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Questions and Answers
What is the likely cause of the termination of a generalized tonic-clonic attack?
What is the predisposition of people who have generalized tonic-clonic attacks?
What type of lesions can cause excess excitability of local brain areas?
What is the typical EEG pattern during the tonic phase of a generalized tonic-clonic seizure?
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What is the likely cause of excess excitability of local brain areas?
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What is the name of the seizures that usually begin in childhood or early adolescence?
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What is the brain system likely involved in absence seizures?
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What is the duration range typically observed during an absence seizure?
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During an absence seizure, which muscle region is most commonly affected by twitchlike contractions?
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What can occasionally initiate a generalized tonic-clonic (grand mal) attack?
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What pattern is exhibited on the brain wave recording of a person with absence seizure epilepsy?
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Which regions of the brain do generalized tonic-clonic seizures presumably involve?
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What is the typical brain wave activity recorded from the thalamus during generalized tonic-clonic seizures?
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Which system is majorly involved during an absence seizure as shown in brain wave recordings?
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What characterizes generalized epileptic seizures at the outset?
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What are generalized seizures primarily subdivided on the basis of?
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What is a characteristic feature of generalized tonic-clonic seizures?
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What may cause cyanosis during a generalized tonic-clonic seizure?
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How long does a usual generalized tonic-clonic seizure last?
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What typically happens to the person after a generalized tonic-clonic seizure?
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