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From which phase of the cell cycle do cancer cells primarily develop as a result of gene mutations?
Which type of cancer arises from connective tissues like bone and muscle?
What is the name for the spread of cancer to distant tissues, typically named after the original cancer site?
What do we call the time it takes for a mutation to lead to the development of cancer?
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What happens when tumour suppressor genes are mutated?
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What is the main role of tumour suppressor genes in a normal cell cycle?
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Why is it common for individuals to be born with one defective copy of a tumour suppressor gene?
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What is the role of promoters in cancer development?
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How do cancer cells usually respond to anti-growth signals?
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What is the process by which cancer cells create new blood vessels for fuel?
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Haematologic malignancies include cancers of the blood, blood-forming cells, and the lymphatic system.
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Carcinomas are the most common type of cancer and primarily arise from connective tissues like bone and muscle.
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In the cell cycle, the G0 phase is characterized by cell division and active synthesis of DNA.
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Tumorigenesis initiation primarily occurs in the M phase of the cell cycle.
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Mutated tumour suppressor genes lead to uncontrolled growth or cancer because the speeding system does not happen.
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Individuals with Her-2 gene mutation have a 99% chance of developing cancerous cells.
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Angiogenesis is the process of blocking blood flow to inhibit normal cells.
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Cancer cells respond effectively to anti-growth signals by halting their division.
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Promoters in cancer development are agents that further damage a cell that has already undergone initiation.
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Cancer cells resist cell death (apoptosis) by activating the immune system.
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