnosed with Equine Protozoal Myeloencephalitis (EPM) and treated with an FDA-approved medication. Despite initial improvement, the horse relapses several months later. What is the most important consideration for managing the relapse?

Which of the following is the most critical component in designing a biosecurity protocol to prevent the spread of Equine Herpes Myeloencephalitis (EHM) in a large boarding facility?

In the context of managing Equine Degenerative Myelopathy (EDM), what is the primary goal of advising Vitamin E supplementation in predisposed individuals?

What is the implication of a negative test for Sarcocystis neurona in a horse displaying clinical signs of neurological disease?

What is the rationale for using valacyclovir in the treatment of Equine Herpes Myeloencephalitis (EHM)?

What is the best description of the typical signalment of horses that develop Equine Degenerative Myelopathy (EDM)?

Which of the following clinical signs is most suggestive of Equine Herpes Myeloencephalitis (EHM) rather than Equine Protozoal Myeloencephalitis (EPM)?

What characteristic finding on CSF analysis is most suggestive of Equine Herpes Myeloencephalitis (EHM)?

What statement best summarizes the approach to managing Equine Protozoal Myeloencephalitis (EPM) to minimize the risk of relapse after successful treatment?

How could Equine Protozoal Myeloencephalitis be prevented?

When does relapse occur for Equine Protozoal Myeloencephalitis (EPM)?

Which of the following is not a clinical sign of Equine Herpes Myelitis (EHM)?

The gene sequence determines pathogenicity for Equine Herpes Virus. Which has a high virulance in order?

Which of the following has FDA approval for Equine Protozoal Myeloencephalitis (EPM) treatment?

Which of the following is not a cause of Equine spinal ataxia?

Which of the following breeds are not predisposed to Equine Degenerative Myelopathy (EDM)?

How often should a horse be treated with Propionibacterium acnes if following label instructions?

What lesion region would be associated with Equine Protozoal Myeloencephalitis?

What is the relationship between Equine Herpesvirus-1 respiratory infection and Equine Herpes Myeloencephalitis (EHМ)?

At what vitamin E µg/mL level is a horse considered normal and adequate to support neurologic and muscular function?

What serum:CSF ratio result would indicate a postive EPM test?

What is the primary neuropathological finding that differentiates Equine Degenerative Myelopathy (EDM) from Neuroaxonal Dystrophy (NAD)?

Considering the complex interplay of factors contributing to Equine Degenerative Myelopathy (EDM), which of the following best describes its etiology?

A veterinary practice is located in a region with a known high seroprevalence of Sarcocystis neurona. Which of the following strategies would be most effective in reducing the incidence of new Equine Protozoal Myeloencephalitis (EPM) cases?

A horse exhibits asymmetric ataxia, hindlimb weakness, and focal muscle atrophy. Diagnostic testing confirms Equine Protozoal Myeloencephalitis (EPM). Despite treatment with an FDA-approved medication, the horse shows only marginal improvement. What is the most appropriate next step?

In a large equine practice, a cluster of horses suddenly develop neurological signs, including fever, ataxia, and urinary incontinence. Equine Herpes Myeloencephalitis (EHM) is suspected. What immediate action should be taken to control the outbreak and minimize further spread?

Following a confirmed outbreak of Equine Herpes Myeloencephalitis (EHM) in a boarding facility, what is the most critical long-term biosecurity measure to implement to prevent future outbreaks?

A young horse presents with symmetrical ataxia and paresis, worse in the hind limbs. Serum vitamin E levels are within the marginal range. To further investigate a possible diagnosis of Equine Degenerative Myelopathy (EDM), what additional diagnostic step would be most informative?

Considering the limitations of antemortem diagnostics for Equine Degenerative Myelopathy (EDM), which combination of clinical findings and diagnostic results would most strongly suggest EDM?

A horse diagnosed with Equine Protozoal Myeloencephalitis (EPM) initially responds well to treatment but relapses several months later with similar neurological deficits. What factor is most likely contributing to the relapse?

What is the most accurate statement regarding the impact of vaccination on Equine Herpes Myeloencephalitis (EHM) outbreaks?

Based on current understanding of Equine Degenerative Myelopathy (EDM), which of the following management strategies is most likely to improve long-term outcomes in affected horses?

A horse presents with acute onset of fever, severe ataxia, and urine dribbling. Cerebrospinal fluid (CSF) analysis reveals xanthochromia and elevated protein levels, with a normal cell count. Which of the following is the most likely diagnosis?

In comparing Equine Protozoal Myeloencephalitis (EPM) and Equine Herpes Myeloencephalitis (EHM), which statement best differentiates the typical clinical presentation of these diseases?

What is the significance of albuminocytologic dissociation in cerebrospinal fluid (CSF) analysis in cases of Equine Herpes Myeloencephalitis (EHM)?

Which factor is least likely to impact the efficacy of treatment for Equine Protozoal Myeloencephalitis (EPM)?

In a region with high Sarcocystis neurona seroprevalence, what result would give the highest confidence that a horse's neurological issues are due to EPM?

Which of the following scenarios poses the highest risk for the development of Equine Herpes Myeloencephalitis (EHM) within a closed herd of horses?

What is the primary reason behind the recommendation to administer vegetable oil alongside ponazuril when treating Equine Protozoal Myeloencephalitis (EPM)?

Which diagnostic result would be most indicative of EHV-1 as the causative agent in a horse displaying acute neurological signs?

What is the most likely implication of a negative Western blot test for Sarcocystis neurona in a horse displaying neurological signs consistent with Equine Protozoal Myeloencephalitis (EPM)?

Which of the following statements most accurately describes the role of the opossum in the transmission of Equine Protozoal Myeloencephalitis (EPM)?

Considering the clinical presentations and diagnostic challenges, what is the most appropriate approach to differentiating between Equine Degenerative Myelopathy (EDM) and Cervical Vertebral Stenotic Myelopathy (CVSM) in a young horse?

In the context of Equine Herpes Myeloencephalitis (EHM), what is the primary rationale for using valacyclovir?

A horse with confirmed Equine Protozoal Myeloencephalitis (EPM) is being treated with ponazuril. What adjunctive therapy would provide NO benefit?

Which statement accurately describes the role of genetic predisposition in Equine Degenerative Myelopathy (EDM)?

Which of the following best describes the diagnostic approach for Equine Degenerative Myelopathy (EDM)?

During an Equine Herpes Myeloencephalitis (EHM) outbreak, which action poses the highest risk for spreading the virus?

When considering treatment options for a horse diagnosed with Equine Protozoal Myeloencephalitis, which additional immune modulating therapy would be least benificial if using traditionally effective approaches?

With regards to the EPM parasite Sarcocystis neurona, where is the most common lesion location?