Chronic Pancreatitis

What is the most common cause of chronic pancreatitis in Western societies?

What is the mnemonic for the predisposing factors of chronic pancreatitis mentioned in the text?

In which age group does chronic pancreatitis peak in the United States?

What percentage of all chronic pancreatitis cases are attributed to alcoholism?

What is the risk of developing pancreatic cancer in patients with hereditary pancreatitis by age 70?

Which of the following is a common complication in chronic pancreatitis patients?

What is the recommended approach for treating exocrine pancreatic insufficiency?

What has shown promise in inhibiting stress on the pancreas by toxic metabolites in some cases?

Which of the following is an FDA-approved pancreatic enzyme preparation?

What may regress after biliary drainage in chronic pancreatitis patients?

What is an effective induction and maintenance agent for treating autoimmune pancreatitis?

What may be indicated in chronic pancreatitis to treat complications, relieve obstruction, and attempt pain relief?

What is a common risk factor for the increased development of pancreatic cancer?

What is the common risk factor for complications in chronic pancreatitis patients?

What is the recommended diet for medical management of chronic pancreatitis?

What may relieve distal bile duct obstruction in chronic pancreatitis?

What is the success rate of endoscopic therapy for chronic pancreatitis?

What procedure provides pain relief in 80% of chronic pancreatitis cases?

What is the risk associated with subtotal or total pancreatectomy with islet autotransplantation?

What may reduce the risk of postoperative pancreatic fistulas?

When is endoscopic or surgical drainage indicated for pseudocysts in chronic pancreatitis?

How are pancreatic ascites or pancreaticopleural fistulas managed?

What procedure may relieve pain in selected patients with chronic pancreatitis?

In what percentage of patients may percutaneous celiac plexus nerve block provide short-lived relief?

What is reported to relieve refractory pain in chronic pancreatitis in a single session?

What is the main cause of death in chronic pancreatitis?

What is associated with poorer quality of life in chronic pancreatitis?

When should all patients with chronic pancreatitis be referred for diagnostic and therapeutic procedures?

What percentage of chronic pancreatitis cases are idiopathic?

Which gene variant is NOT associated with genetic factors predisposing to chronic pancreatitis?

Which classification system for chronic pancreatitis includes 'Toxic-metabolic' as a category?

What hypothesis explains the pathogenesis of chronic pancreatitis by linking it to a first acute pancreatitis event?

What percentage of adults develop diabetes mellitus within 25 years after the clinical onset of chronic pancreatitis?

Which of the following is NOT a clinical finding of chronic pancreatitis?

What is the main cause of pain in chronic pancreatitis?

What symptom may occur late in the course of chronic pancreatitis?

What percentage of chronic pancreatitis cases are caused by obstructions in the pancreas?

Which type of chronic pancreatitis is a complication of immune checkpoint inhibitor therapy?

What is the main characteristic of chronic pancreatitis?

What intervention may resolve chronic pain in chronic pancreatitis?

Which test is recommended to confirm exocrine pancreatic insufficiency?

What is a detectable feature in about 40% of patients with chronic pancreatitis?

What is recommended for diagnosis and shows calcifications, ductal dilatation, and gland atrophy?

Which imaging study is the most sensitive and may show dilated ducts, stones, strictures, or pseudocysts?

What is the gold standard for diagnosis when imaging studies are inconclusive?

What is the basis for the diagnosis of autoimmune pancreatitis in the United States?

What are the complications of chronic pancreatitis?

What imaging features are characteristic of autoimmune pancreatitis?

What is the most sensitive imaging study for chronic pancreatitis?

What is the recommended test for confirming exocrine pancreatic insufficiency?

What is the gold standard for diagnosis when imaging studies are inconclusive?

What is the basis for the diagnosis of autoimmune pancreatitis in the United States?

Chronic pancreatitis occurs most often in patients with alcoholism.

Tobacco smoking is not a risk factor for idiopathic chronic pancreatitis.

About 2% of patients with hyperparathyroidism develop pancreatitis.

In tropical Africa and Asia, tropical pancreatitis is related in part to overnutrition.

Patients with hereditary pancreatitis have a 19% risk of pancreatic cancer by age 70.

Opioid addiction is common in chronic pancreatitis patients.

Exocrine pancreatic insufficiency is treated with pancreatic enzyme replacement therapy.

Antioxidant therapy has shown promise in inhibiting stress on the pancreas by toxic metabolites in some cases.

FDA-approved pancreatic enzyme preparations include Viokace, Creon, Ultresa, Zenpep, and Pancreaze.

Liver fibrosis may regress after biliary drainage in chronic pancreatitis patients.

Distal bile duct obstruction in chronic pancreatitis may be relieved by endoscopic placement of stents.

Endoscopic therapy or surgery may be indicated in chronic pancreatitis to treat complications, relieve obstruction, and attempt pain relief.

Chronic pancreatitis patients have a low risk of developing pancreatic cancer related to tobacco and alcohol use.

Autoimmune pancreatitis is primarily treated with surgical resection.

Chronic pancreatitis patients are advised to increase alcohol consumption for pain relief.

Chronic pancreatitis is commonly managed with a high-fat diet.

Serum amylase and lipase levels are always elevated during acute attacks of chronic pancreatitis.

Elevated alkaline phosphatase and bilirubin levels in chronic pancreatitis can result from bile duct compression.

Vitamin B12 malabsorption is rare in patients with chronic pancreatitis.

Genetic testing for major trypsinogen gene pathogenic variants is not recommended in younger patients with unclear etiology of chronic pancreatitis.

Endoscopic ultrasonographic criteria for chronic pancreatitis diagnosis include hypoechoic foci with shadowing.

ERCP is the most sensitive imaging study for chronic pancreatitis.

Histology is not considered the gold standard for diagnosis when imaging studies are inconclusive.

Autoimmune pancreatitis does not have characteristic imaging features.

The diagnosis of autoimmune pancreatitis in the United States is not based on the HISORt criteria.

Opioid addiction is not a complication of chronic pancreatitis.

Liver and bile duct issues are not complications of chronic pancreatitis.

Osteoporosis is not a complication of chronic pancreatitis.

Endoscopic therapy is successful in 80% of chronic pancreatitis cases

Subtotal or total pancreatectomy with islet autotransplantation is considered a first-line treatment for chronic pancreatitis

Perioperative administration of somatostatin or octreotide has no effect on the risk of postoperative pancreatic fistulas

Endoscopic or surgical drainage is indicated for symptomatic pseudocysts, especially those over 6 cm in diameter

Radiation therapy to the pancreas is a well-established method for relieving refractory pain in chronic pancreatitis

Chronic pancreatitis often leads to disability and reduced life expectancy, with pancreatic cancer as the main cause of death

Chronic pancreatitis is not associated with any specific risk factors

Percutaneous celiac plexus nerve block is considered ineffective for chronic pain and nondilated ducts

Management of chronic pancreatitis does not involve referral for diagnostic and therapeutic procedures for all patients

Chronic pancreatitis can be managed conservatively without the need for medical intervention

Management of chronic pancreatitis is not warranted for severe pain, new jaundice, or fever

Surgery is successful in 80% of cases for obstruction of the duodenal end of the pancreatic duct

Chronic pancreatitis can only be caused by obstructions like strictures, stones, or tumors in the pancreas.

Approximately half of early-onset chronic pancreatitis cases are predisposed by genetic factors.

The SAPE (Sentinel Acute Pancreatitis Event) hypothesis explains the pathogenesis of chronic pancreatitis.

Over 80% of adults develop diabetes mellitus within 25 years after the clinical onset of chronic pancreatitis.

Pain in chronic pancreatitis may vary over time, with a correlation with disease duration or imaging findings.

Steatorrhea, indicated by bulky, foul, fatty stools, may occur early in the course of chronic pancreatitis.

Surgical intervention is not effective in resolving chronic pain in chronic pancreatitis.

More than 80% of adults with chronic pancreatitis develop jaundice within 25 years after the clinical onset.

The TIGAR-O classification system for chronic pancreatitis does not include the category 'Recurrent and severe acute pancreatitis' as a cause.

Chronic pancreatitis often leads to disability and reduced life expectancy, with pancreatic cancer as the main cause of death.

Chronic pancreatitis is not a self-perpetuating disease and can be easily resolved with appropriate management.

Genetic factors predisposing to chronic pancreatitis include variants in the CFTR, PSTI (SPINK1), CTRC, CPA1, UGT1A7, PRSS1, CLDN2, and TRPV6 genes.

Chronic pancreatitis can only be caused by obstructions like strictures, stones, or tumors in the pancreas

Approximately 50% of early-onset chronic pancreatitis cases are predisposed by genetic factors

TIGAR-O classification system for chronic pancreatitis includes 'Recurrent and severe acute pancreatitis' as a category

Chronic pancreatitis is not a self-perpetuating disease

Over 80% of adults develop diabetes mellitus within 15 years after the clinical onset of chronic pancreatitis

Pain in chronic pancreatitis has no correlation with disease duration or imaging findings

Steatorrhea may occur early in the course of chronic pancreatitis

Surgical intervention tailored to the cause of pain may not resolve chronic pain in chronic pancreatitis

Chronic pancreatitis is not characterized by chronic or recurrent episodes of acute pancreatitis

Chronic pancreatitis can be caused by autoimmune factors

M-ANNHEIM classification system for chronic pancreatitis includes 'pancreatitis with multiple risk factors' as a category

Chronic pancreatitis cannot be a complication of immune checkpoint inhibitor therapy

Match the following risk factors with their association to chronic pancreatitis:

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Match the risk factor with the associated increased risk of pancreatic cancer:

Match the medical measure with its associated management for chronic pancreatitis:

Match the FDA-approved pancreatic enzyme preparation with its name:

Match the management option with its indication in chronic pancreatitis:

Match the following diagnostic tests with their indications in chronic pancreatitis:

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Match the following statements with their correct association in chronic pancreatitis:

Match the following management interventions with their efficacy in chronic pancreatitis:

Match the following interventions for reducing postoperative pancreatic fistulas with their effectiveness:

Match the following management approaches for specific complications of chronic pancreatitis with their indications:

Match the genetic factor with its association with chronic pancreatitis:

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Match the statement with its association with chronic pancreatitis:

Match the risk factor with the associated increased risk of chronic pancreatitis:

Match the region with the most common cause of chronic pancreatitis:

Match the imaging finding with its association with chronic pancreatitis:

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Match the risk factor with its associated risk of pancreatic cancer:

Match the medical measure with its indication in chronic pancreatitis:

Match the FDA-approved pancreatic enzyme preparation with its name:

Match the procedure with its indication in chronic pancreatitis:

Match the statement with its correct association with chronic pancreatitis:

Match the imaging finding with its association to chronic pancreatitis:

Match the intervention with its potential impact on chronic pancreatitis:

Match the management with the complication of chronic pancreatitis:

Match the impact on quality of life with associated factors in chronic pancreatitis:

Match the procedure with its potential impact on chronic pancreatitis:

Match the genetic factor with its association with chronic pancreatitis:

Match the classification system with its components for chronic pancreatitis:

Match the risk factor with its association with chronic pancreatitis:

Match the medical measure with its associated management for chronic pancreatitis:

Match the imaging study with its features in chronic pancreatitis diagnosis:

Match the complication with its association in chronic pancreatitis:

Match the diagnostic feature with its association in chronic pancreatitis:

Match the diagnosis criterion with its characteristic in chronic pancreatitis: