Podcast
Questions and Answers
What is a possible consequence of cellular injury in the context of aortic stenosis?
What is a possible consequence of cellular injury in the context of aortic stenosis?
- Vitamin D deficiency
- Metastatic calcification
- Phosphate retention
- Dystrophic calcification (correct)
Which of the following is not a contributing factor to the client's condition of aortic stenosis?
Which of the following is not a contributing factor to the client's condition of aortic stenosis?
- Increased calcium intake over time
- Cellular injury
- Vitamin D deficiency (correct)
- Paget disease
What is the underlying mechanism of dystrophic calcification in heart valves?
What is the underlying mechanism of dystrophic calcification in heart valves?
- Calcium intake over time
- Cancer with metastases
- Phosphate retention leading to calcium deposits
- Deposition of calcium following cellular injury (correct)
Which of the following is associated with metastatic calcification?
Which of the following is associated with metastatic calcification?
What is the primary cause of the client's aortic stenosis?
What is the primary cause of the client's aortic stenosis?
Study Notes
Aortic Stenosis due to Calcification of the Aortic Valve
- Calcification of the aortic valve is caused by dystrophic calcification, which is the deposition of calcium following cellular injury.
- Cellular injury can result in calcium deposition in heart valves, leading to conditions such as aortic stenosis.
- This type of calcification is distinct from metastatic calcification, which is associated with increased serum calcium levels and can be caused by factors such as Paget disease, cancer with metastases, or excess vitamin D.
- Metastatic calcification can also be caused by phosphate retention, leading to calcium deposits.
- In the context of aortic stenosis, dystrophic calcification is the primary mechanism of calcification, rather than metastatic calcification.
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Description
Test your knowledge on a 68-year-old male client with aortic stenosis secondary to calcification of the aortic valve. Identify key aspects of the client's condition and potential contributing factors.