T Hemoglobin

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Which condition is characterized by the production of significant fetal hemoglobin (hemoglobin F) well into adulthood?

Hereditary persistence of fetal hemoglobin (HPFH) (correct)

Thalassemia

Sickle cell anemia

Hemoglobin Gun Hill

What is the result of a point mutation in Hbß, leading to a change from Glu to Val at amino acid 6?

Thalassemia

Hereditary persistence of fetal hemoglobin (HPFH)

Hemoglobin Gun Hill

Sickle cell anemia (correct)

Which condition is associated with the deletion of five amino acids in the β-chains of the abnormal protein?

Sickle cell anemia

Hb Barts

Thalassemia

Hemoglobin Gun Hill (correct)

What is the characteristic feature of Hb Barts and HbH in alpha-thalassemia?

(β4) and (γ4) Signup and view all the answers

In uncontrolled diabetes, what can increase more than twice normal, leading to the generation of more HbA1c than normal?

Blood glucose level Signup and view all the answers

What is the result of a point mutation in Hbß, leading to a change from Glu to Val at amino acid 6?

(microcytic anemia) Signup and view all the answers

What happens in α thalassemia when one of the 4 gene copies are not expressed?

leads to increased death Signup and view all the answers

What are the subunits that form tetramers in beta thalassemia?

β subunits Signup and view all the answers

What is associated with a point mutation in Hbß that leads to the formation of Hb C or Hb E?

Chronic hemolytic anemia Signup and view all the answers

What results from a mutation or incorrect splicing in beta thalassemia?

Thalassemia Signup and view all the answers

T Hemoglobin

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Questions and Answers

Which condition is characterized by the production of significant fetal hemoglobin (hemoglobin F) well into adulthood?

What is the result of a point mutation in Hbß, leading to a change from Glu to Val at amino acid 6?

Which condition is associated with the deletion of five amino acids in the β-chains of the abnormal protein?

What is the characteristic feature of Hb Barts and HbH in alpha-thalassemia?

In uncontrolled diabetes, what can increase more than twice normal, leading to the generation of more HbA1c than normal?

What is the result of a point mutation in Hbß, leading to a change from Glu to Val at amino acid 6?

What happens in α thalassemia when one of the 4 gene copies are not expressed?

What are the subunits that form tetramers in beta thalassemia?

What is associated with a point mutation in Hbß that leads to the formation of Hb C or Hb E?

What results from a mutation or incorrect splicing in beta thalassemia?

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