Podcast
Questions and Answers
Which heart sound is rarely heard in normal adults, especially when the ventricle is stiff or atrial pressure is high?
Which heart sound is rarely heard in normal adults, especially when the ventricle is stiff or atrial pressure is high?
- First sound
- Second sound
- Third sound
- Fourth sound (correct)
What is the main cause of the second heart sound?
What is the main cause of the second heart sound?
- Acceleration of blood in the heart
- Deceleration of blood in the heart
- Vibrations associated with closure of aortic and pulmonary valves (correct)
- High atrial pressure
What is the frequency range of the first heart sound?
What is the frequency range of the first heart sound?
- 10-20 Hz
- 70-80 Hz
- 25-45 Hz (correct)
- 50-60 Hz
In which phase of the cardiac cycle is the third heart sound typically heard?
In which phase of the cardiac cycle is the third heart sound typically heard?
Where is the best location to hear the third heart sound in most individuals?
Where is the best location to hear the third heart sound in most individuals?
What causes reduplication (physiologic splitting) of the second heart sound?
What causes reduplication (physiologic splitting) of the second heart sound?
Why does the A-V bundle myofibrils have a slow rate of conduction compared to the atrial and ventricular muscles?
Why does the A-V bundle myofibrils have a slow rate of conduction compared to the atrial and ventricular muscles?
Why is the opening of the heart valves not audible when listening with a stethoscope?
Why is the opening of the heart valves not audible when listening with a stethoscope?
What causes the first heart sound (S1) to be low in pitch and relatively long-lasting?
What causes the first heart sound (S1) to be low in pitch and relatively long-lasting?
What contributes to the rapid snap sound heard when the aortic and pulmonary valves close at the end of systole?
What contributes to the rapid snap sound heard when the aortic and pulmonary valves close at the end of systole?
What role do gap junctions play in the conduction of the cardiac action potential?
What role do gap junctions play in the conduction of the cardiac action potential?
Why does one not hear the opening of heart valves even though they are moving?
Why does one not hear the opening of heart valves even though they are moving?
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